=== Generating (published_papers) ===
=== Generating (teaching_experience) ===
=== Generating (education) ===
=== Generating (misc) ===
=== Generating (research_projects) ===
=== Generating (presentations) ===
==== begin registerFile(/WWW/pub2/data/ERD/person/246736/researchmap/published_papers.jsonl) ====
line:1, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970599"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/117775","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/36421669","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=392995","label":"url"}],"paper_title":{"en":"Caffeic Acid Phenethyl Ester Induces Vascular Endothelial Growth Factor Production and Inhibits CXCL10 Production in Human Dental Pulp Cells","ja":"Caffeic Acid Phenethyl Ester Induces Vascular Endothelial Growth Factor Production and Inhibits CXCL10 Production in Human Dental Pulp Cells"},"authors":{"en":[{"name":"Kuramoto Hitomi"},{"name":"Nakanishi Tadashi"},{"name":"Takegawa Daisuke"},{"name":"Katsuhiro MIEDA"},{"name":"Hosaka Keiichi"}],"ja":[{"name":"蔵本 瞳"},{"name":"中西 正"},{"name":"武川 大輔"},{"name":"三枝 克啓"},{"name":"保坂 啓一"}]},"description":{"en":"The survival rate of root non-vital teeth is lower than that of vital teeth. Therefore, to preserve the dental pulp is very important. The vascular endothelial growth factor (VEGF) is the most potent angiogenic factor involved in the vitality of dental pulp including reparative dentin formation. Caffeic acid phenethyl ester (CAPE) is a physiologically active substance of propolis and has some bioactivities such as anti-inflammatory effects. However, there are no reports on the effects of CAPE on dental pulp inflammation. In this study, we investigated the effects of CAPE on VEGF and inflammatory cytokine production in human dental pulp cells (HDPCs) to apply CAPE to an ideal dental pulp protective agent. We found that CAPE induced VEGF production from HDPCs. Moreover, CAPE induced the phosphorylation of p38 mitogen-activated protein kinase (MAPK), extracellular signal-regulated kinases (ERK), and stress-activated protein kinase/c-Jun N-terminal kinase (SAP/JNK) in HDPCs. Furthermore, CAPE inhibited C-X-C motif chemokine ligand 10 (CXCL10) production in Pam3CSK4- and tumor necrosis factor-alpha (TNF-α)-stimulated HDPCs. In conclusion, these results suggest that CAPE might be useful as a novel biological material for vital pulp therapy by exerting the effects of VEGF production and anti-inflammatory activities.","ja":"The survival rate of root non-vital teeth is lower than that of vital teeth. Therefore, to preserve the dental pulp is very important. The vascular endothelial growth factor (VEGF) is the most potent angiogenic factor involved in the vitality of dental pulp including reparative dentin formation. Caffeic acid phenethyl ester (CAPE) is a physiologically active substance of propolis and has some bioactivities such as anti-inflammatory effects. However, there are no reports on the effects of CAPE on dental pulp inflammation. In this study, we investigated the effects of CAPE on VEGF and inflammatory cytokine production in human dental pulp cells (HDPCs) to apply CAPE to an ideal dental pulp protective agent. We found that CAPE induced VEGF production from HDPCs. Moreover, CAPE induced the phosphorylation of p38 mitogen-activated protein kinase (MAPK), extracellular signal-regulated kinases (ERK), and stress-activated protein kinase/c-Jun N-terminal kinase (SAP/JNK) in HDPCs. Furthermore, CAPE inhibited C-X-C motif chemokine ligand 10 (CXCL10) production in Pam3CSK4- and tumor necrosis factor-alpha (TNF-α)-stimulated HDPCs. In conclusion, these results suggest that CAPE might be useful as a novel biological material for vital pulp therapy by exerting the effects of VEGF production and anti-inflammatory activities."},"publication_date":"2022-11-15","publication_name":{"en":"Current Issues in Molecular Biology","ja":"Current Issues in Molecular Biology"},"volume":"Vol.44","number":"No.11","starting_page":"5691","ending_page":"5699","languages":["eng"],"referee":true,"identifiers":{"doi":["10.3390/cimb44110385"],"issn":["1467-3045"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:2, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970600"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/114566","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/31930126","label":"url"},{"@id":"https://www.scopus.com/record/display.url?eid=2-s2.0-85077441042&origin=inward","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=362484","label":"url"}],"paper_title":{"en":"Caffeic acid phenethyl ester (CAPE) induces VEGF expression and production in rat odontoblastic cells","ja":"Caffeic acid phenethyl ester (CAPE) induces VEGF expression and production in rat odontoblastic cells"},"authors":{"en":[{"name":"Kuramoto Hitomi"},{"name":"Hirao Kouji"},{"name":"Yumoto Hiromichi"},{"name":"Hosokawa Yuki"},{"name":"Nakanishi Tadashi"},{"name":"Takegawa Daisuke"},{"name":"Washio Ayako"},{"name":"Kitamura Chiaki"},{"name":"Matsuo Takashi"}],"ja":[{"name":"蔵本 瞳"},{"name":"平尾 功治"},{"name":"湯本 浩通"},{"name":"細川 由樹"},{"name":"中西 正"},{"name":"武川 大輔"},{"name":"Washio Ayako"},{"name":"Kitamura Chiaki"},{"name":"松尾 敬志"}]},"description":{"en":"Caffeic acid phenethyl ester (CAPE), the main component of propolis, has various biological activities including anti-inflammatory effect and wound healing promotion. Odontoblasts located in the outermost layer of dental pulp play crucial roles such as production of growth factors and formation of hard tissue termed reparative dentin in host defense against dental caries. In this study, we investigated the effects of CAPE on the upregulation of vascular endothelial growth factor (VEGF) and calcification activities of odontoblasts, leading to development of novel therapy for dental pulp inflammation caused by dental caries. CAPE significantly induced mRNA expression and production of VEGF in rat clonal odontoblast-like KN-3 cells cultured in normal medium or osteogenic induction medium. CAPE treatment enhanced nuclear factor-kappa B (NF-B) transcription factor activation, and furthermore, the specific inhibitor of NF-B significantly reduced VEGF production. The expression of VEGF receptor- (VEGFR-) 2, not VEGFR-1, was up regulated in KN-3 cells treated with CAPE. In addition, VEGF significantly increased mineralization activity in KN-3 cells. These findings suggest that CAPE might be useful as a novel biological material for the dental pulp conservative therapy.","ja":"Caffeic acid phenethyl ester (CAPE), the main component of propolis, has various biological activities including anti-inflammatory effect and wound healing promotion. Odontoblasts located in the outermost layer of dental pulp play crucial roles such as production of growth factors and formation of hard tissue termed reparative dentin in host defense against dental caries. In this study, we investigated the effects of CAPE on the upregulation of vascular endothelial growth factor (VEGF) and calcification activities of odontoblasts, leading to development of novel therapy for dental pulp inflammation caused by dental caries. CAPE significantly induced mRNA expression and production of VEGF in rat clonal odontoblast-like KN-3 cells cultured in normal medium or osteogenic induction medium. CAPE treatment enhanced nuclear factor-kappa B (NF-B) transcription factor activation, and furthermore, the specific inhibitor of NF-B significantly reduced VEGF production. The expression of VEGF receptor- (VEGFR-) 2, not VEGFR-1, was up regulated in KN-3 cells treated with CAPE. In addition, VEGF significantly increased mineralization activity in KN-3 cells. These findings suggest that CAPE might be useful as a novel biological material for the dental pulp conservative therapy."},"publication_date":"2019-12-20","publication_name":{"en":"BioMed Research International","ja":"BioMed Research International"},"volume":"Vol.Article ID 5390720","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1155/2019/5390720"],"issn":["2314-6133"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:3, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970601"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390282680498132224/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=343554","label":"url"}],"paper_title":{"en":"Anti-inflammatory Effects of Catechin on Rat Odontoblastic Cells (KN-3)","ja":"ラット象牙芽細胞様細胞 (KN-3) におけるカテキンの抗炎症作用"},"authors":{"en":[{"name":"Hirao Kouji"},{"name":"Yumoto Hiromichi"},{"name":"Hosokawa Yuki"},{"name":"Kuramoto Hitomi"},{"name":"鷲尾 絢子"},{"name":"Nakanishi Tadashi"},{"name":"Takegawa Daisuke"},{"name":"北村 知昭"},{"name":"Matsuo Takashi"}],"ja":[{"name":"平尾 功治"},{"name":"湯本 浩通"},{"name":"細川 由樹"},{"name":"蔵本 瞳"},{"name":"鷲尾 絢子"},{"name":"中西 正"},{"name":"武川 大輔"},{"name":"北村 知昭"},{"name":"松尾 敬志"}]},"publication_date":"2017-10","publication_name":{"en":"The Japanese Journal of Conservative Dentistry","ja":"日本歯科保存学雑誌"},"volume":"Vol.60","number":"No.5","starting_page":"235","ending_page":"244","languages":["jpn"],"referee":true,"identifiers":{"doi":["10.11471/shikahozon.60.235"],"issn":["2188-0808"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:4, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970602"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/112386","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/27747243","label":"url"},{"@id":"https://www.scopus.com/record/display.url?eid=2-s2.0-84991669747&origin=inward","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=327689","label":"url"}],"paper_title":{"en":"Functional roles of NOD1 in odontoblasts on dental pulp innate immunity","ja":"Functional roles of NOD1 in odontoblasts on dental pulp innate immunity"},"authors":{"en":[{"name":"Hosokawa Yuki"},{"name":"Hirao Kouji"},{"name":"Yumoto Hiromichi"},{"name":"Washio Ayako"},{"name":"Nakanishi Tadashi"},{"name":"Takegawa Daisuke"},{"name":"Kitamura Chiaki"},{"name":"Matsuo Takashi"}],"ja":[{"name":"Hosokawa Yuki"},{"name":"平尾 功治"},{"name":"湯本 浩通"},{"name":"Washio Ayako"},{"name":"中西 正"},{"name":"武川 大輔"},{"name":"Kitamura Chiaki"},{"name":"松尾 敬志"}]},"description":{"en":"Caries-related pathogens are first recognized by odontoblasts and induce inflammatory events that develop to pulpitis. Generally, initial sensing of microbial pathogens is mediated by pattern recognition receptors, such as Toll-like receptor and nucleotide-binding oligomerization domain (NOD); however, little is known about NODs in odontoblasts. In this study, the levels of NODs expressed in rat odontoblastic cell line, KN-3, were assessed by flow cytometry and the levels of chemokines in NOD-specific ligand-stimulated KN-3 cells were analyzed by real-time PCR and ELISA. The signal transduction pathway activated with NOD-specific ligand was assessed by blocking assay with specific inhibitors and reporter assay. In KN-3 cells, the expression level of NOD1 was stronger than that of NOD2 and the production of chemokines, such as CINC-1, CINC-2, CCL20, and MCP-1, was upregulated by stimulation with NOD1-specific ligand, but not with NOD2-specific ligand. CINC-2 and CCL20 production by stimulation with NOD1-specific ligand was reduced by p38 MAPK and AP-1 signaling inhibitors. Furthermore, the reporter assay demonstrated AP-1 activation in NOD1-specific ligand-stimulated KN-3 cells. These findings indicated that NOD1 expressed in odontoblasts functions to upregulate the chemokines expression via p38-AP-1 signaling pathway and suggested that NOD1 may play important roles in the initiation and progression of pulpitis.","ja":"Caries-related pathogens are first recognized by odontoblasts and induce inflammatory events that develop to pulpitis. Generally, initial sensing of microbial pathogens is mediated by pattern recognition receptors, such as Toll-like receptor and nucleotide-binding oligomerization domain (NOD); however, little is known about NODs in odontoblasts. In this study, the levels of NODs expressed in rat odontoblastic cell line, KN-3, were assessed by flow cytometry and the levels of chemokines in NOD-specific ligand-stimulated KN-3 cells were analyzed by real-time PCR and ELISA. The signal transduction pathway activated with NOD-specific ligand was assessed by blocking assay with specific inhibitors and reporter assay. In KN-3 cells, the expression level of NOD1 was stronger than that of NOD2 and the production of chemokines, such as CINC-1, CINC-2, CCL20, and MCP-1, was upregulated by stimulation with NOD1-specific ligand, but not with NOD2-specific ligand. CINC-2 and CCL20 production by stimulation with NOD1-specific ligand was reduced by p38 MAPK and AP-1 signaling inhibitors. Furthermore, the reporter assay demonstrated AP-1 activation in NOD1-specific ligand-stimulated KN-3 cells. These findings indicated that NOD1 expressed in odontoblasts functions to upregulate the chemokines expression via p38-AP-1 signaling pathway and suggested that NOD1 may play important roles in the initiation and progression of pulpitis."},"publication_date":"2016-09-25","publication_name":{"en":"BioMed Research International","ja":"BioMed Research International"},"volume":"Vol.2016","starting_page":"9325436","ending_page":"9325436","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1155/2016/9325436"],"issn":["2314-6141"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:5, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970603"},"force":{"see_also":[{"@id":"https://ci.nii.ac.jp/naid/40021045756/","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1522262181026542720/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=307661","label":"url"}],"paper_title":{"en":"Effects of perioperative oral management in thoracoscopic lung lobectomy : Retrospective study","ja":"胸腔鏡下肺葉切除術における周術期口腔機能管理の効果に関する検討―後ろ向き研究―"},"authors":{"en":[{"name":"Yamamura Yoshiko"},{"name":"Takizawa Hiromitsu"},{"name":"Matsumoto Fumihiro"},{"name":"Momota Yukihiro"},{"name":"Aota Keiko"},{"name":"Takegawa Daisuke"},{"name":"近藤 智香"},{"name":"山ノ井 朋子"},{"name":"Takano Hideyuki"},{"name":"Kani Kohichi"},{"name":"十川 悠香"},{"name":"Kawano Fumiaki"},{"name":"Matsuo Takashi"},{"name":"Sakiyama Shoji"},{"name":"Azuma Masayuki"}],"ja":[{"name":"山村 佳子"},{"name":"滝沢 宏光"},{"name":"松本 文博"},{"name":"桃田 幸弘"},{"name":"青田 桂子"},{"name":"武川 大輔"},{"name":"近藤 智香"},{"name":"山ノ井 朋子"},{"name":"高野 栄之"},{"name":"可児 耕一"},{"name":"十川 悠香"},{"name":"河野 文昭"},{"name":"松尾 敬志"},{"name":"先山 正二"},{"name":"東 雅之"}]},"publication_date":"2016-03","publication_name":{"en":"Oral Care","ja":"日本口腔ケア学会雑誌"},"volume":"Vol.10","number":"No.1","starting_page":"106","ending_page":"110","languages":["jpn"],"referee":true,"identifiers":{"issn":["1881-9141"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:6, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970604"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/24847951","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=315932","label":"url"}],"paper_title":{"en":"Catechins inhibit vascular endothelial growth factor production and cyclooxygenase-2 expression in human dental pulp cells","ja":"Catechins inhibit vascular endothelial growth factor production and cyclooxygenase-2 expression in human dental pulp cells"},"authors":{"en":[{"name":"Nakanishi Tadashi"},{"name":"Mukai Kayo"},{"name":"Hosokawa Yoshitaka"},{"name":"Takegawa Daisuke"},{"name":"Matsuo Takashi"}],"ja":[{"name":"中西 正"},{"name":"向井 佳代"},{"name":"細川 義隆"},{"name":"武川 大輔"},{"name":"松尾 敬志"}]},"description":{"en":"To investigate the effect of catechins on vascular endothelial growth factor (VEGF) production and cyclooxygenase-2 (COX-2) expression in human dental pulp cells (HDPC) stimulated with bacteria-derived factors or pro-inflammatory cytokines. Morphologically fibroblastic cells established from explant cultures of healthy human dental pulp tissues were used as HDPC. HDPC pre-treated with catechins, epigallocatechin-3-gallate (EGCG) or epicatechin gallate (ECG), were exposed to lipopolysaccharide (LPS), peptidoglycan (PG), interlukin-1β (IL-1β) or tumour necrosis factor-α (TNF-α). VEGF production was examined by enzyme-linked immunosorbent assay, and COX-2 expression was assessed by immunoblot. EGCG and ECG significantly reduced LPS- or PG-mediated VEGF production in the HDPC in a dose-dependent manner. EGCG also prevented IL-1β-mediated VEGF production. Although TNF-α did not enhance VEGF production in the dental pulp cells, treatment of 20 μg mL(-1) of EGCG decreased the level of VEGF. In addition, the catechins attenuated COX-2 expression induced by LPS and IL-1β. The up-regulated VEGF and COX-2 expressions in the HDPC stimulated with these bacteria-derived factors or IL-1β were diminished by the treatment of EGCG and ECG. These findings suggest that the catechins may be beneficial as an anti-inflammatory tool of the treatment for pulpal inflammation.","ja":"To investigate the effect of catechins on vascular endothelial growth factor (VEGF) production and cyclooxygenase-2 (COX-2) expression in human dental pulp cells (HDPC) stimulated with bacteria-derived factors or pro-inflammatory cytokines. Morphologically fibroblastic cells established from explant cultures of healthy human dental pulp tissues were used as HDPC. HDPC pre-treated with catechins, epigallocatechin-3-gallate (EGCG) or epicatechin gallate (ECG), were exposed to lipopolysaccharide (LPS), peptidoglycan (PG), interlukin-1β (IL-1β) or tumour necrosis factor-α (TNF-α). VEGF production was examined by enzyme-linked immunosorbent assay, and COX-2 expression was assessed by immunoblot. EGCG and ECG significantly reduced LPS- or PG-mediated VEGF production in the HDPC in a dose-dependent manner. EGCG also prevented IL-1β-mediated VEGF production. Although TNF-α did not enhance VEGF production in the dental pulp cells, treatment of 20 μg mL(-1) of EGCG decreased the level of VEGF. In addition, the catechins attenuated COX-2 expression induced by LPS and IL-1β. The up-regulated VEGF and COX-2 expressions in the HDPC stimulated with these bacteria-derived factors or IL-1β were diminished by the treatment of EGCG and ECG. These findings suggest that the catechins may be beneficial as an anti-inflammatory tool of the treatment for pulpal inflammation."},"publication_date":"2015-03","publication_name":{"en":"International Endodontic Journal","ja":"International Endodontic Journal"},"volume":"Vol.48","number":"No.3","starting_page":"277","ending_page":"282","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1111/iej.12312"],"issn":["0143-2885"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:7, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970605"},"force":{"see_also":[{"@id":"http://ci.nii.ac.jp/naid/120005823521/","label":"url"},{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/109866","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1050564287418444544/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=317411","label":"url"}],"paper_title":{"en":"Current Status and Problems Regarding the Perioperative Oral Function Management in the Tokushima University Hospital","ja":"徳島大学病院における周術期口腔機能管理の現状と課題"},"authors":{"en":[{"name":"Aota Keiko"},{"name":"Yamamura Yoshiko"},{"name":"山ノ井 朋子"},{"name":"Takegawa Daisuke"},{"name":"Kani Kohichi"},{"name":"Takano Hideyuki"},{"name":"Momota Yukihiro"},{"name":"Matsumoto Fumihiro"},{"name":"Sugawara Chieko"},{"name":"Yoshioka Masami"},{"name":"Kawano Fumiaki"},{"name":"Matsuo Takashi"},{"name":"Azuma Masayuki"}],"ja":[{"name":"青田 桂子"},{"name":"山村 佳子"},{"name":"山ノ井 朋子"},{"name":"武川 大輔"},{"name":"可児 耕一"},{"name":"高野 栄之"},{"name":"桃田 幸弘"},{"name":"松本 文博"},{"name":"菅原 千恵子"},{"name":"吉岡 昌美"},{"name":"河野 文昭"},{"name":"松尾 敬志"},{"name":"東 雅之"}]},"description":{"en":"It has been shown that oral hygiene affects the onset of perioperative complications. The usefulness of perioperative oral function management aiming at the outbreak decrease in treatment complications and an early discharge was recognized. As a result, perioperative oral function management fee was founded at revision of medical service fees in Fiscal year 2012. In this clinical study, we evaluated the implementation of perioperative oral function management in Tokushima University Hospital. We examined 781 patients, including 563 patients for surgery and 218 patients for chemotherapy and radiotherapy. The mean age of patients was 58.8 ± 12.4 years old. The implementation rate of perioperative oral function management was 9.7% in the patients of surgery, and 17.4% in those of chemotherapy and radiotherapy. The highly required medical department was neurosurgery in the patients of surgery, and hematology in those of chemotherapy and radiotherapy. The mean number of tooth present was 21.3 ± 7.1 in the patients of surgery, and 19.8 ± 7.2 in those chemotherapy and radiotherapy. The rate of dental treatment was required in 40.5% of total patients who received surgery, and in 51.4% of patients who received chemotherapy and radiotherapy.The rate of patients who received denture treatment attained to 11.9% of the whole patients receiving surgery, and 13.3% of patients receiving chemotherapy and radiotherapy. It was revealed that there were many patients required potential demands in perioperative oral function management, and that there were many patients who need dental or denture treatment. We would like to develop perioperative oral function management by the interprofessional collaboration in health and social care.7月発行だが，6月号である．","ja":"It has been shown that oral hygiene affects the onset of perioperative complications. The usefulness of perioperative oral function management aiming at the outbreak decrease in treatment complications and an early discharge was recognized. As a result, perioperative oral function management fee was founded at revision of medical service fees in Fiscal year 2012. In this clinical study, we evaluated the implementation of perioperative oral function management in Tokushima University Hospital. We examined 781 patients, including 563 patients for surgery and 218 patients for chemotherapy and radiotherapy. The mean age of patients was 58.8 ± 12.4 years old. The implementation rate of perioperative oral function management was 9.7% in the patients of surgery, and 17.4% in those of chemotherapy and radiotherapy. The highly required medical department was neurosurgery in the patients of surgery, and hematology in those of chemotherapy and radiotherapy. The mean number of tooth present was 21.3 ± 7.1 in the patients of surgery, and 19.8 ± 7.2 in those chemotherapy and radiotherapy. The rate of dental treatment was required in 40.5% of total patients who received surgery, and in 51.4% of patients who received chemotherapy and radiotherapy.The rate of patients who received denture treatment attained to 11.9% of the whole patients receiving surgery, and 13.3% of patients receiving chemotherapy and radiotherapy. It was revealed that there were many patients required potential demands in perioperative oral function management, and that there were many patients who need dental or denture treatment. We would like to develop perioperative oral function management by the interprofessional collaboration in health and social care.7月発行だが，6月号である．"},"publication_date":"2015-01","publication_name":{"en":"Journal of Oral Health and Biosciences","ja":"Journal of Oral Health and Biosciences"},"volume":"Vol.28","number":"No.1","starting_page":"29","ending_page":"36","languages":["jpn"],"referee":true,"identifiers":{"issn":["2188-7888"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:8, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970606"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=297988","label":"url"}],"paper_title":{"en":"徳島大学病院脳卒中センターにおける口腔管理の意義について","ja":"徳島大学病院脳卒中センターにおける口腔管理の意義について"},"authors":{"en":[{"name":"Yamamura Yoshiko"},{"name":"Aota Keiko"},{"name":"Takegawa Daisuke"},{"name":"十川 悠香"},{"name":"近藤 智香"},{"name":"山ノ井 朋子"},{"name":"Takano Hideyuki"},{"name":"Kani Kohichi"},{"name":"Matsumoto Fumihiro"},{"name":"Momota Yukihiro"},{"name":"Satomi Junichiro"},{"name":"Kawano Fumiaki"},{"name":"Matsuo Takashi"},{"name":"Nagahiro Shinji"},{"name":"Azuma Masayuki"}],"ja":[{"name":"山村 佳子"},{"name":"青田 桂子"},{"name":"武川 大輔"},{"name":"十川 悠香"},{"name":"近藤 智香"},{"name":"山ノ井 朋子"},{"name":"高野 栄之"},{"name":"可児 耕一"},{"name":"松本 文博"},{"name":"桃田 幸弘"},{"name":"里見 淳一郎"},{"name":"河野 文昭"},{"name":"松尾 敬志"},{"name":"永廣 信治"},{"name":"東 雅之"}]},"publication_date":"2015","publication_name":{"en":"Journal of Oral Health and Biosciences","ja":"Journal of Oral Health and Biosciences"},"volume":"Vol.9","number":"No.1","starting_page":"37","ending_page":"41","languages":["jpn"],"referee":true,"identifiers":{"issn":["2188-7888"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:9, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970607"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/25386367","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=287689","label":"url"}],"paper_title":{"en":"High-Wattage Pulsed Irradiation of Linearly Polarized Near-Infrared Light to Stellate Ganglion Area for Burning Mouth Syndrome","ja":"High-Wattage Pulsed Irradiation of Linearly Polarized Near-Infrared Light to Stellate Ganglion Area for Burning Mouth Syndrome"},"authors":{"en":[{"name":"Momota Yukihiro"},{"name":"Kani Kohichi"},{"name":"Takano Hideyuki"},{"name":"Matsumoto Fumihiro"},{"name":"Aota Keiko"},{"name":"Takegawa Daisuke"},{"name":"Tomoko Yamanoi"},{"name":"Chika Kondo"},{"name":"Tomioka Shigemasa"},{"name":"Azuma Masayuki"}],"ja":[{"name":"桃田 幸弘"},{"name":"可児 耕一"},{"name":"高野 栄之"},{"name":"松本 文博"},{"name":"青田 桂子"},{"name":"武川 大輔"},{"name":"Tomoko Yamanoi"},{"name":"Chika Kondo"},{"name":"富岡 重正"},{"name":"東 雅之"}]},"description":{"en":"The purpose of this study was to apply high-wattage pulsed irradiation of linearly polarized near-infrared light to the stellate ganglion area for burning mouth syndrome (BMS) and to assess the efficacy of the stellate ganglion area irradiation (SGR) on BMS using differential time-/frequency-domain parameters (D parameters). Three patients with BMS received high-wattage pulsed SGR; the response to SGR was evaluated by visual analogue scale (VAS) representing the intensity of glossalgia and D parameters used in heart rate variability analysis. High-wattage pulsed SGR significantly decreased the mean value of VAS in all cases without any adverse event such as thermal injury. D parameters mostly correlated with clinical condition of BMS. High-wattage pulsed SGR was safe and effective for the treatment of BMS; D parameters are useful for assessing efficacy of SGR on BMS.","ja":"The purpose of this study was to apply high-wattage pulsed irradiation of linearly polarized near-infrared light to the stellate ganglion area for burning mouth syndrome (BMS) and to assess the efficacy of the stellate ganglion area irradiation (SGR) on BMS using differential time-/frequency-domain parameters (D parameters). Three patients with BMS received high-wattage pulsed SGR; the response to SGR was evaluated by visual analogue scale (VAS) representing the intensity of glossalgia and D parameters used in heart rate variability analysis. High-wattage pulsed SGR significantly decreased the mean value of VAS in all cases without any adverse event such as thermal injury. D parameters mostly correlated with clinical condition of BMS. High-wattage pulsed SGR was safe and effective for the treatment of BMS; D parameters are useful for assessing efficacy of SGR on BMS."},"publication_date":"2014-10-19","publication_name":{"en":"Case Reports in Dentistry","ja":"Case Reports in Dentistry"},"languages":["eng"],"referee":true,"identifiers":{"doi":["10.1155/2014/171657"],"issn":["2090-6447"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:10, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970608"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/25146019","label":"url"},{"@id":"https://www.scopus.com/record/display.url?eid=2-s2.0-85027943562&origin=inward","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=300752","label":"url"}],"paper_title":{"en":"Modulatory roles of interferon-γ through indoleamine 2, 3-dioxygenase induction in innate immune response of dental pulp cells.","ja":"Modulatory roles of interferon-γ through indoleamine 2, 3-dioxygenase induction in innate immune response of dental pulp cells."},"authors":{"en":[{"name":"Takegawa Daisuke"},{"name":"Nakanishi Tadashi"},{"name":"Hirao Kouji"},{"name":"Yumoto Hiromichi"},{"name":"Takahashi Kanako"},{"name":"Matsuo Takashi"}],"ja":[{"name":"武川 大輔"},{"name":"中西 正"},{"name":"平尾 功治"},{"name":"湯本 浩通"},{"name":"高橋 加奈子"},{"name":"松尾 敬志"}]},"description":{"en":"Marked infiltration of inflammatory cells such as activated T cells producing interferon-γ (IFN-γ) is observed in severe pulpitis. However, the roles of IFN-γ in the innate immune response of dental pulp have not been reported. Indoleamine 2, 3-dioxygenase (IDO) is a regulator of immune responses, and the IDO expression is induced by IFN-γ in many cells whose expression in dental pulp is unknown. The purpose of this study was to determine the role of IFN-γ in the immune response through microbial pattern recognition receptors (PRRs) such as Toll-like receptors or nucleotide-binding oligomerization domain-like receptors on the production of proinflammatory cytokines such as CXCL10 and interleukin (IL)-6 and the expression of IDO in cultured human dental pulp cells (HDPCs). HDPCs were established from explant cultures of healthy pulp tissues. CXCL10 and IL-6 production was determined using enzyme-linked immunosorbent assay. Confirmation of IDO localization in dental pulp tissues was examined using immunohistochemistry. IDO expression in HDPCs was analyzed by immunoblot. IFN-γ significantly up-regulated CXCL10 and IL-6 production in the HDPCs stimulated with ligands for PRRs in a concentration-dependent manner. The expression of IDO was detected in inflamed pulp tissue. In addition, IFN-γ in combination with the PRR ligands enhanced IDO expression in HDPCs compared with IFN-γ alone. Moreover, CXCL10 production in IFN-γ-stimulated HDPCs was inhibited by an IDO inhibitor. This study showed the synergistic effects by IFN-γ on cytokine production and IDO expression in HDPCs, suggesting that IFN-γ may modulate the innate immune response of dental pulp.","ja":"Marked infiltration of inflammatory cells such as activated T cells producing interferon-γ (IFN-γ) is observed in severe pulpitis. However, the roles of IFN-γ in the innate immune response of dental pulp have not been reported. Indoleamine 2, 3-dioxygenase (IDO) is a regulator of immune responses, and the IDO expression is induced by IFN-γ in many cells whose expression in dental pulp is unknown. The purpose of this study was to determine the role of IFN-γ in the immune response through microbial pattern recognition receptors (PRRs) such as Toll-like receptors or nucleotide-binding oligomerization domain-like receptors on the production of proinflammatory cytokines such as CXCL10 and interleukin (IL)-6 and the expression of IDO in cultured human dental pulp cells (HDPCs). HDPCs were established from explant cultures of healthy pulp tissues. CXCL10 and IL-6 production was determined using enzyme-linked immunosorbent assay. Confirmation of IDO localization in dental pulp tissues was examined using immunohistochemistry. IDO expression in HDPCs was analyzed by immunoblot. IFN-γ significantly up-regulated CXCL10 and IL-6 production in the HDPCs stimulated with ligands for PRRs in a concentration-dependent manner. The expression of IDO was detected in inflamed pulp tissue. In addition, IFN-γ in combination with the PRR ligands enhanced IDO expression in HDPCs compared with IFN-γ alone. Moreover, CXCL10 production in IFN-γ-stimulated HDPCs was inhibited by an IDO inhibitor. This study showed the synergistic effects by IFN-γ on cytokine production and IDO expression in HDPCs, suggesting that IFN-γ may modulate the innate immune response of dental pulp."},"publication_date":"2014-05-09","publication_name":{"en":"Journal of Endodontics","ja":"Journal of Endodontics"},"volume":"Vol.40","number":"No.9","starting_page":"1382","ending_page":"1387","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1016/j.joen.2014.03.018"],"issn":["1878-3554"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:11, {"insert":{"user_id":"R000055824","type":"published_papers","id":"42970609"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=287692","label":"url"}],"paper_title":{"en":"一次性舌痛症における手掌部発汗の発現-皮膚水分量測定の有用性について-","ja":"一次性舌痛症における手掌部発汗の発現-皮膚水分量測定の有用性について-"},"authors":{"en":[{"name":"Momota Yukihiro"},{"name":"Kani Kohichi"},{"name":"Takano Hideyuki"},{"name":"Matsumoto Fumihiro"},{"name":"Aota Keiko"},{"name":"Takegawa Daisuke"},{"name":"山ノ井 朋子"},{"name":"近藤 智香"},{"name":"Azuma Masayuki"}],"ja":[{"name":"桃田 幸弘"},{"name":"可児 耕一"},{"name":"高野 栄之"},{"name":"松本 文博"},{"name":"青田 桂子"},{"name":"武川 大輔"},{"name":"山ノ井 朋子"},{"name":"近藤 智香"},{"name":"東 雅之"}]},"publication_date":"2014","publication_name":{"en":"日本歯科人間ドック学会誌","ja":"日本歯科人間ドック学会誌"},"volume":"Vol.9","starting_page":"18","ending_page":"23","languages":["jpn"],"referee":true,"published_paper_type":"scientific_journal"},"priority":"input_data"}
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==== begin registerFile(/WWW/pub2/data/ERD/person/246736/researchmap/misc.jsonl) ====
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line:2, {"insert":{"user_id":"R000055824","type":"misc","id":"42970557"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1520575444014801664/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=392830","label":"url"}],"paper_title":{"en":"デジタル技術を融合した次世代CR修復 : クオリティの高い修復を効率化するDigitally-guided Composite Injection Technique(11)バイレイヤー(2層)クリアインデックスを用いた下顎正中離開の修復","ja":"デジタル技術を融合した次世代CR修復 : クオリティの高い修復を効率化するDigitally-guided Composite Injection Technique(11)バイレイヤー(2層)クリアインデックスを用いた下顎正中離開の修復"},"authors":{"en":[{"name":"Takegawa Daisuke"},{"name":"Kamoi Kouhei"},{"name":"Hosaka Keiichi"}],"ja":[{"name":"武川 大輔"},{"name":"鴨居 浩平"},{"name":"保坂 啓一"}]},"publication_date":"2022-11","publication_name":{"en":"The Nippon Dental Review","ja":"日本歯科評論"},"volume":"Vol.82","number":"No.11","starting_page":"10","ending_page":"13","languages":["jpn"],"identifiers":{"issn":["0289-0909"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
line:3, {"insert":{"user_id":"R000055824","type":"misc","id":"42970558"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/112960","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/30128058","label":"url"},{"@id":"https://www.scopus.com/record/display.url?eid=2-s2.0-85044379499&origin=inward","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=343607","label":"url"}],"paper_title":{"en":"The Roles of Odontoblasts in Dental Pulp Innate Immunity","ja":"The Roles of Odontoblasts in Dental Pulp Innate Immunity"},"authors":{"en":[{"name":"Yumoto Hiromichi"},{"name":"Hirao Kouji"},{"name":"Hosokawa Yuki"},{"name":"Kuramoto Hitomi"},{"name":"Takegawa Daisuke"},{"name":"Nakanishi Tadashi"},{"name":"Matsuo Takashi"}],"ja":[{"name":"湯本 浩通"},{"name":"平尾 功治"},{"name":"細川 由樹"},{"name":"蔵本 瞳"},{"name":"武川 大輔"},{"name":"中西 正"},{"name":"松尾 敬志"}]},"description":{"en":"Odontoblasts located in the outermost layer of dental pulp form a natural barrier between mineralized tissues, dentin, and soft tissues, dental pulp, of the vital tooth, and they first recognize caries-related pathogens and sense external irritations. Therefore, odontoblasts possess a specialized innate immune system to fight oral pathogens invading into dentin. Generally, the rapid initial sensing of microbial pathogens, especially pathogen-associated molecular patterns (PAMPs) shared by microorganisms, are mediated by pattern recognition receptors (PRRs), such as Toll-like receptor and the nucleotide-binding oligomerization domain (NOD). The innate immune responses in odontoblasts initiated by sensing oral pathogens provide host protective events, such as inflammatory reactions, to produce a variety of pro-inflammatory mediators, including chemokines and cytokines. These attract various inflammatory cells and cause antibacterial reactions, such as the production of defensins, to kill microorganisms in the proximal region of the odontoblast layer. This review focuses on innate immunity, especially cellular and molecular mechanisms regarding the sensing of PAMPs from oral pathogens by PRRs, in odontoblasts and provides information for future studies for the development of novel therapeutic strategies, including diagnosis and treatment, to prevent exceeding dental pulp inflammation and preserve the dental pulp tissues.","ja":"Odontoblasts located in the outermost layer of dental pulp form a natural barrier between mineralized tissues, dentin, and soft tissues, dental pulp, of the vital tooth, and they first recognize caries-related pathogens and sense external irritations. Therefore, odontoblasts possess a specialized innate immune system to fight oral pathogens invading into dentin. Generally, the rapid initial sensing of microbial pathogens, especially pathogen-associated molecular patterns (PAMPs) shared by microorganisms, are mediated by pattern recognition receptors (PRRs), such as Toll-like receptor and the nucleotide-binding oligomerization domain (NOD). The innate immune responses in odontoblasts initiated by sensing oral pathogens provide host protective events, such as inflammatory reactions, to produce a variety of pro-inflammatory mediators, including chemokines and cytokines. These attract various inflammatory cells and cause antibacterial reactions, such as the production of defensins, to kill microorganisms in the proximal region of the odontoblast layer. This review focuses on innate immunity, especially cellular and molecular mechanisms regarding the sensing of PAMPs from oral pathogens by PRRs, in odontoblasts and provides information for future studies for the development of novel therapeutic strategies, including diagnosis and treatment, to prevent exceeding dental pulp inflammation and preserve the dental pulp tissues."},"publication_date":"2018-08","publication_name":{"en":"Japanese Dental Science Review","ja":"Japanese Dental Science Review"},"volume":"Vol.54","number":"No.3","starting_page":"105","ending_page":"117","languages":["eng"],"invited":true,"identifiers":{"doi":["10.1016/j.jdsr.2018.03.001"],"issn":["1882-7616"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
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==== end registerFile(/WWW/pub2/data/ERD/person/246736/researchmap/misc.jsonl, KpDwEY8BzHGxATo5KDCb) ====
==== begin registerFile(/WWW/pub2/data/ERD/person/246736/researchmap/presentations.jsonl) ====
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line:2, {"insert":{"user_id":"R000055824","type":"presentations","id":"42970569"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=327514","label":"url"}],"presentation_title":{"en":"Interferon-gamma modulates the innate immune response in odontoblast-like cells","ja":"Interferon-gamma modulates the innate immune response in odontoblast-like cells"},"presenters":{"en":[{"name":"Takegawa Daisuke"},{"name":"Nakanishi Tadashi"},{"name":"Hirao Kouji"},{"name":"Yumoto Hiromichi"},{"name":"Hosokawa Yuki"},{"name":"Matsuo Takashi"}],"ja":[{"name":"武川 大輔"},{"name":"中西 正"},{"name":"平尾 功治"},{"name":"湯本 浩通"},{"name":"細川 由樹"},{"name":"松尾 敬志"}]},"event":{"en":"95th General session and exhibition of the IADR","ja":"95th General session and exhibition of the IADR"},"publication_date":"2017-03-24","languages":["eng"],"is_international_presentation":true},"priority":"input_data"}
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