Tokushima University / Educator and Researcher Directory --- Kotani, Yumiko

Field of Study

○	Medicine
○	Pediatrics

Subject of Study

○
○	糖尿病

Book / Paper

Academic Paper (Judged Full Paper):

1.	Tatsuo Mori, Aya Gohji, Yoshihiro Touda, Asami Okada, Yumiko Kotani and Shoji Kagami : Usefulness of individual support and medication for autism spectrum disorder traits in a boy with cyclic vomiting syndrome, No to Hattatsu, Vol.53, No.1, 54-57, 2021. (Summary) Herein we report the case of a 7-year-old boy with cyclic vomiting syndrome (CVS) successfully treated with judicious management and medication of autism spectrum disorder (ASD) traits. Radical surgery for esophageal atresia (Gross C) was performed during the neonatal period. The patient's mother and grandmother both had migraines. The cyclic vomiting began when he was 1 year and 4 months of age, and the episodes occurred 1-3 times monthly. Various drug treatments were tested, but were not effective in controlling the vomiting events. The patient was referred to our hospital at the age of 7 years and 4 months for consultation and treatment. We arrived at a diagnosis of ASD due to observable traits including avoidance of eye contact, obsessiveness, and difficulty in communication. Subsequently, we began individual support and medication for ASD traits to alleviate the CVS. The school has begun to support for his behaviors associated with ASD. Consequently, the psychological anxiety regarding school attendance was addressed, allowing the patient to return to school. When prophylactic drug treatment was changed to the combination of risperidone, amitriptin, and sodium valproate, the vomiting attacks discontinued. The patient is now aged 9 years and 3 months, and has experienced no recurrent vomiting episodes in the past 14 months. In conclusion, it is important to consider the possible coexistence of developmental disorders in patients with intractable CVS, and to provide the reasonable accommodation and treatment for optimal outcomes. (Keyword) 周期性嘔吐症 / 自閉スペクトラム症 / risperidone / amitriptin / 個別支援 (Link to Publication Site) ● Publication site (DOI): 10.11251/ojjscn.53.54 (Link to Search Site for Scientific Articles) ● CiNii @ National Institute of Informatics (CRID): 1390005667266618880 ● Summary page in Scopus @ Elsevier: 2-s2.0-85105807980 (DOI: 10.11251/ojjscn.53.54, CiNii: 1390005667266618880, Elsevier: Scopus)
2.	Asami Okada, Tomohiro Kohmoto, Takuya Naruto, Ichiro Yokota, Yumiko Kotani, Aki Shimada, Yoko Miyamoto, Rizu Takahashi, Aya Goji, Kiyoshi Masuda, Shoji Kagami and Issei Imoto : The first Japanese patient with mandibular hypoplasia, deafness, progeroid features and lipodystrophy diagnosed via POLD1 mutation detection., Human Genome Variation, Vol.4, 17031, 2017. (Summary) Mandibular hypoplasia, deafness, progeroid features and lipodystrophy (MDPL) syndrome is a rare autosomal dominant disorder caused by heterozygous mutations. To date, 13 patients affected by mutation-caused MDPL have been described. We report a clinically undiagnosed 11-year-old male who noted joint contractures at 6 years of age. Targeted exome sequencing identified a known mutation [NM_002691.3:c.1812_1814del, p.(Ser605del)] that diagnosed him as the first Japanese/East Asian MDPL case. (Tokushima University Institutional Repository) ● Metadata: 115129 (Link to Publication Site) ● Publication site (DOI): 10.1038/hgv.2017.31 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 28791128 ● Search Scopus @ Elsevier (PMID): 28791128 ● Search Scopus @ Elsevier (DOI): 10.1038/hgv.2017.31 (Tokushima University Institutional Repository: 115129, DOI: 10.1038/hgv.2017.31, PubMed: 28791128)
3.	Teruyoshi Kageji, Takeshi Miyamoto, Yumiko Kotani, Tuyoshi Kaji, Yoshimi Bando, Yoshifumi Mizobuchi, Kohhei Nakajima and Shinji Nagahiro : Congenital craniopharyngioma treated by radical surgery: case report and review of the literature, Child's Nervous System, Vol.33, No.2, 357-362, 2016. (Summary) Craniopharyngiomas are 5-10 % of all pediatric tumors, but are seldomly encountered in the perinatal period. Only seven instances of a truly antenatal diagnosis of a congenital craniopharyngioma that subsequently underwent radical surgery have been reported. We present the case of a patient who received the diagnosis of a suprasellar tumor during the prenatal period and received radical surgery. We report a case of a neonatal craniopharyngioma treated surgically. The pregnancy progressed uneventfully until a routine ultrasound at 37 weeks of gestation showed a 15 × 15 mm high echoic mass in the center of the fetal head. Neonatal Gd-enhanced T1-weighted MRI at 5 days of life showed a homogenously enhanced mass (16×22×15 mm) in the sellar and suprasellar lesion. As the tumor showed rapid growth at the 3rd month of life, the patient underwent a surgical treatment and the mass was totally removed. Three years later, the physical and mental development of the patient was normal, and Gd-MRI studies showed no tumor recurrence. The present case is the eighth case of a truly antenatal diagnosis of a craniopharyngioma that underwent successful radical surgery. Craniopharyngioma is a benign tumor and thought to be a slow growing tumor in childhood. The results of radical surgery were very poor, and the mortality and morbidity rates were high in the previous reports due to the huge size of tumor at operation. The present case demonstrated the rapid growth in short interval of Gd-MRI. This is the first report of tumor kinetics of congenital craniopharyngioma with previous reports. The calculated tumor doubling time in our case was 37 days. (Keyword) Craniopharyngioma / Female / Humans / magnetic resonance imaging / Neurosurgical Procedures / Pituitary Neoplasms / Pregnancy / Tomography, X-Ray Computed / Ultrasonography, Prenatal (Link to Publication Site) ● Publication site (DOI): 10.1007/s00381-016-3249-1 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 27669698 ● Summary page in Scopus @ Elsevier: 2-s2.0-84991294478 (DOI: 10.1007/s00381-016-3249-1, PubMed: 27669698, Elsevier: Scopus)
4.	Mayu Yuasa, Takeshi Ishigami, Kazutoshi Murao, Yoshiaki Kubo, Yumiko Kotani and Hiroaki Kohno : A case of methylmalonic academia with acrodermatitis enteropathica-like eruption due to essential amino acids deficiency, Japanese Journal of Clinical Dermatology, Vol.68, No.7, 525-529, 2014. (Keyword) メチルマロン酸血症 / 腸性肢端皮膚炎様皮疹 / 必須アミノ酸欠乏 (Link to Search Site for Scientific Articles) ● CiNii @ National Institute of Informatics (CRID): 1390565159844814208 (CiNii: 1390565159844814208)
5.	Miwa Yamaguchi, Akio Kuroda, Yumiko Kotani, Akiko Matsumura, Sakurako Kamano, Hirokazu Uemura, Ichiro Yokota, Shoji Kagami, Kokichi Arisawa and Munehide Matsuhisa : Evaluation of the Sugar Content in the Side Dishes Served in Elementary School Lunches, Journal of the The Japan Diabetes Society, Vol.55, No.12, 952-956, 2012. (Summary) Kuroda et al. proposed a simple method for calculating the carbohydrate content based on a food exchange list (Shokuhin Koukanhyou), with a sugar content of dietary staples of 40 % for rice, 50 % for bread, and 20 % for boiled noodles and a side dish containing 20 g sugar. This study focused on the sugar content in in school lunches, which have a nutrient composition that is different from diabetic meals. The total energy content of school lunches and the sugar content in their side dishes were investigated in 42 school lunches served in elementary school. The average energy intake in the total lunch increased depending on the school grade (670±44 to 752±50 kcal; means±standard deviation). On the other hand, the sugar content of side dishes increased from 29.7±7.5 g in the 1<sup>st</sup>-2<sup>nd</sup> grade to 31.2±8.1 g in the 5<sup>th</sup>-6<sup>th</sup> grade, thus it could be estimated to be 30 g regardless of the energy intake. Two hundred milliliters of milk containing approximately 10 g of carbohydrate was served in every school lunch, which accounted for the 10 g increase in comparison to hospital lunches. These results indicate that the sugar content in the side dishes of elementary school lunches can be estimated to be 30 g regardless of the energy intake.<br> (Keyword) 食事療法 / カーボカウント / 糖質 (Link to Publication Site) ● Publication site (DOI): 10.11213/tonyobyo.55.952 (Link to Search Site for Scientific Articles) ● CiNii @ National Institute of Informatics (CRID): 1390001204908494592 ● Summary page in Scopus @ Elsevier: 2-s2.0-84873382013 (DOI: 10.11213/tonyobyo.55.952, CiNii: 1390001204908494592, Elsevier: Scopus)
6.	Toshie Saito, Maki Urushihara, Yumiko Kotani, Shoji Kagami and Hiroyuki Kobori : Increased urinary angiotensinogen is precedent to increased urinary albumin in patients with type 1 diabetes., The American Journal of the Medical Sciences, Vol.338, No.6, 478-480, 2009. (Summary) Thus, in patients, an increase in urinary angiotensinogen levels is observed, and this increase is precedent to an increase in urinary albumin levels, suggesting that urinary angiotensinogen may function as an early marker of diabetic nephropathy. (Keyword) Adolescent / Albuminuria / Angiotensinogen / Animals / Case-Control Studies / Diabetes Mellitus, Type 1 / Diabetic Nephropathies / Enzyme-Linked Immunosorbent Assay / Female / Humans / Male / Rats / Renin-Angiotensin System / Time Factors (Link to Publication Site) ● Publication site (DOI): 10.1097/MAJ.0b013e3181b90c25 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 19884815 ● Search Scopus @ Elsevier (PMID): 19884815 ● Search Scopus @ Elsevier (DOI): 10.1097/MAJ.0b013e3181b90c25 (DOI: 10.1097/MAJ.0b013e3181b90c25, PubMed: 19884815)
7.	Seiko Kitamura, Ichiro Yokota, Hiroshi Hosoda, Yumiko Kotani, Junko Matsuda, Etsuo Naito, Michinori Ito, Kenji Kangawa and Yasuhiro Kuroda : Ghrelin concentration in cord and neonatal blood -Relation to fetal growth and energy balance-, The Journal of Clinical Endocrinology and Metabolism, Vol.88, No.11, 5473-5477, 2003. (Summary) To investigate the relationship between ghrelin and both fetal and neonatal growth parameters and energy balance, we measured plasma ghrelin concentrations in 54 cord blood samples (male, n = 34; female, n = 20; gestational age, 37.0-41.6 wk; birth weight, 2206-4326 g) and 47 neonatal blood samples (male, n = 27; female, n = 20; postnatal d 3-8). The plasma ghrelin concentrations in cord blood ranged from 110.6-446.1 pmol/liter (median, 206.7 pmol/liter), which were equal to or higher than those in normal weight adults. These values were inversely correlated with birth weight (r = -0.40; P = 0.002), birth length (r = -0.36; P = 0.007), placental weight (r = -0.35; P = 0.01), and IGF-I concentration (r = -0.49; P = 0.0002), but were not significantly correlated with the GH concentration (r = 0.22; P = 0.12). The ghrelin concentrations in small for gestational age newborn were significantly higher than those in appropriate for gestational age newborns (P = 0.0008). The ghrelin concentrations in the vein were significantly higher than those in the artery in 8 cord blood samples (P = 0.01), which suggests that the placenta is an important source of fetal ghrelin. In neonates, the ghrelin concentrations ranged from 133.0-481.7 pmol/liter (median, 268.3 pmol/liter), which were significantly higher than those in cord blood (P < 0.0001). These results suggest that ghrelin may contribute to fetal and neonatal growth. (Keyword) HORMONE SECRETAGOGUE / CIRCULATING GHRELIN / ACYLATED PEPTIDE / BIRTH-WEIGHT / RAT-BRAIN / IGF-II / HUMANS / LEPTIN / EXPRESSION / RECEPTOR (Link to Publication Site) ● Publication site (DOI): 10.1210/jc.2002-021350 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 14602792 ● Search Scopus @ Elsevier (PMID): 14602792 ● Search Scopus @ Elsevier (DOI): 10.1210/jc.2002-021350 (DOI: 10.1210/jc.2002-021350, PubMed: 14602792)

Academic Paper (Unrefereed Paper):

1.	Ichiro Yokota, Maki Moritani, K Nishisho, T Miyoshi, Yumiko Kotani and Shoji Kagami : Detection of glucokinase gene defects in non-obese Japanese children diagnosed with diabetes by school medical examinations., Endocrine Journal, Vol.58, No.9, 741-746, 2011. (Summary) We examined children who were diagnosed with asymptomatic type 2 diabetes by school medical examinations to investigate the existence of glucokinase (GCK) gene defects in this group. Among 20 children diagnosed with asymptomatic type 2 diabetes by school medical examinations between 2003 and 2009 at our 2 hospitals, 8 were classified as non-obese type. Among them, we screened 5 children (2 boys and 3 girls; age: 8-13 years) who had mild elevation of fasting plasma glucose (108-134 mg/dL) with slightly high internationally standardized HbA1c levels (6.3-6.9%) at first close examination. Written informed consent was obtained and all families agreed to participate in this study. We found 4 different mutations (G223S, G81C, S336X and T228M) in 4 of the examined children. The blood glucose control levels had not become worse in any children during the 2-6 years follow-up period. The inheritance of diabetes with GCK gene defect was later confirmed in 1 family. These results suggest that GCK gene defects exist in non-obese children who are diagnosed with asymptomatic diabetes by school medical examinations. Cases of diabetes that are caused by GCK mutations may not be as rare in Japanese subjects as previously described and could be found in patients tentatively diagnosed as type 2 diabetes. (Keyword) Adolescent / Child / DNA / Diabetes Mellitus, Type 2 / Female / Genetic Variation / Glucokinase / Humans / Japan / Male / Polymerase Chain Reaction / Polymorphism, Single Nucleotide / Sequence Analysis, DNA (Link to Publication Site) ● Publication site (DOI): 10.1507/endocrj.K11E-062 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 21720051 ● Search Scopus @ Elsevier (PMID): 21720051 ● Search Scopus @ Elsevier (DOI): 10.1507/endocrj.K11E-062 (DOI: 10.1507/endocrj.K11E-062, PubMed: 21720051)

Proceeding of International Conference:

1.	Etsuo Naito, Toshiaki Hashimoto, T Kumagai, S Yamashita, Yumiko Kotani and Shoji Kagami : Thiamine-responsive pyruvate dehydrogenase deficiency in two patients with normal development and muscular symptoms, The American Society of Human Genetics 60th Annual Meeting, Oct. 2010.

Proceeding of Domestic Conference:

1.	Aya Gohji, Hiroyuki Morino, Asami Okada, Tatsuo Mori, Ken-ichi Suga, Yumiko Kotani, 瀬山理惠, 内山由理 and 松本直通 : Coffin-siris syndrome with persistent open anterior fontanelle in a boy arising from a novel de novo ARID2 variant, 日本人類遺伝学会第67回大会, Dec. 2022.
2.	前田拓也, 花田直子, 岡田朝美, Yumiko Kotani, Makoto Kinoshita, Masahito Nakataki and Shusuke Numata : 先天性ホモシスチン尿症に器質性双極性感情障害を来した一症例, 第62回中国・四国精神神経学会, Nov. 2022.
3.	岡田朝美, T Kohmoto, T Naruto, I Yokota, Yumiko Kotani, A Shimada, Y Miyamoto, R Takahahi, A Goji, K Masuda, Shoji Kagami and Issei Imoto : The first Japanese patient of MDPL syndrome diagnosed via POLD1 mutation detection, 日本人類遺伝学会第62回大会, Nov. 2017.
4.	小野朱美, Yasunobu Hayabuchi, 岡田朝美, Yumiko Kotani and Shoji Kagami : 母体糖尿病による心筋肥大と認識されていたPompe病の1乳児例, 第26回日本小児心筋疾患学会学術集会, Oct. 2017.
5.	Yumiko Kotani, Shoji Kagami, Hiroki Ishibashi and 位田忍 : 卵巣ヘルニア手術を契機に発見された46,XY性分化異常症の1例, 第147回日本小児科学会徳島地方会, Dec. 2016.
6.	菊井聡子, Yoshiko Tani, 山田静恵, 西麻希, Sonoko Yasui, 橋本脩平, 足立知咲, 長尾紀子, 小笠有加, 大町はる佳, Akiko Matsumura, Yumiko Kotani, 内藤悦雄 and Yasuhiro Hamada : 精神症状をきたし低栄養状態に陥った先天性ホモシスチン尿症患者の1症例, 第19回日本病態栄養学会, Jan. 2016.
7.	山尾雅美, 西尾進, 鳥居裕太, 平田有紀奈, 藤岡啓介, Yumiko Kotani, Shoichiro Takao, Naoki Muguruma, Hirotsugu Yamada and Masataka Sata : カテーテル感染を契機に巨大脾腫をきたした一例, 第23回日本超音波医学会四国地方会学術集会, Oct. 2013.
8.	Yumiko Kotani and Shoji Kagami : 生活習慣の改善とリラグルチドが著効したIgA腎症に合併した2型糖尿病の1例, 第139回日本小児科学会徳島地方会, Dec. 2012.
9.	Etsuo Naito, Yumiko Kotani and Shoji Kagami : 新生児タンデムマススクリーニングで発見された中鎖アシルCoA脱水素酵素(MCAD)欠損症の1女児例, 第139回日本小児科学会徳島地方会, Dec. 2012.
10.	宮本健志, Yoshifumi Mizobuchi, Teruyoshi Kageji, Shinji Nagahiro, Yumiko Kotani, Shoji Kagami, Takashi Kaji and Minoru Irahara : 胎児期に発見された頭蓋咽頭腫の一例, 第74回㈳日本脳神経外科学会中国四国支部学術集会, Dec. 2012.
11.	Miwa Yamaguchi, Yumiko Kotani, Akiko Matsumura, Ichiro Yokota, Shoji Kagami, Kokichi Arisawa, Akio Kuroda and Munehide Matsuhisa : 学校給食の副食糖質量の検討, 第55回日本糖尿病学会年次学術集会, May 2012.
12.	Miwa Yamaguchi, Yumiko Kotani, Akio Kuroda, Ichiro Yokota, Munehide Matsuhisa, Kokichi Arisawa and Shoji Kagami : 徳島市学校給食の副食糖質量の検討, 第15回日本病態栄養学会年次学術集会, Jan. 2012.
13.	Miwa Yamaguchi, Akiko Matsumura, Yumiko Kotani, Akio Kuroda, Ichiro Yokota, Munehide Matsuhisa, Kokichi Arisawa and Shoji Kagami : 外来栄養指導でカーボカウントの継続が見られた例, 第15回日本病態栄養学会年次学術集会, Jan. 2012.
14.	Yumiko Kotani and Shoji Kagami : 小児1型糖尿病に対するインスリン持続皮下注射療法導入の当院での現状, 第137回日本小児科学会徳島地方会, Dec. 2011.
15.	富本亜由美, Yumiko Kotani, Shoji Kagami, 重松陽介, 矢崎正英 and 内藤悦夫 : タンデムマススクリーニングで発見されたシトリン欠損症, 第137回に本小児科学会徳島地方会, Dec. 2011.
16.	Yumiko Kotani, Masako Sei, Ichiro Yokota and Shoji Kagami : 学校検尿について, 第49回日本糖尿病学会中国四国地方会, Nov. 2011.
17.	Miwa Yamaguchi, Yumiko Kotani, Ichiro Yokota, Akio Kuroda, Munehide Matsuhisa, Kokichi Arisawa and Shoji Kagami : 学校給食の副食糖質量の検討, 日本糖尿病学会中国四国地方会第49回総会, Nov. 2011.
18.	Yumiko Kotani, 苛原誠, Shoji Kagami, Kazuhiro Mori, 山上貴司, 松岡優, Ichiro Yokota, 増江道哉, 西堀弘記 and 福山誠介 : 新生児早期発症高インスリン性低血糖症の1例, 第63回中国四国小児科学会, Nov. 2011.
19.	Maki Urushihara, Yumiko Kotani, 小堀浩幸 and Shoji Kagami : 1型糖尿病では尿中アンジオテンシノーゲン排泄の増加はアルブミン尿より先行する, 第114回日本小児科学会学術集会, Aug. 2011.
20.	森達夫, Kenji Mori, 永井隆, Yumiko Kotani and Shoji Kagami : 難治性の全身性ジストニアを来たしたHyperinsulinism-hyperammonaemia syndromeの1例, 第114回日本小児科学会学術集会, Aug. 2011.
21.	森達夫, Kenji Mori, 永井隆, Yumiko Kotani, Shoji Kagami, Ryuji Kaji and Satoshi Goto : 2次性に難治性の全身性ジストニアを来たしたhyperinsulinism-hyperammonaemia syndrome(HHS)の1女児例, 第22回日本小児神経学会中国・四国地方会, Jul. 2011.
22.	Miwa Yamaguchi, Yumiko Kotani, Ichiro Yokota, Akio Kuroda, Munehide Matsuhisa and Shoji Kagami : 小児期発症1型糖尿病患者へのカーボカウント教育の試み, 小児思春期糖尿病学会, Jul. 2011.
23.	Ichiro Yokota, 西庄かほる, Maki Moritani, Yumiko Kotani and Shoji Kagami : 学校検診で発見された糖尿病におけるグルコキナーゼ(GCK)遺伝子変異の検討, 第135回日本小児科学会徳島地方会, Dec. 2010.
24.	Miwa Yamaguchi, Yumiko Kotani and Shoji Kagami : 1型糖尿病児へのカーボカウント導入の試みとQOLへの影響, 第135回日本小児科学会徳島地方会, Dec. 2010.
25.	Etsuo Naito, 田久保憲行, 重松陽介, Yumiko Kotani and Shoji Kagami : タンデムマスを用いた新生児マス・スクリーニングにより発見されたシトルリン血症1型軽症例とシトリン欠損症との鑑別, 第135回日本小児科学会徳島地方会, Dec. 2010.
26.	Nami Inoue, Hiroyoshi Watanabe, 岡村和美, Yumiko Kotani and Shoji Kagami : 脳腫瘍経験者の長期フォローアップに関する検討, 第135回日本小児科学会徳島地方会, Dec. 2010.
27.	森達夫, Yoshihiro Touda, Yumiko Kotani, Emiko Fujii, Kenji Mori, Ichiro Yokota, 山上貴司, 松岡優 and Shoji Kagami : 2次性に難治性の全身性ジストニアを来たした3症例, 第21回日本小児神経学会中国・四国地方会, Jul. 2010.
28.	Etsuo Naito, 島川清司, Toshiaki Hashimoto, Yumiko Kotani and Shoji Kagami : E1αサブユニット遺伝子変異を有したピルビン酸脱水素酵素欠損症32家系の保因者診断, 第52回日本小児神経学会総会, May 2010.

Et cetera, Workshop:

1.	岡田朝美, Yumiko Kotani, Shoji Kagami, Issei Imoto and Ichiro Yokota : POLD1 遺伝子変異によって診断された日本人初のMDPL 症候群患者, 第149回日本小児科学会徳島地方会, Dec. 2017.
2.	岡田朝美, 田山貴広, 小野朱美, Yumiko Kotani, Yasunobu Hayabuchi and Shoji Kagami : 母体糖尿病による心筋肥大と認識されていたPompe 病の1 乳児例, 第149回日本小児科学会徳島地方会, Dec. 2017.
3.	Yumiko Kotani, 岡田朝美, 近藤梨恵子, Sato Matsura, 鈴江真史, 岸揚子, 山上貴司, 長谷川高誠 and Shoji Kagami : 多彩な骨折歴をもつCOL1A2 遺伝子異常症の1 例, 第148回日本小児科学会徳島地方会, Jun. 2017.
4.	岡田朝美, Yumiko Kotani, Shoji Kagami, 蔭山彩人, 松田拓 and Kohhei Nakajima : 成長曲線から疑うことが可能であった間脳下垂体腫瘍の2学童例, 第150回日本小児科学会徳島地方会, Jun. 2016.
5.	Etsuo Naito, Yumiko Kotani, Shoji Kagami, Nami Inoue and 重松陽介 : 新生児タンデムマススクリーニングを契機に診断できたグルタル酸血症Ⅰ型(p.Ser255Leu変異)の母親, 第140回日本小児科学会徳島地方会, Jun. 2014.
6.	中村真理, 吉田友紀, Hiroe Tani, Hiroko Hashimoto, Kenji Mori, Yumiko Kotani and Shoji Kagami : 母親が認識する1型糖尿病患児の療養管理における父親の役割に関する研究, 小児保健とくしま, No.22, 13-19, 2014.

Grants-in-Aid for Scientific Research (KAKEN Grants Database @ NII.ac.jp)

Search by Researcher Number (60423414)

Designated Assistant Professor : Kotani, Yumiko

Research

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Book / Paper

Academic Paper (Judged Full Paper):

Academic Paper (Unrefereed Paper):

Proceeding of International Conference:

Proceeding of Domestic Conference:

Et cetera, Workshop:

Grants-in-Aid for Scientific Research (KAKEN Grants Database @ NII.ac.jp)