=== Generating (published_papers) ===
=== Generating (teaching_experience) ===
=== Generating (misc) ===
=== Generating (research_projects) ===
=== Generating (books_etc) ===
=== Generating (committee_memberships) ===
=== Generating (association_memberships) ===
=== Generating (presentations) ===
==== begin registerFile(/WWW/pub2/data/ERD/person/375084/researchmap/published_papers.jsonl) ====
line:1, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47538997"},"force":{"see_also":[{"@id":"https://rdcu.be/dNRlJ","label":"url"},{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119491","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/39009630","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1050019590267929472/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=409256","label":"url"}],"paper_title":{"en":"Quantitative evaluation of 67Gacitrate scintigraphy in the management of nephritis","ja":"Quantitative evaluation of 67Gacitrate scintigraphy in the management of nephritis"},"authors":{"en":[{"name":"Noritake MATSUDA"},{"name":"Otsuka Hideki"},{"name":"Kasai Ryosuke"},{"name":"Otani Tamaki"},{"name":"LOCSIN CHRISTINE ANNE LEAH BOLLOS"},{"name":"Azane Shota"},{"name":"Kunikane Yamato"},{"name":"Otomi Youichi"},{"name":"Yuya UEKI"},{"name":"Okabe Mana"},{"name":"Amano Masafumi"},{"name":"Tamaki Masanori"},{"name":"Wakino Shu"},{"name":"Takao Shoichiro"},{"name":"Harada Masafumi"}],"ja":[{"name":"松田 憲武"},{"name":"大塚 秀樹"},{"name":"笠井 亮佑"},{"name":"大谷 環樹"},{"name":"Leah"},{"name":"阿實 翔太"},{"name":"国金 大和"},{"name":"音見 暢一"},{"name":"植木 勇弥"},{"name":"岡部 真菜"},{"name":"天野 雅史"},{"name":"田蒔 昌憲"},{"name":"𦚰野 修"},{"name":"髙尾 正一郎"},{"name":"原田 雅史"}]},"description":{"en":"In Ga-citrate scintigraphy (Ga-S), visual assessment is used by evaluating renal-uptake comparison with liver and spine and is simple and objective. We adopted the standardized uptake value (SUV) for Ga-citrate and proposed two quantitative indices, active nephritis volume (ANV) and total nephritis uptake (TNU). This study clarified the utility of new Ga-S-based quantitative indices in nephritis management. Before SUV measurement, the Becquerel calibration factor of Ga-citrate was obtained using a phantom experiment. Seventy patients who underwent SPECT/CT imaging were studied. SUV, ANV, and TNU were calculated using a quantitative analysis software for bone SPECT. SUV, ANV, and TNU were analyzed using the (1) threshold method (set 40%) and constant-value method for (2) vertebral SUV, and (3) vertebral SUV. ROC analysis was used to evaluate SUV, ANV, and TNU diagnostic abilities to distinguish nephritis presence and absence as well as interstitial nephritis (IN) and non-IN. The area under the curve (AUC) for nephritis presence or absence had a good value (0.80) for SUV (1), ANV (3), and TNU (3). The AUC for differentiation between IN and non-IN groups had a good value (0.80) for SUV (1). Thus, the new Ga-S-based quantitative indices were useful to evaluate nephritis and distinguish IN and non-IN.","ja":"In Ga-citrate scintigraphy (Ga-S), visual assessment is used by evaluating renal-uptake comparison with liver and spine and is simple and objective. We adopted the standardized uptake value (SUV) for Ga-citrate and proposed two quantitative indices, active nephritis volume (ANV) and total nephritis uptake (TNU). This study clarified the utility of new Ga-S-based quantitative indices in nephritis management. Before SUV measurement, the Becquerel calibration factor of Ga-citrate was obtained using a phantom experiment. Seventy patients who underwent SPECT/CT imaging were studied. SUV, ANV, and TNU were calculated using a quantitative analysis software for bone SPECT. SUV, ANV, and TNU were analyzed using the (1) threshold method (set 40%) and constant-value method for (2) vertebral SUV, and (3) vertebral SUV. ROC analysis was used to evaluate SUV, ANV, and TNU diagnostic abilities to distinguish nephritis presence and absence as well as interstitial nephritis (IN) and non-IN. The area under the curve (AUC) for nephritis presence or absence had a good value (0.80) for SUV (1), ANV (3), and TNU (3). The AUC for differentiation between IN and non-IN groups had a good value (0.80) for SUV (1). Thus, the new Ga-S-based quantitative indices were useful to evaluate nephritis and distinguish IN and non-IN."},"publication_date":"2024-07-04","publication_name":{"en":"Scientific Reports","ja":"Scientific Reports"},"volume":"Vol.14","number":"No.16313","starting_page":"16313","ending_page":"16313","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1038/s41598-024-66823-2"],"issn":["2045-2322"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:2, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47538998"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119529","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38928090","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410124","label":"url"}],"paper_title":{"en":"Deficiency Causes Mitoribosome Excess in Diabetic Nephropathy Mediated by Transcriptional Repressor HIC1.","ja":"Deficiency Causes Mitoribosome Excess in Diabetic Nephropathy Mediated by Transcriptional Repressor HIC1."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Sakamaki Yusuke"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"Sakamaki Yusuke"},{"name":"𦚰野 修"}]},"description":{"en":"overexpression preserved mitoribosomal function, suggesting its protective role in DN.","ja":"overexpression preserved mitoribosomal function, suggesting its protective role in DN."},"publication_date":"2024-06-09","publication_name":{"en":"International Journal of Molecular Sciences","ja":"International Journal of Molecular Sciences"},"volume":"Vol.25","number":"No.12","languages":["eng"],"referee":true,"identifiers":{"doi":["10.3390/ijms25126384"],"issn":["1422-0067"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:3, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47538999"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119528","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38587753","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410122","label":"url"}],"paper_title":{"en":"Ability of NAD and Sirt1 to epigenetically suppress albuminuria.","ja":"Ability of NAD and Sirt1 to epigenetically suppress albuminuria."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"},{"name":"Inagaki Taizo"},{"name":"Minato Masanori"},{"name":"Yamaguchi Sumiyo"},{"name":"Shimizu Ikuko"},{"name":"Miyakami Shinji"},{"name":"Tada Miho"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"},{"name":"稲垣 太造"},{"name":"湊 将典"},{"name":"Yamaguchi Sumiyo"},{"name":"清水 郁子"},{"name":"Miyakami Shinji"},{"name":"多田 美穂"},{"name":"𦚰野 修"}]},"description":{"en":"The time for diabetic nephropathy (DN) to progress from mild to severe is long. Thus, methods to continuously repress DN are required to exert long-lasting effects mediated through epigenetic regulation. In this study, we demonstrated the ability of nicotinamide adenine dinucleotide (NAD) and its metabolites to reduce albuminuria through Sirt1- or Nampt-dependent epigenetic regulation. We previously reported that proximal tubular Sirt1 was lowered before glomerular Sirt1. Repressed glomerular Sirt1 was found to epigenetically elevate Claudin-1. In addition, we reported that proximal tubular Nampt deficiency epigenetically augmented TIMP-1 levels in Sirt6-mediated pathways, leading to type-IV collagen deposition and diabetic fibrosis. Altogether, we propose that the Sirt1/Claudin-1 axis may be crucial in the onset of albuminuria at the early stages of DN and that the Nampt/Sirt6/TIMP-1 axis promotes diabetic fibrosis in the middle to late stages of DN. Finally, administration of NMN, an NAD precursor, epigenetically potentiates the regression of the onset of DN to maintain Sirt1 and repress Claudin-1 in podocytes, suggesting the potential use of NAD metabolites as epigenetic medications for DN.","ja":"The time for diabetic nephropathy (DN) to progress from mild to severe is long. Thus, methods to continuously repress DN are required to exert long-lasting effects mediated through epigenetic regulation. In this study, we demonstrated the ability of nicotinamide adenine dinucleotide (NAD) and its metabolites to reduce albuminuria through Sirt1- or Nampt-dependent epigenetic regulation. We previously reported that proximal tubular Sirt1 was lowered before glomerular Sirt1. Repressed glomerular Sirt1 was found to epigenetically elevate Claudin-1. In addition, we reported that proximal tubular Nampt deficiency epigenetically augmented TIMP-1 levels in Sirt6-mediated pathways, leading to type-IV collagen deposition and diabetic fibrosis. Altogether, we propose that the Sirt1/Claudin-1 axis may be crucial in the onset of albuminuria at the early stages of DN and that the Nampt/Sirt6/TIMP-1 axis promotes diabetic fibrosis in the middle to late stages of DN. Finally, administration of NMN, an NAD precursor, epigenetically potentiates the regression of the onset of DN to maintain Sirt1 and repress Claudin-1 in podocytes, suggesting the potential use of NAD metabolites as epigenetic medications for DN."},"publication_date":"2024-04-08","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"volume":"Vol.28","number":"No.7","starting_page":"599","ending_page":"607","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-024-02502-w"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:4, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539000"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38467892","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411328","label":"url"}],"paper_title":{"en":"Impact of dietary habits on renal function in Saku, a rural Japanese town: a cohort study.","ja":"Impact of dietary habits on renal function in Saku, a rural Japanese town: a cohort study."},"authors":{"en":[{"name":"Adachi Keika"},{"name":"Yasuda Marie"},{"name":"Ida Makiko"},{"name":"Kanda Takeshi"},{"name":"Morita Akemi"},{"name":"Wakino Shu"},{"name":"Watanabe Shaw"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Adachi Keika"},{"name":"Yasuda Marie"},{"name":"Ida Makiko"},{"name":"Kanda Takeshi"},{"name":"Morita Akemi"},{"name":"𦚰野 修"},{"name":"Watanabe Shaw"},{"name":"Itoh Hiroshi"}]},"description":{"en":"High protein intake leads to a decline in renal function in the advanced stages of chronic kidney disease (CKD). An effective diet for maintaining renal function in healthy individuals or patients in the early stages of CKD has not been established. This cohort study was conducted in Saku, Nagano Prefecture, Japan, to investigate the impact of dietary habits on renal function. In this cross-sectional cohort study, we used the Saku Control Obesity Program (UMIN000016892), including 4,446 participants who submitted a brief-type self-administered diet history questionnaire and underwent routine physical examination. The amount of food intake was divided into quartiles. After adjusting for age and sex, multivariate logistic regression analysis was used to calculate the odds ratio (OR) for the risk of developing CKD (estimated glomerular filtration rate [eGFR] < 60 mL/min/1.73 m). In total, 3,899 participants were analyzed. The overall prevalence of patients with eGFR < 60 mL/min/1.73 m was 11% (n = 434, male; 7.1%, female; 4.1%). The groups with a high intake of chicken (approximately 63.4 g/day, adjusted OR: 0.632, P = 0.003), natto (fermented bean; approximately 21.7 g/day, adjusted OR: 0.679, P = 0.01), and plant protein (approximately 0.8 g/ideal body weight/day, adjusted OR: 0.695, P = 0.042) showed a low risk of developing CKD compared to the group with the lowest intake. Our cross-sectional study showed that the intake of chicken meat, natto, and plant protein was associated with high eGFR levels. This information can be of value for preventing CKD incidence in healthy Japanese individuals.","ja":"High protein intake leads to a decline in renal function in the advanced stages of chronic kidney disease (CKD). An effective diet for maintaining renal function in healthy individuals or patients in the early stages of CKD has not been established. This cohort study was conducted in Saku, Nagano Prefecture, Japan, to investigate the impact of dietary habits on renal function. In this cross-sectional cohort study, we used the Saku Control Obesity Program (UMIN000016892), including 4,446 participants who submitted a brief-type self-administered diet history questionnaire and underwent routine physical examination. The amount of food intake was divided into quartiles. After adjusting for age and sex, multivariate logistic regression analysis was used to calculate the odds ratio (OR) for the risk of developing CKD (estimated glomerular filtration rate [eGFR] < 60 mL/min/1.73 m). In total, 3,899 participants were analyzed. The overall prevalence of patients with eGFR < 60 mL/min/1.73 m was 11% (n = 434, male; 7.1%, female; 4.1%). The groups with a high intake of chicken (approximately 63.4 g/day, adjusted OR: 0.632, P = 0.003), natto (fermented bean; approximately 21.7 g/day, adjusted OR: 0.679, P = 0.01), and plant protein (approximately 0.8 g/ideal body weight/day, adjusted OR: 0.695, P = 0.042) showed a low risk of developing CKD compared to the group with the lowest intake. Our cross-sectional study showed that the intake of chicken meat, natto, and plant protein was associated with high eGFR levels. This information can be of value for preventing CKD incidence in healthy Japanese individuals."},"publication_date":"2024-03-12","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"volume":"Vol.28","number":"No.8","starting_page":"751","ending_page":"763","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-024-02479-6"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:5, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539001"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119548","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38454797","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411330","label":"url"}],"paper_title":{"en":"Prospective Multicenter Registry-Based Study on Thyroid Storm: The Guidelines for the Management from Japan are Useful.","ja":"Prospective Multicenter Registry-Based Study on Thyroid Storm: The Guidelines for the Management from Japan are Useful."},"authors":{"en":[{"name":"Furukawa Yasushi"},{"name":"Tanaka Keiko"},{"name":"Isozaki Osamu"},{"name":"Suzuki Atsushi"},{"name":"Iburi Tadao"},{"name":"Tsuboi Kumiko"},{"name":"Iguchi Moritake"},{"name":"Kanamoto Naotetsu"},{"name":"Minamitani Kanshi"},{"name":"Wakino Shu"},{"name":"Satoh Tetsurou"},{"name":"Teramukai Satoshi"},{"name":"Kimura Eizen"},{"name":"Miyake Yoshihiro"},{"name":"Akamizu Takashi"}],"ja":[{"name":"Furukawa Yasushi"},{"name":"Tanaka Keiko"},{"name":"Isozaki Osamu"},{"name":"Suzuki Atsushi"},{"name":"Iburi Tadao"},{"name":"Tsuboi Kumiko"},{"name":"Iguchi Moritake"},{"name":"Kanamoto Naotetsu"},{"name":"Minamitani Kanshi"},{"name":"𦚰野 修"},{"name":"Satoh Tetsurou"},{"name":"Teramukai Satoshi"},{"name":"Kimura Eizen"},{"name":"Miyake Yoshihiro"},{"name":"Akamizu Takashi"}]},"description":{"en":"The mortality rate in thyroid storm (TS) has been reported to be higher than 10%. We aimed to evaluate the effectiveness of the 2016 guidelines for the management of TS proposed by the Japan Thyroid Association and Japan Endocrine Society. Prospective registry-based study through a secure web platform. Prospective multicenter registry. Patients with new-onset TS were registered in the Research Electronic Data Capture (REDCap). On day 30 after admission, clinical information and prognosis of each patient were added to the platform. On day 180, the prognosis was described. This study included 110 patients with TS. The median of Acute Physiology and Chronic Health Evaluation (APACHE) II score was 13, higher than the score in the previous nationwide epidemiological study, 10 (p = 0.001). Nonetheless, the mortality rate at day 30 was 5.5%, approximately half compared with 10.7% in the previous nationwide survey. Lower body mass index, shock and lower left ventricular ejection fraction were positively associated with poor prognosis at day 30, while the lack of fever ≥ 38℃ was related to the outcome. The mortality rate in patients with an APACHE II score ≥12 for whom the guidelines were not followed was significantly higher than the rate in patients for whom the guidelines were followed (50% vs. 4.7%) (p = 0.01). Prognosis seemed better than in the previous nationwide survey, even though disease severity was higher. The mortality rate was lower when the guidelines were followed. Thus, the guidelines are useful for managing TS.","ja":"The mortality rate in thyroid storm (TS) has been reported to be higher than 10%. We aimed to evaluate the effectiveness of the 2016 guidelines for the management of TS proposed by the Japan Thyroid Association and Japan Endocrine Society. Prospective registry-based study through a secure web platform. Prospective multicenter registry. Patients with new-onset TS were registered in the Research Electronic Data Capture (REDCap). On day 30 after admission, clinical information and prognosis of each patient were added to the platform. On day 180, the prognosis was described. This study included 110 patients with TS. The median of Acute Physiology and Chronic Health Evaluation (APACHE) II score was 13, higher than the score in the previous nationwide epidemiological study, 10 (p = 0.001). Nonetheless, the mortality rate at day 30 was 5.5%, approximately half compared with 10.7% in the previous nationwide survey. Lower body mass index, shock and lower left ventricular ejection fraction were positively associated with poor prognosis at day 30, while the lack of fever ≥ 38℃ was related to the outcome. The mortality rate in patients with an APACHE II score ≥12 for whom the guidelines were not followed was significantly higher than the rate in patients for whom the guidelines were followed (50% vs. 4.7%) (p = 0.01). Prognosis seemed better than in the previous nationwide survey, even though disease severity was higher. The mortality rate was lower when the guidelines were followed. Thus, the guidelines are useful for managing TS."},"publication_date":"2024-03-08","publication_name":{"en":"The Journal of Clinical Endocrinology and Metabolism","ja":"The Journal of Clinical Endocrinology and Metabolism"},"languages":["eng"],"referee":true,"identifiers":{"doi":["10.1210/clinem/dgae124"],"issn":["1945-7197"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:6, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539002"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38267800","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411331","label":"url"}],"paper_title":{"en":"Rationale and design of the Japanese Biomarkers in Nephrotic Syndrome (J-MARINE) study.","ja":"Rationale and design of the Japanese Biomarkers in Nephrotic Syndrome (J-MARINE) study."},"authors":{"en":[{"name":"Kurasawa Shimon"},{"name":"Kato Sawako"},{"name":"Ozeki Takaya"},{"name":"Akiyama Shin'ichi"},{"name":"Ishimoto Takuji"},{"name":"Mizuno Masashi"},{"name":"Tsuboi Naotake"},{"name":"Kato Noritoshi"},{"name":"Wakino Shu"},{"name":"Maruyama Shoichi"}],"ja":[{"name":"Kurasawa Shimon"},{"name":"Kato Sawako"},{"name":"Ozeki Takaya"},{"name":"Akiyama Shin'ichi"},{"name":"Ishimoto Takuji"},{"name":"Mizuno Masashi"},{"name":"Tsuboi Naotake"},{"name":"Kato Noritoshi"},{"name":"𦚰野 修"},{"name":"Maruyama Shoichi"}]},"description":{"en":"This study will provide valuable insights into biomarkers for NS and serve as a biorepository for future studies.","ja":"The Japanese Biomarkers in Nephrotic Syndrome (J-MARINE) study is a nationwide, multicenter, and prospective cohort study in Japan, enrolling adult (≥18 years) patients with minimal change disease (MCD), focal segmental glomerulosclerosis (FSGS), membranous nephropathy (MN), membranoproliferative glomerulonephritis (MPGN), C3 glomerulopathy (C3G), and lupus nephritis (LN). Baseline clinical information and plasma and urine samples will be collected at the time of immunosuppressive therapy initiation or biopsy. Follow-up data and plasma and urine samples will be collected longitudinally based on the designated protocols. Candidate biomarkers will be measured: CD80, cytotoxic T-lymphocyte antigen 4, and soluble urokinase plasminogen activator receptor for MCD and FSGS; anti-phospholipase A2 receptor and thrombospondin type-1 domain-containing protein 7A antibodies for MN; fragment Ba, C3a, factor I, and properdin for MPGN/C3G; and CD11b, CD16b, and CD163 for LN. Outcomes include complete and partial remission, relapse of proteinuria, a 30% reduction in estimated glomerular filtration rate (eGFR), eGFR decline, and initiation of renal replacement therapy. The diagnostic accuracy and predictive ability for clinical outcomes will be assessed for each biomarker."},"publication_date":"2024-01-25","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"volume":"Vol.28","number":"No.5","starting_page":"431","ending_page":"439","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-023-02449-4"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:7, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539003"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/37490135","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400893","label":"url"}],"paper_title":{"en":"N-methyl-2-pyridone-5-carboxamide (N-Me-2PY) has potent anti-fibrotic and anti-inflammatory activity in a fibrotic kidney model: is it an old uremic toxin?","ja":"N-methyl-2-pyridone-5-carboxamide (N-Me-2PY) has potent anti-fibrotic and anti-inflammatory activity in a fibrotic kidney model: is it an old uremic toxin?"},"authors":{"en":[{"name":"Yoshimura Norito"},{"name":"Yamada Katsutoshi"},{"name":"Ono Takashi"},{"name":"Notoya Mitsuru"},{"name":"Yukioka Hideo"},{"name":"Takahashi Rina"},{"name":"Wakino Shu"},{"name":"Kanda Takeshi"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Yoshimura Norito"},{"name":"Yamada Katsutoshi"},{"name":"Ono Takashi"},{"name":"Notoya Mitsuru"},{"name":"Yukioka Hideo"},{"name":"Takahashi Rina"},{"name":"𦚰野 修"},{"name":"Kanda Takeshi"},{"name":"Itoh Hiroshi"}]},"description":{"en":"N-Me-2PY, N-Me-4PY, and nicotinamide N-oxide (NNO) inhibited TGFβ1-induced fibrosis and inflammatory gene expression in kidney fibroblasts. N-Me-2PY strongly suppressed the expression of types I and III collagen, αSMA, and IL-6. N-Me-2PY also suppressed TGFβ1-induced type I collagen and IL-6 expression in renal tubular epithelial cells. No toxic effect was observed with N-Me-2PY treatment, while attenuating renal fibrosis and tubular dilation in UUO mice. Suppression of various fibrosis- and inflammation-related genes was also observed. N-Me-2PY did not inhibit TGFβ1-induced Smad3 phosphorylation but inhibited Akt phosphorylation, suggesting that N-Me-2PY exerts anti-fibrotic and anti-inflammatory effects through Akt inhibition, similar to pirfenidone.","ja":"NAD + metabolites, such as N-Me-2PY, are not uremic toxins but are potential therapeutic agents that have anti-fibrotic effects in CKD."},"publication_date":"2023-07-25","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-023-02379-1"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:8, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539004"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/37395555","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400894","label":"url"}],"paper_title":{"en":"Efficacy of molnupiravir and sotrovimab in Japanese dialysis patients with COVID-19 in clinical practice during the Omicron (BA.1 and BA.2) pandemic.","ja":"Efficacy of molnupiravir and sotrovimab in Japanese dialysis patients with COVID-19 in clinical practice during the Omicron (BA.1 and BA.2) pandemic."},"authors":{"en":[{"name":"Kikuchi Kan"},{"name":"Nangaku Masaomi"},{"name":"Ryuzaki Munekazu"},{"name":"Yamakawa Tomoyuki"},{"name":"Ota Yoshihiro"},{"name":"Hanafusa Norio"},{"name":"Sakai Ken"},{"name":"Kanno Yoshihiko"},{"name":"Ando Ryoichi"},{"name":"Shinoda Toshio"},{"name":"Wakino Shu"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"},{"name":"Akizawa Tadao"}],"ja":[{"name":"Kikuchi Kan"},{"name":"Nangaku Masaomi"},{"name":"Ryuzaki Munekazu"},{"name":"Yamakawa Tomoyuki"},{"name":"Ota Yoshihiro"},{"name":"Hanafusa Norio"},{"name":"Sakai Ken"},{"name":"Kanno Yoshihiko"},{"name":"Ando Ryoichi"},{"name":"Shinoda Toshio"},{"name":"𦚰野 修"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"},{"name":"Akizawa Tadao"}]},"description":{"en":"Sotrovimab showed efficacy in Omicron BA.1 but attenuated in BA.2. Molnupiravir also showed efficacy in BA.2, suggesting administration of molnupiravir would be important.","ja":"A total of 1480 patients were included. The mortality of the molnupiravir, sotrovimab, and combination groups were significantly improved compared to the control group (p < 0.001). Multivariate analysis indicated that antiviral therapy improves the survival of dialysis patients with COVID-19 (hazard ratio was 0.184 for molnupiravir, 0.389 for sotrovimab, and 0.254 for combination groups, respectively)."},"publication_date":"2023-07-03","publication_name":{"en":"Therapeutic Apheresis and Dialysis","ja":"Therapeutic Apheresis and Dialysis"},"languages":["eng"],"referee":true,"identifiers":{"doi":["10.1111/1744-9987.14033"],"issn":["1744-9987"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:9, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539005"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/37199399","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400706","label":"url"}],"paper_title":{"en":"PCK1 Protects against Mitoribosomal Defects in Diabetic Nephropathy in Mouse Models.","ja":"PCK1 Protects against Mitoribosomal Defects in Diabetic Nephropathy in Mouse Models."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Sakamaki Yusuke"},{"name":"Tamaki Masanori"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"Sakamaki Yusuke"},{"name":"田蒔 昌憲"},{"name":"𦚰野 修"}]},"description":{"en":"PCK1 preserves mitoribosomal function and may play a novel protective role in DN.","ja":"PCK1 preserves mitoribosomal function and may play a novel protective role in DN."},"publication_date":"2023-05-18","publication_name":{"en":"Journal of the American Society of Nephrology","ja":"Journal of the American Society of Nephrology"},"volume":"Vol.34","number":"No.8","starting_page":"1343","ending_page":"1365","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1681/ASN.0000000000000156"],"issn":["1533-3450"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:10, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539006"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118478","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/36992238","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400895","label":"url"}],"paper_title":{"en":"T-Cell Response and Antibody Production Induced by the COVID-19 Booster Vaccine in Japanese Chronic Kidney Disease Patients Treated with Hemodialysis.","ja":"T-Cell Response and Antibody Production Induced by the COVID-19 Booster Vaccine in Japanese Chronic Kidney Disease Patients Treated with Hemodialysis."},"authors":{"en":[{"name":"Yoshifuji Ayumi"},{"name":"Toda Masataro"},{"name":"Ryuzaki Munekazu"},{"name":"Oyama Emi"},{"name":"Kikuchi Kan"},{"name":"Kawai Toru"},{"name":"Sakai Ken"},{"name":"Koinuma Masayoshi"},{"name":"Katayama Kazuhiko"},{"name":"Yokoyama Takashi"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kanno Yoshihiko"},{"name":"Kon Hirofumi"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"Wakino Shu"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}],"ja":[{"name":"Yoshifuji Ayumi"},{"name":"Toda Masataro"},{"name":"Ryuzaki Munekazu"},{"name":"Oyama Emi"},{"name":"Kikuchi Kan"},{"name":"Kawai Toru"},{"name":"Sakai Ken"},{"name":"Koinuma Masayoshi"},{"name":"Katayama Kazuhiko"},{"name":"Yokoyama Takashi"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kanno Yoshihiko"},{"name":"Kon Hirofumi"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"𦚰野 修"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}]},"description":{"en":"COVID test (T-SPOT) were measured prior to, three weeks after, and three months after the booster administration. The HD group had significantly higher SARS-CoV-2 IgG levels and neutralizing antibody titers against the original strain at three weeks and three months after the booster vaccination compared to the control group, albeit the HD group had lower SARS-CoV-2 IgG levels and neutralizing antibody titers before the booster administration. Moreover, the HD group had significantly higher T-SPOT levels at all three time points compared to the control group. The HD group also had significantly higher local and systemic adverse reaction rates than the control group. By booster vaccination, HD patients could acquire more effective SARS-CoV-2-specific humoral and cellular immunity than the control group.","ja":"COVID test (T-SPOT) were measured prior to, three weeks after, and three months after the booster administration. The HD group had significantly higher SARS-CoV-2 IgG levels and neutralizing antibody titers against the original strain at three weeks and three months after the booster vaccination compared to the control group, albeit the HD group had lower SARS-CoV-2 IgG levels and neutralizing antibody titers before the booster administration. Moreover, the HD group had significantly higher T-SPOT levels at all three time points compared to the control group. The HD group also had significantly higher local and systemic adverse reaction rates than the control group. By booster vaccination, HD patients could acquire more effective SARS-CoV-2-specific humoral and cellular immunity than the control group."},"publication_date":"2023-03-14","publication_name":{"en":"Vaccines","ja":"Vaccines"},"volume":"Vol.11","number":"No.3","languages":["eng"],"referee":true,"identifiers":{"doi":["10.3390/vaccines11030653"],"issn":["2076-393X"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:11, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539007"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118479","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/36849798","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400896","label":"url"}],"paper_title":{"en":"Sodium benzoate attenuates 2,8-dihydroxyadenine nephropathy by inhibiting monocyte/macrophage TNF-α expression.","ja":"Sodium benzoate attenuates 2,8-dihydroxyadenine nephropathy by inhibiting monocyte/macrophage TNF-α expression."},"authors":{"en":[{"name":"Oshima Yoichi"},{"name":"Wakino Shu"},{"name":"Kanda Takeshi"},{"name":"Tajima Takaya"},{"name":"Itoh Tomoaki"},{"name":"Uchiyama Kiyotaka"},{"name":"Yoshimoto Keiko"},{"name":"Sasabe Jumpei"},{"name":"Yasui Masato"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Oshima Yoichi"},{"name":"𦚰野 修"},{"name":"Kanda Takeshi"},{"name":"Tajima Takaya"},{"name":"Itoh Tomoaki"},{"name":"Uchiyama Kiyotaka"},{"name":"Yoshimoto Keiko"},{"name":"Sasabe Jumpei"},{"name":"Yasui Masato"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Sodium benzoate (SB), a known D-amino acid oxidase (DAO) enzyme inhibitor, has an anti-inflammatory effect, although its role in renal damage has not been explored. 2,8-dihydroxyadenine crystal induced chronic kidney disease, in which TNF-α is involved in the pathogenesis, was established by oral adenine administration in C57BL/6JJcl mice (AdCKD) with or without SB to investigate its renal protective effects. SB significantly attenuated AdCKD by decreasing serum creatinine and urea nitrogen levels, and kidney interstitial fibrosis and tubular atrophy scores. The survival of AdCKD mice improved 2.6-fold by SB administration. SB significantly decreased the number of infiltrating macrophages observed in the positive F4/80 immunohistochemistry area and reduced the expression of macrophage markers and inflammatory genes, including TNF-α, in the kidneys of AdCKD. Human THP-1 cells stimulated with either lipopolysaccharide or TNF-α showed increased expression of inflammatory genes, although this was significantly reduced by SB, confirming the anti-inflammatory effects of SB. SB exhibited renal protective effects in AdCKD in DAO enzyme deficient mice, suggesting that anti-inflammatory effect of SB was independent of DAO enzyme activity. Moreover, binding to motif DNA sequence, protein level, and mRNA level of NF-κB RelB were significantly inhibited by SB in AdCKD kidneys and lipopolysaccharide treated THP-1 cells, respectively. We report that anti-inflammatory property of SB is independent of DAO enzymatic activity and is associated with down regulated NF-κB RelB as well as its downstream inflammatory genes such as TNF-α in AdCKD.","ja":"Sodium benzoate (SB), a known D-amino acid oxidase (DAO) enzyme inhibitor, has an anti-inflammatory effect, although its role in renal damage has not been explored. 2,8-dihydroxyadenine crystal induced chronic kidney disease, in which TNF-α is involved in the pathogenesis, was established by oral adenine administration in C57BL/6JJcl mice (AdCKD) with or without SB to investigate its renal protective effects. SB significantly attenuated AdCKD by decreasing serum creatinine and urea nitrogen levels, and kidney interstitial fibrosis and tubular atrophy scores. The survival of AdCKD mice improved 2.6-fold by SB administration. SB significantly decreased the number of infiltrating macrophages observed in the positive F4/80 immunohistochemistry area and reduced the expression of macrophage markers and inflammatory genes, including TNF-α, in the kidneys of AdCKD. Human THP-1 cells stimulated with either lipopolysaccharide or TNF-α showed increased expression of inflammatory genes, although this was significantly reduced by SB, confirming the anti-inflammatory effects of SB. SB exhibited renal protective effects in AdCKD in DAO enzyme deficient mice, suggesting that anti-inflammatory effect of SB was independent of DAO enzyme activity. Moreover, binding to motif DNA sequence, protein level, and mRNA level of NF-κB RelB were significantly inhibited by SB in AdCKD kidneys and lipopolysaccharide treated THP-1 cells, respectively. We report that anti-inflammatory property of SB is independent of DAO enzymatic activity and is associated with down regulated NF-κB RelB as well as its downstream inflammatory genes such as TNF-α in AdCKD."},"publication_date":"2023-02-27","publication_name":{"en":"Scientific Reports","ja":"Scientific Reports"},"volume":"Vol.13","number":"No.1","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1038/s41598-023-30056-6"],"issn":["2045-2322"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:12, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539008"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/36611128","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400897","label":"url"}],"paper_title":{"en":"Canagliflozin protects the cardiovascular system through effects on the gut environment in non-diabetic nephrectomized rats.","ja":"Canagliflozin protects the cardiovascular system through effects on the gut environment in non-diabetic nephrectomized rats."},"authors":{"en":[{"name":"Matsui Ayumi"},{"name":"Yoshifuji Ayumi"},{"name":"Irie Junichiro"},{"name":"Tajima Takaya"},{"name":"Uchiyama Kiyotaka"},{"name":"Itoh Tomoaki"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Matsui Ayumi"},{"name":"Yoshifuji Ayumi"},{"name":"Irie Junichiro"},{"name":"Tajima Takaya"},{"name":"Uchiyama Kiyotaka"},{"name":"Itoh Tomoaki"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"The increases in colonic glucose concentration, Lactobacillus numbers and tight junction protein expression, and the decreases in serum uremic toxin concentrations and cardiac interstitial fibrosis may have been caused by the inhibition of SGLT1 by canagliflozin because similar effects were not identified in tofogliflozin-treated rats.","ja":"The increases in colonic glucose concentration, Lactobacillus numbers and tight junction protein expression, and the decreases in serum uremic toxin concentrations and cardiac interstitial fibrosis may have been caused by the inhibition of SGLT1 by canagliflozin because similar effects were not identified in tofogliflozin-treated rats."},"publication_date":"2023-01-07","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"volume":"Vol.27","number":"No.4","starting_page":"295","ending_page":"308","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-022-02312-y"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:13, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539009"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119041","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=401904","label":"url"}],"paper_title":{"en":"Drug-induced de novo thrombotic microangiopathy diagnosed 2 years after renal transplantation: A case report and literature review","ja":"Drug-induced de novo thrombotic microangiopathy diagnosed 2 years after renal transplantation: A case report and literature review"},"authors":{"en":[{"name":"Yamaguchi Kunihisa"},{"name":"Ozaki Keisuke"},{"name":"Fukawa Tomoya"},{"name":"Sasaki Yutaro"},{"name":"Wakino Shu"},{"name":"Takahashi Masayuki"}],"ja":[{"name":"山口 邦久"},{"name":"尾崎 啓介"},{"name":"布川 朋也"},{"name":"佐々木 雄太郎"},{"name":"𦚰野 修"},{"name":"高橋 正幸"}]},"publication_date":"2023-01-04","publication_name":{"en":"Renal Replacement Therapy","ja":"Renal Replacement Therapy"},"volume":"Vol.9","starting_page":"1","ending_page":"6","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1186/s41100-022-00453-0"],"issn":["2059-1381"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:14, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539010"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119549","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1390861559985635456/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411348","label":"url"}],"paper_title":{"en":"8．栄養","ja":"8．栄養"},"authors":{"en":[{"name":"Wakino Shu"}],"ja":[{"name":"𦚰野 修"}]},"publication_date":"2023","publication_name":{"en":"J. Jpn. Soc. Dial. Ther.","ja":"透析会誌"},"volume":"Vol.56","number":"No.12","starting_page":"565","ending_page":"568","languages":["jpn"],"referee":true,"identifiers":{"doi":["10.4009/jsdt.56.565"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:15, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539011"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118481","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35999867","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400899","label":"url"}],"paper_title":{"en":"Investigation for the efficacy of COVID-19 vaccine in Japanese CKD patients treated with hemodialysis.","ja":"Investigation for the efficacy of COVID-19 vaccine in Japanese CKD patients treated with hemodialysis."},"authors":{"en":[{"name":"Yoshifuji Ayumi"},{"name":"Toda Masataro"},{"name":"Ryuzaki Munekazu"},{"name":"Kikuchi Kan"},{"name":"Kawai Toru"},{"name":"Sakai Ken"},{"name":"Oyama Emi"},{"name":"Koinuma Masayoshi"},{"name":"Katayama Kazuhiko"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kanno Yoshihiko"},{"name":"Kon Hirofumi"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"Wakino Shu"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}],"ja":[{"name":"Yoshifuji Ayumi"},{"name":"Toda Masataro"},{"name":"Ryuzaki Munekazu"},{"name":"Kikuchi Kan"},{"name":"Kawai Toru"},{"name":"Sakai Ken"},{"name":"Oyama Emi"},{"name":"Koinuma Masayoshi"},{"name":"Katayama Kazuhiko"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kanno Yoshihiko"},{"name":"Kon Hirofumi"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"𦚰野 修"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}]},"description":{"en":"Dialysis patients are predisposed to severe disease and have a high mortality rate in coronavirus disease 2019 (COVID-19) due to their comorbidities and immunocompromised conditions. Therefore, dialysis patients should be prioritized for vaccination. This study aimed to examine how long the effects of the vaccine are maintained and what factors affect antibody titers.","ja":"HD patients had significantly lower antibody titers than age- and sex-matched non-dialysis individuals over 3 months after vaccination. Dialysis time was identified as a factor affecting SARS-CoV-2 IgG titers in HD group, with longer dialysis time resulting in higher maximum SARS-CoV-2 IgG titers."},"publication_date":"2022-08-19","publication_name":{"en":"Renal Replacement Therapy","ja":"Renal Replacement Therapy"},"volume":"Vol.8","number":"No.1","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1186/s41100-022-00427-2"],"issn":["2059-1381"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:16, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539012"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35716954","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400900","label":"url"}],"paper_title":{"en":"Proximal-tubule molecular relay from early Protein diaphanous homolog 1 to late Rho-associated protein kinase 1 regulates kidney function in obesity-induced kidney damage.","ja":"Proximal-tubule molecular relay from early Protein diaphanous homolog 1 to late Rho-associated protein kinase 1 regulates kidney function in obesity-induced kidney damage."},"authors":{"en":[{"name":"Ida-Naitoh Makiko"},{"name":"Tokuyama Hirobumi"},{"name":"Futatsugi Koji"},{"name":"Yasuda Marie"},{"name":"Adachi Keika"},{"name":"Kanda Takeshi"},{"name":"Tanabe Yoshiyuki"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Ida-Naitoh Makiko"},{"name":"Tokuyama Hirobumi"},{"name":"Futatsugi Koji"},{"name":"Yasuda Marie"},{"name":"Adachi Keika"},{"name":"Kanda Takeshi"},{"name":"Tanabe Yoshiyuki"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"The small GTPase protein RhoA has two effectors, ROCK (Rho-associated protein kinase 1) and mDIA1 (protein diaphanous homolog 1), which cooperate reciprocally. However, temporal regulation of RhoA and its effectors in obesity-induced kidney damage remains unclear. Here, we investigated the role of RhoA activation in the proximal tubules at the early and late stages of obesity-induced kidney damage. In mice, a three-week high-fat-diet induced proximal tubule hypertrophy and damage without increased albuminuria, and RhoA/mDIA1 activation without ROCK activation. Conversely, a 12-week high-fat diet induced proximal tubule hypertrophy, proximal tubule damage, increased albuminuria, and RhoA/ROCK activation without mDIA1 elevation. Proximal tubule hypertrophy resulting from cell cycle arrest accompanied by downregulation of the multifunctional cyclin-dependent kinase inhibitor p27Kip1 was elicited by RhoA activation. Mice overexpressing proximal tubule-specific and dominant-negative RHOA display amelioration of high-fat diet-induced kidney hypertrophy, cell cycle abnormalities, inflammation, and renal impairment. In human proximal tubule cells, mechanical stretch mimicking hypertrophy activated ROCK, which triggered inflammation. In human kidney samples from normal individuals with a body mass index of about 25, proximal tubule cell size correlated with body mass index, proximal tubule cell damages, and mDIA1 expression. Thus, RhoA activation in proximal tubules is critical for the initiation and progression of obesity-induced kidney damage. Hence, the switch in the downstream RhoA effector in proximal tubule represents a transition from normal to pathogenic kidney adaptation and to body weight gain, leading to obesity-induced kidney damage.","ja":"The small GTPase protein RhoA has two effectors, ROCK (Rho-associated protein kinase 1) and mDIA1 (protein diaphanous homolog 1), which cooperate reciprocally. However, temporal regulation of RhoA and its effectors in obesity-induced kidney damage remains unclear. Here, we investigated the role of RhoA activation in the proximal tubules at the early and late stages of obesity-induced kidney damage. In mice, a three-week high-fat-diet induced proximal tubule hypertrophy and damage without increased albuminuria, and RhoA/mDIA1 activation without ROCK activation. Conversely, a 12-week high-fat diet induced proximal tubule hypertrophy, proximal tubule damage, increased albuminuria, and RhoA/ROCK activation without mDIA1 elevation. Proximal tubule hypertrophy resulting from cell cycle arrest accompanied by downregulation of the multifunctional cyclin-dependent kinase inhibitor p27Kip1 was elicited by RhoA activation. Mice overexpressing proximal tubule-specific and dominant-negative RHOA display amelioration of high-fat diet-induced kidney hypertrophy, cell cycle abnormalities, inflammation, and renal impairment. In human proximal tubule cells, mechanical stretch mimicking hypertrophy activated ROCK, which triggered inflammation. In human kidney samples from normal individuals with a body mass index of about 25, proximal tubule cell size correlated with body mass index, proximal tubule cell damages, and mDIA1 expression. Thus, RhoA activation in proximal tubules is critical for the initiation and progression of obesity-induced kidney damage. Hence, the switch in the downstream RhoA effector in proximal tubule represents a transition from normal to pathogenic kidney adaptation and to body weight gain, leading to obesity-induced kidney damage."},"publication_date":"2022-06-15","publication_name":{"en":"Kidney International","ja":"Kidney International"},"volume":"Vol.102","number":"No.4","starting_page":"798","ending_page":"814","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1016/j.kint.2022.05.018"],"issn":["1523-1755"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:17, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539013"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35610734","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400902","label":"url"}],"paper_title":{"en":"Effectiveness of SARS-CoV-2 vaccines on hemodialysis patients in Japan: A nationwide cohort study.","ja":"Effectiveness of SARS-CoV-2 vaccines on hemodialysis patients in Japan: A nationwide cohort study."},"authors":{"en":[{"name":"Kikuchi Kan"},{"name":"Nangaku Masaomi"},{"name":"Ryuzaki Munekazu"},{"name":"Yamakawa Tomoyuki"},{"name":"Yoshihiro Ota"},{"name":"Hanafusa Norio"},{"name":"Sakai Ken"},{"name":"Kanno Yoshihiko"},{"name":"Ando Ryoichi"},{"name":"Shinoda Toshio"},{"name":"Wakino Shu"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"},{"name":"Akizawa Tadao"}],"ja":[{"name":"Kikuchi Kan"},{"name":"Nangaku Masaomi"},{"name":"Ryuzaki Munekazu"},{"name":"Yamakawa Tomoyuki"},{"name":"Yoshihiro Ota"},{"name":"Hanafusa Norio"},{"name":"Sakai Ken"},{"name":"Kanno Yoshihiko"},{"name":"Ando Ryoichi"},{"name":"Shinoda Toshio"},{"name":"𦚰野 修"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"},{"name":"Akizawa Tadao"}]},"description":{"en":"Our prospective observational study showed that SRAS-CoV-2 vaccination in hemodialysis patients is vital for reducing need of oxygen supplementation and mortality risk.","ja":"Our prospective observational study showed that SRAS-CoV-2 vaccination in hemodialysis patients is vital for reducing need of oxygen supplementation and mortality risk."},"publication_date":"2022-06-14","publication_name":{"en":"Therapeutic Apheresis and Dialysis","ja":"Therapeutic Apheresis and Dialysis"},"volume":"Vol.27","number":"No.1","starting_page":"19","ending_page":"23","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1111/1744-9987.13887"],"issn":["1744-9987"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:18, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539014"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35699924","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400901","label":"url"}],"paper_title":{"en":"Diagnosis of monoclonal immunotactoid glomerulopathy with positive λ chain by immunoelectron microscopy.","ja":"Diagnosis of monoclonal immunotactoid glomerulopathy with positive λ chain by immunoelectron microscopy."},"authors":{"en":[{"name":"Sugita Erina"},{"name":"Sonoda Homare"},{"name":"Ryuzaki Masaki"},{"name":"Hashiguchi Akinori"},{"name":"Tokuyama Hirobumi"},{"name":"Wakino Shu"},{"name":"Kanda Takeshi"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Sugita Erina"},{"name":"Sonoda Homare"},{"name":"Ryuzaki Masaki"},{"name":"Hashiguchi Akinori"},{"name":"Tokuyama Hirobumi"},{"name":"𦚰野 修"},{"name":"Kanda Takeshi"},{"name":"Itoh Hiroshi"}]},"description":{"en":"We report the case of a 73-year-old-man who developed immunotactoid glomerulopathy (ITG). ITG is a rare disease characterized by proliferative glomerulonephritis and capillary wall deposits with a 10-60 nm diameter microtubular substructure. In monoclonal ITG, immunofluorescence analysis typically exhibits IgG with light chain restriction. Recent reviews recommend distinguishing monoclonal ITG from polyclonal ITG because monoclonal ITG is associated with a higher incidence of hematological disorders and better responsiveness to clone-directed therapy and renal prognosis. In our case, IgG, IgA, and IgM were negative by routine immunofluorescence; however, immunoelectron microscopy revealed positive λ chain. At 6 months after renal biopsy, the IgG λ chain was detected in the serum and urine, reflecting possible monoclonality. Therefore, it is useful to perform immunoelectron microscopy and follow-up with serum and urine protein electrophoresis and immunofixation to diagnose monoclonal ITG, even when routine immunofluorescence shows negative or nonspecific findings.","ja":"We report the case of a 73-year-old-man who developed immunotactoid glomerulopathy (ITG). ITG is a rare disease characterized by proliferative glomerulonephritis and capillary wall deposits with a 10-60 nm diameter microtubular substructure. In monoclonal ITG, immunofluorescence analysis typically exhibits IgG with light chain restriction. Recent reviews recommend distinguishing monoclonal ITG from polyclonal ITG because monoclonal ITG is associated with a higher incidence of hematological disorders and better responsiveness to clone-directed therapy and renal prognosis. In our case, IgG, IgA, and IgM were negative by routine immunofluorescence; however, immunoelectron microscopy revealed positive λ chain. At 6 months after renal biopsy, the IgG λ chain was detected in the serum and urine, reflecting possible monoclonality. Therefore, it is useful to perform immunoelectron microscopy and follow-up with serum and urine protein electrophoresis and immunofixation to diagnose monoclonal ITG, even when routine immunofluorescence shows negative or nonspecific findings."},"publication_date":"2022-06-14","publication_name":{"en":"CEN Case Reports","ja":"CEN Case Reports"},"volume":"Vol.12","number":"No.1","starting_page":"7","ending_page":"13","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s13730-022-00714-1"],"issn":["2192-4449"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:19, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539015"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118486","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35494536","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400904","label":"url"}],"paper_title":{"en":"Committee report: Questionnaire survey on the treatment of COVID-19 in patients receiving dialysis therapy.","ja":"Committee report: Questionnaire survey on the treatment of COVID-19 in patients receiving dialysis therapy."},"authors":{"en":[{"name":"Yoshifuji Ayumi"},{"name":"Ryuzaki Munekazu"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kawai Toru"},{"name":"Kanno Yoshihiko"},{"name":"Kikuchi Kan"},{"name":"Kon Hiroshi"},{"name":"Sakai Ken"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"Wakino Shu"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}],"ja":[{"name":"Yoshifuji Ayumi"},{"name":"Ryuzaki Munekazu"},{"name":"Uehara Yuki"},{"name":"Ohmagari Norio"},{"name":"Kawai Toru"},{"name":"Kanno Yoshihiko"},{"name":"Kikuchi Kan"},{"name":"Kon Hiroshi"},{"name":"Sakai Ken"},{"name":"Shinoda Toshio"},{"name":"Takano Yaoko"},{"name":"Tanaka Junko"},{"name":"Hora Kazuhiko"},{"name":"Nakazawa Yasushi"},{"name":"Hasegawa Naoki"},{"name":"Hanafusa Norio"},{"name":"Hinoshita Fumihiko"},{"name":"Morikane Keita"},{"name":"𦚰野 修"},{"name":"Nakamoto Hidetomo"},{"name":"Takemoto Yoshiaki"}]},"description":{"en":"Our survey revealed a variety of treatment practices in each facility. Further evidence and innovations are required to improve the prognosis of patients with COVID-19 receiving dialysis therapy.","ja":"Sixty-six centers (62.9%) responded to the questionnaire. Antivirals were administered in 27.7% of facilities treating mild disease (most patients received favipiravir) and 66.7% of facilities treating moderate disease (most patients with moderate or more severe conditions received remdesivir). Whether and how remdesivir is administered varies between centers. Steroids were initiated most frequently in moderate II disease (50.8%), while 43.1% of the facilities initiated steroids in mild or moderate I disease. The type of steroid, dose, and the duration of administration were generally consistent, with most facilities administering dexamethasone 6 mg orally or 6.6 mg intravenously for 10 days. Steroid pulse therapy was administered in 48.5% of the facilities, and tocilizumab was administered in 25.8% of the facilities, mainly to patients on ventilators or equivalent medications, or to the cases of exacerbations. Furthermore, some facilities used a polymethylmethacrylate membrane during dialysis, nafamostat as an anticoagulant, and continuous hemodiafiltration in severe cases. There was limited experience of polymyxin B-immobilized fiber column-direct hemoperfusion and extracorporeal membrane oxygenation. The discharge criteria for patients receiving dialysis therapy were longer than those set by the Ministry of Health, Labor and Welfare in 22.7% of the facilities."},"publication_date":"2022-04-25","publication_name":{"en":"Renal Replacement Therapy","ja":"Renal Replacement Therapy"},"volume":"Vol.8","number":"No.1","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1186/s41100-022-00405-8"],"issn":["2059-1381"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:20, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539016"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35362819","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400905","label":"url"}],"paper_title":{"en":"Effects of a remote patient monitoring system for patients on automated peritoneal dialysis: a randomized crossover controlled trial.","ja":"Effects of a remote patient monitoring system for patients on automated peritoneal dialysis: a randomized crossover controlled trial."},"authors":{"en":[{"name":"Uchiyama Kiyotaka"},{"name":"Morimoto Kohkichi"},{"name":"Washida Naoki"},{"name":"Kusahana Ei"},{"name":"Nakayama Takashin"},{"name":"Itoh Tomoaki"},{"name":"Kasai Takahiro"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Uchiyama Kiyotaka"},{"name":"Morimoto Kohkichi"},{"name":"Washida Naoki"},{"name":"Kusahana Ei"},{"name":"Nakayama Takashin"},{"name":"Itoh Tomoaki"},{"name":"Kasai Takahiro"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"The study was registered in the Japan Registry of Clinical Trials (jRCT Number: jRCTs032190005).","ja":"Significant improvements were observed in the TSQM-9 subscales of Effectiveness (64.4 ± 18.8 vs. 57.8 ± 18.8; P = 0.006) and Convenience (76.3 ± 15.4 vs. 63.3 ± 17.3; P < 0.001) in patients on Sharesource®. Moreover, Sharesource® reduced the total amount of healthcare resource consumption (0.80 ± 1.32 vs. 1.87 ± 2.39 times/12 weeks; P = 0.02) and consultation time during regular monthly visits (813 ± 269 vs. 1024 ± 292 s; P < 0.001). A significant increase in ultrafiltration volume was found associated with more frequent modification of APD prescription in patients with Sharesource®. Sharesource® also improved the HRQOL subscale of General Health and Vitality."},"publication_date":"2022-04-01","publication_name":{"en":"International Urology and Nephrology","ja":"International Urology and Nephrology"},"volume":"Vol.54","number":"No.10","starting_page":"2673","ending_page":"2681","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s11255-022-03178-5"],"issn":["1573-2584"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:21, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539017"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118485","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/35497789","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1050578979247097344/","label":"url"},{"@id":"https://www.scopus.com/record/display.url?eid=2-s2.0-85123373964&origin=inward","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400903","label":"url"}],"paper_title":{"en":"Home-Based Exercise Program Ameliorates Renal Function Decline in Patients With CKD Stage 4.","ja":"Home-Based Exercise Program Ameliorates Renal Function Decline in Patients With CKD Stage 4."},"authors":{"en":[{"name":"Adachi Keika"},{"name":"Uchiyama Kiyotaka"},{"name":"Muraoka Kaori"},{"name":"Nakayama Takashin"},{"name":"Yasuda Marie"},{"name":"Miyashita Kazutoshi"},{"name":"Tokuyama Hirobumi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Adachi Keika"},{"name":"Uchiyama Kiyotaka"},{"name":"Muraoka Kaori"},{"name":"Nakayama Takashin"},{"name":"Yasuda Marie"},{"name":"Miyashita Kazutoshi"},{"name":"Tokuyama Hirobumi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"publication_date":"2022-01-07","publication_name":{"en":"Kidney International Reports","ja":"Kidney International Reports"},"volume":"Vol.7","number":"No.4","starting_page":"899","ending_page":"903","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1016/j.ekir.2022.01.006"],"issn":["2468-0249"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:22, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539018"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118488","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/34554649","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400906","label":"url"}],"paper_title":{"en":"Home-based aerobic exercise and resistance training for severe chronic kidney disease: a randomized controlled trial.","ja":"Home-based aerobic exercise and resistance training for severe chronic kidney disease: a randomized controlled trial."},"authors":{"en":[{"name":"Uchiyama Kiyotaka"},{"name":"Adachi Keika"},{"name":"Muraoka Kaori"},{"name":"Nakayama Takashin"},{"name":"Oshida Takuma"},{"name":"Yasuda Marie"},{"name":"Hishikawa Akihito"},{"name":"Minakuchi Hitoshi"},{"name":"Miyashita Kazutoshi"},{"name":"Tokuyama Hirobumi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Uchiyama Kiyotaka"},{"name":"Adachi Keika"},{"name":"Muraoka Kaori"},{"name":"Nakayama Takashin"},{"name":"Oshida Takuma"},{"name":"Yasuda Marie"},{"name":"Hishikawa Akihito"},{"name":"Minakuchi Hitoshi"},{"name":"Miyashita Kazutoshi"},{"name":"Tokuyama Hirobumi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Our 6 month home-based exercise programme improved aerobic capacity and HRQOL in patients with Stage 4 CKD, with possible beneficial effects on kidney function and CKD-related parameters.","ja":"Our 6 month home-based exercise programme improved aerobic capacity and HRQOL in patients with Stage 4 CKD, with possible beneficial effects on kidney function and CKD-related parameters."},"publication_date":"2021-09-23","publication_name":{"en":"Journal of Cachexia, Sarcopenia and Muscle","ja":"Journal of Cachexia, Sarcopenia and Muscle"},"volume":"Vol.12","number":"No.6","starting_page":"1789","ending_page":"1802","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1002/jcsm.12775"],"issn":["2190-6009"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:23, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539019"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/34529243","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400907","label":"url"}],"paper_title":{"en":"Fabry disease associated with multiple myeloma: a case report.","ja":"Fabry disease associated with multiple myeloma: a case report."},"authors":{"en":[{"name":"Adachi Keika"},{"name":"Tokuyama Hirobumi"},{"name":"Oshima Yoichi"},{"name":"Itoh Tomoaki"},{"name":"Hashiguchi Akinori"},{"name":"Yamakawa Hiroyuki"},{"name":"Togawa Tadayasu"},{"name":"Sakuraba Hitoshi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Adachi Keika"},{"name":"Tokuyama Hirobumi"},{"name":"Oshima Yoichi"},{"name":"Itoh Tomoaki"},{"name":"Hashiguchi Akinori"},{"name":"Yamakawa Hiroyuki"},{"name":"Togawa Tadayasu"},{"name":"Sakuraba Hitoshi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Fabry disease (FD) is an X-linked genetic lysosomal disorder caused by alpha-galactosidase A (GLA) deficiency. Multiple myeloma (MM) predominately affects older adults, which ranks as the second commonest hematological malignancy. Their overlap has rarely been reported. We present a case of the coexistence of FD and MM in a patient. We report the case of a 68-year-old woman who was referred to our hospital for the evaluation of thoracic spine tumor with bone destruction. On admission, her serum creatinine (Cr) level was elevated to 12.70 mg/dL from the baseline value of 0.91 mg/dL. Bone marrow aspiration revealed MM. Renal biopsy showed myeloma cast nephropathy, which was the primary cause of acute kidney injury. Renal pathology also showed podocyte swelling and tubule myeloid bodies in a mosaic pattern compatible with female FD. Consequently, the patient was diagnosed as FD based on the germ line mutation in GLA. The patient was treated with bortezomib and dexamethasone therapy, which significantly improved the renal function. This is the second case demonstrating a potential pathogenic relationship between FD and MM. Since FD is one of the few genetic diseases for which there are therapeutic agents with fewer side effects, diagnostic value of FD is high. If an MM patient has multiple organ abnormalities or any familial history, the physician should suspect FD.","ja":"Fabry disease (FD) is an X-linked genetic lysosomal disorder caused by alpha-galactosidase A (GLA) deficiency. Multiple myeloma (MM) predominately affects older adults, which ranks as the second commonest hematological malignancy. Their overlap has rarely been reported. We present a case of the coexistence of FD and MM in a patient. We report the case of a 68-year-old woman who was referred to our hospital for the evaluation of thoracic spine tumor with bone destruction. On admission, her serum creatinine (Cr) level was elevated to 12.70 mg/dL from the baseline value of 0.91 mg/dL. Bone marrow aspiration revealed MM. Renal biopsy showed myeloma cast nephropathy, which was the primary cause of acute kidney injury. Renal pathology also showed podocyte swelling and tubule myeloid bodies in a mosaic pattern compatible with female FD. Consequently, the patient was diagnosed as FD based on the germ line mutation in GLA. The patient was treated with bortezomib and dexamethasone therapy, which significantly improved the renal function. This is the second case demonstrating a potential pathogenic relationship between FD and MM. Since FD is one of the few genetic diseases for which there are therapeutic agents with fewer side effects, diagnostic value of FD is high. If an MM patient has multiple organ abnormalities or any familial history, the physician should suspect FD."},"publication_date":"2021-09-16","publication_name":{"en":"CEN Case Reports","ja":"CEN Case Reports"},"volume":"Vol.11","number":"No.1","starting_page":"146","ending_page":"153","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s13730-021-00613-x"],"issn":["2192-4449"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:24, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539020"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/34143371","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400908","label":"url"}],"paper_title":{"en":"Efficacy of dexmedetomidine on peritoneal dialysis catheter insertion.","ja":"Efficacy of dexmedetomidine on peritoneal dialysis catheter insertion."},"authors":{"en":[{"name":"Nakayama Takashin"},{"name":"Uchiyama Kiyotaka"},{"name":"Morimoto Kohkichi"},{"name":"Washida Naoki"},{"name":"Kasai Takahiro"},{"name":"Nakamichi Ran"},{"name":"Kusahana Ei"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Nakayama Takashin"},{"name":"Uchiyama Kiyotaka"},{"name":"Morimoto Kohkichi"},{"name":"Washida Naoki"},{"name":"Kasai Takahiro"},{"name":"Nakamichi Ran"},{"name":"Kusahana Ei"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"The use of DEX in PD catheter insertion under spinal anesthesia could safely improve operative analgesia.","ja":"Of a total of 44 patients, 9 patients received DEX, and 35 did not. After propensity score matching, each group consisted of 8 patients. Peak NRS was significantly lower (P = 0.003) in the DEX group compared with the non-DEX group. Maximum mean BP during the operation was also significantly lower in the DEX group compared with the non-DEX group (P = 0.020), with no significant differences in minimum mean BP between the two groups (P = 0.831). The DEX group showed a trend of shortened operative time (P = 0.068). There were no significant differences in the occurrence of nausea (P = 1.000). Moreover, there was no clinically important adverse event associated with use of DEX."},"publication_date":"2021-06-18","publication_name":{"en":"International Urology and Nephrology","ja":"International Urology and Nephrology"},"volume":"Vol.54","number":"No.1","starting_page":"209","ending_page":"215","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s11255-021-02916-5"],"issn":["1573-2584"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:25, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539021"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33795425","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400909","label":"url"}],"paper_title":{"en":"Pre-emptive Short-term Nicotinamide Mononucleotide Treatment in a Mouse Model of Diabetic Nephropathy.","ja":"Pre-emptive Short-term Nicotinamide Mononucleotide Treatment in a Mouse Model of Diabetic Nephropathy."},"authors":{"en":[{"name":"Yasuda Itaru"},{"name":"Hasegawa Kazuhiro"},{"name":"Sakamaki Yusuke"},{"name":"Muraoka Hirokazu"},{"name":"Kawaguchi Takahisa"},{"name":"Kusahana Ei"},{"name":"Ono Takashi"},{"name":"Kanda Takeshi"},{"name":"Tokuyama Hirobumi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Yasuda Itaru"},{"name":"Hasegawa Kazuhiro"},{"name":"Sakamaki Yusuke"},{"name":"Muraoka Hirokazu"},{"name":"Kawaguchi Takahisa"},{"name":"Kusahana Ei"},{"name":"Ono Takashi"},{"name":"Kanda Takeshi"},{"name":"Tokuyama Hirobumi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"salvage pathway, both of which indicate NMN legacy effects on DN.","ja":"salvage pathway, both of which indicate NMN legacy effects on DN."},"publication_date":"2021-04-01","publication_name":{"en":"Journal of the American Society of Nephrology","ja":"Journal of the American Society of Nephrology"},"volume":"Vol.32","number":"No.6","starting_page":"1355","ending_page":"1370","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1681/ASN.2020081188"],"issn":["1533-3450"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:26, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539022"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33476039","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400910","label":"url"}],"paper_title":{"en":"Development of nephropathy in an adult patient after Fontan palliation for cyanotic congenital heart disease.","ja":"Development of nephropathy in an adult patient after Fontan palliation for cyanotic congenital heart disease."},"authors":{"en":[{"name":"Hayashi Kaori"},{"name":"Hashiguchi Akinori"},{"name":"Ikemiyagi Masako"},{"name":"Tokuyama Hirobumi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Hayashi Kaori"},{"name":"Hashiguchi Akinori"},{"name":"Ikemiyagi Masako"},{"name":"Tokuyama Hirobumi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Cyanotic congenital heart disease is occasionally associated with kidney dysfunction, which is known as cyanotic nephropathy or cyanotic glomerulopathy. The clinical presentation of cyanotic nephropathy includes proteinuria, decreased estimated glomerular filtration rate, hyperuricemia, thrombocytopenia, or polycythemia. Although advances in surgical procedures have improved the prognosis of cyanotic congenital heart diseases, adult cases of cyanotic nephropathy are still rare, and there are few reports of kidney biopsy in adults with cyanotic nephropathy. Here, we present the case of a 41-year-old patient with Fontan palliation who developed nephrotic range proteinuria and had a kidney biopsy, which showed glomerular hypertrophy with segmental glomerulosclerosis around vascular poles, suggesting adaptive focal segmental glomerulosclerosis. This case provides further understanding of kidney dysfunction due to cyanotic congenital heart disease and shows the need for attention in the management for prevention of progression to end-stage renal disease and in the selection of renal replacement therapy.","ja":"Cyanotic congenital heart disease is occasionally associated with kidney dysfunction, which is known as cyanotic nephropathy or cyanotic glomerulopathy. The clinical presentation of cyanotic nephropathy includes proteinuria, decreased estimated glomerular filtration rate, hyperuricemia, thrombocytopenia, or polycythemia. Although advances in surgical procedures have improved the prognosis of cyanotic congenital heart diseases, adult cases of cyanotic nephropathy are still rare, and there are few reports of kidney biopsy in adults with cyanotic nephropathy. Here, we present the case of a 41-year-old patient with Fontan palliation who developed nephrotic range proteinuria and had a kidney biopsy, which showed glomerular hypertrophy with segmental glomerulosclerosis around vascular poles, suggesting adaptive focal segmental glomerulosclerosis. This case provides further understanding of kidney dysfunction due to cyanotic congenital heart disease and shows the need for attention in the management for prevention of progression to end-stage renal disease and in the selection of renal replacement therapy."},"publication_date":"2021-01-21","publication_name":{"en":"CEN Case Reports","ja":"CEN Case Reports"},"volume":"Vol.10","number":"No.3","starting_page":"354","ending_page":"358","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s13730-021-00573-2"],"issn":["2192-4449"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:27, {"insert":{"user_id":"5000065922","type":"published_papers"},"similar_merge":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118492","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33452384","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400911","label":"url"}],"paper_title":{"en":"Diabetic condition induces hypertrophy and vacuolization in glomerular parietal epithelial cells.","ja":"Diabetic condition induces hypertrophy and vacuolization in glomerular parietal epithelial cells."},"authors":{"en":[{"name":"Kawaguchi Takahisa"},{"name":"Hasegawa Kazuhiro"},{"name":"Yasuda Itaru"},{"name":"Muraoka Hirokazu"},{"name":"Umino Hiroyuki"},{"name":"Tokuyama Hirobumi"},{"name":"Hashiguchi Akinori"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Kawaguchi Takahisa"},{"name":"Hasegawa Kazuhiro"},{"name":"Yasuda Itaru"},{"name":"Muraoka Hirokazu"},{"name":"Umino Hiroyuki"},{"name":"Tokuyama Hirobumi"},{"name":"Hashiguchi Akinori"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Diabetic nephropathy (DN) is accompanied by characteristic changes in the glomerulus, but little is known about the effect of diabetes on parietal epithelial cells (PECs). In this study, a descriptive analysis of PECs was undertaken in diabetic db/db mice and in diabetic patients. PEC hypertrophy was significantly more prominent in diabetic mice than in nondiabetic mice, and this was evident even at the early stage. Additionally, the number of vacuoles in PECs was markedly increased in diabetic mice, suggesting the presence of cellular injury in PECs in DN. Although rare, binuclear cells were observed in mice with early diabetes. In cultured PECs, a high glucose condition, compared with normal glucose condition, induced cellular hypertrophy and apoptosis. Flow cytometry showed that some PECs in the G0 phase reentered the cell cycle but got arrested in the S phase. Finally, in human diabetic subjects, hypertrophy and vacuolization were observed in the PECs. Our data showed that PECs undergo substantial changes in DN and may participate in rearrangement for differentiation into podocytes.","ja":"Diabetic nephropathy (DN) is accompanied by characteristic changes in the glomerulus, but little is known about the effect of diabetes on parietal epithelial cells (PECs). In this study, a descriptive analysis of PECs was undertaken in diabetic db/db mice and in diabetic patients. PEC hypertrophy was significantly more prominent in diabetic mice than in nondiabetic mice, and this was evident even at the early stage. Additionally, the number of vacuoles in PECs was markedly increased in diabetic mice, suggesting the presence of cellular injury in PECs in DN. Although rare, binuclear cells were observed in mice with early diabetes. In cultured PECs, a high glucose condition, compared with normal glucose condition, induced cellular hypertrophy and apoptosis. Flow cytometry showed that some PECs in the G0 phase reentered the cell cycle but got arrested in the S phase. Finally, in human diabetic subjects, hypertrophy and vacuolization were observed in the PECs. Our data showed that PECs undergo substantial changes in DN and may participate in rearrangement for differentiation into podocytes."},"publication_date":"2021-01-15","publication_name":{"en":"Scientific Reports","ja":"Scientific Reports"},"volume":"Vol.11","number":"No.1","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1038/s41598-021-81027-8"],"issn":["2045-2322"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:28, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539023"},"force":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33393071","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400912","label":"url"}],"paper_title":{"en":"Malignancy-associated membranous nephropathy with PLA2R double-positive for glomeruli and carcinoma.","ja":"Malignancy-associated membranous nephropathy with PLA2R double-positive for glomeruli and carcinoma."},"authors":{"en":[{"name":"Yasuda Itaru"},{"name":"Tokuyama Hirobumi"},{"name":"Hashiguchi Akinori"},{"name":"Hasegawa Kazuhiro"},{"name":"Uchiyama Kiyotaka"},{"name":"Ryuzaki Masaki"},{"name":"Yasuda Marie"},{"name":"Mizuno Ryuichi"},{"name":"Ishidoya Shigeto"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Yasuda Itaru"},{"name":"Tokuyama Hirobumi"},{"name":"Hashiguchi Akinori"},{"name":"Hasegawa Kazuhiro"},{"name":"Uchiyama Kiyotaka"},{"name":"Ryuzaki Masaki"},{"name":"Yasuda Marie"},{"name":"Mizuno Ryuichi"},{"name":"Ishidoya Shigeto"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"Phospholipase A2 receptor (PLA2R) is the most common primary target antigen of idiopathic membranous nephropathy (MN) although PLA2R antibodies are also reported to be present in malignancy-associated MN. However, a case of PLA2R-positive MN secondary to PLA2R-positive carcinoma has not been reported. A 26-year-old Japanese woman presented with general fatigue, fever, and nonproductive cough. Computed tomography demonstrated a left kidney mass with pathologic diagnosis of Xp11.2 translocation renal cell carcinoma (RCC). After the second time of administration with Sunitinib, the patients exhibited significant proteinuria and hypoalbuminemia. Renal biopsy revealed a diagnosis of diffuse MN secondary to RCC. Immunofluorescence staining showed granular patterns positive for immunoglobulin (Ig) G, IgA, and C3c. PLA2R and IgG1-3 were positive, while IgG4 was negative. For the treatment of severe nephrotic syndrome, we attempted steroid therapy without any clinical improvement. Open nephrectomy was performed and surprisingly, RCC was stained for PLA2R with polarity for the basal side. At outpatient follow-up, 4 months after the operation, urinary protein had still persisted, although serum albumin was slightly increased. We report a case of PLA2R-positive MN secondary to PLA2R-positive RCC.","ja":"Phospholipase A2 receptor (PLA2R) is the most common primary target antigen of idiopathic membranous nephropathy (MN) although PLA2R antibodies are also reported to be present in malignancy-associated MN. However, a case of PLA2R-positive MN secondary to PLA2R-positive carcinoma has not been reported. A 26-year-old Japanese woman presented with general fatigue, fever, and nonproductive cough. Computed tomography demonstrated a left kidney mass with pathologic diagnosis of Xp11.2 translocation renal cell carcinoma (RCC). After the second time of administration with Sunitinib, the patients exhibited significant proteinuria and hypoalbuminemia. Renal biopsy revealed a diagnosis of diffuse MN secondary to RCC. Immunofluorescence staining showed granular patterns positive for immunoglobulin (Ig) G, IgA, and C3c. PLA2R and IgG1-3 were positive, while IgG4 was negative. For the treatment of severe nephrotic syndrome, we attempted steroid therapy without any clinical improvement. Open nephrectomy was performed and surprisingly, RCC was stained for PLA2R with polarity for the basal side. At outpatient follow-up, 4 months after the operation, urinary protein had still persisted, although serum albumin was slightly increased. We report a case of PLA2R-positive MN secondary to PLA2R-positive RCC."},"publication_date":"2021-01-03","publication_name":{"en":"CEN Case Reports","ja":"CEN Case Reports"},"volume":"Vol.10","number":"No.2","starting_page":"281","ending_page":"286","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s13730-020-00556-9"],"issn":["2192-4449"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:29, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539024"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118494","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33091919","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400914","label":"url"}],"paper_title":{"en":"Exercise Parameters Predict Technique Survival in Patients on Peritoneal Dialysis.","ja":"Exercise Parameters Predict Technique Survival in Patients on Peritoneal Dialysis."},"authors":{"en":[{"name":"Nakayama Takashin"},{"name":"Uchiyama Kiyotaka"},{"name":"Washida Naoki"},{"name":"Morimoto Kohkichi"},{"name":"Muraoka Kaori"},{"name":"Adachi Keika"},{"name":"Kasai Takahiro"},{"name":"Miyashita Kazutoshi"},{"name":"Wakino Shu"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Nakayama Takashin"},{"name":"Uchiyama Kiyotaka"},{"name":"Washida Naoki"},{"name":"Morimoto Kohkichi"},{"name":"Muraoka Kaori"},{"name":"Adachi Keika"},{"name":"Kasai Takahiro"},{"name":"Miyashita Kazutoshi"},{"name":"𦚰野 修"},{"name":"Itoh Hiroshi"}]},"description":{"en":"The ISWT is an important predictor of technique survival for patients on PD. Monitoring and enhancing ISWT as a marker of aerobic capacity might improve PD-related outcomes.","ja":"The ISWT is an important predictor of technique survival for patients on PD. Monitoring and enhancing ISWT as a marker of aerobic capacity might improve PD-related outcomes."},"publication_date":"2020-10-22","publication_name":{"en":"Blood Purification","ja":"Blood Purification"},"volume":"Vol.50","number":"No.3","starting_page":"380","ending_page":"389","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1159/000511293"],"issn":["1421-9735"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:30, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539025"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/118495","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/33024237","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400915","label":"url"}],"paper_title":{"en":"The effect of aldosterone and aldosterone blockade on the progression of chronic kidney disease: a randomized placebo-controlled clinical trial.","ja":"The effect of aldosterone and aldosterone blockade on the progression of chronic kidney disease: a randomized placebo-controlled clinical trial."},"authors":{"en":[{"name":"Minakuchi Hitoshi"},{"name":"Wakino Shu"},{"name":"Urai Hidenori"},{"name":"Kurokochi Arata"},{"name":"Hasegawa Kazuhiro"},{"name":"Kanda Takeshi"},{"name":"Tokuyama Hirobumi"},{"name":"Itoh Hiroshi"}],"ja":[{"name":"Minakuchi Hitoshi"},{"name":"𦚰野 修"},{"name":"Urai Hidenori"},{"name":"Kurokochi Arata"},{"name":"Hasegawa Kazuhiro"},{"name":"Kanda Takeshi"},{"name":"Tokuyama Hirobumi"},{"name":"Itoh Hiroshi"}]},"description":{"en":"/year, p = 0.0047). In the final intervention study, in the eplerenone group, eGFR dropped at 6 months after the initiation of the study, and thereafter eGFR was maintained until the end of the study. At 24 months and 36 months, eGFR was significantly higher in the eplerenone group than in the placebo group. In conclusion, MRA can be an effective strategy in preventing CKD progression, especially in patients with high plasma aldosterone.","ja":"/year, p = 0.0047). In the final intervention study, in the eplerenone group, eGFR dropped at 6 months after the initiation of the study, and thereafter eGFR was maintained until the end of the study. At 24 months and 36 months, eGFR was significantly higher in the eplerenone group than in the placebo group. In conclusion, MRA can be an effective strategy in preventing CKD progression, especially in patients with high plasma aldosterone."},"publication_date":"2020-10-06","publication_name":{"en":"Scientific Reports","ja":"Scientific Reports"},"volume":"Vol.10","number":"No.1","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1038/s41598-020-73638-4"],"issn":["2045-2322"]},"published_paper_type":"scientific_journal"},"priority":"input_data"}
line:31, {"insert":{"user_id":"5000065922","type":"published_papers","id":"47539026"},"force":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119552","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1390858518834673280/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411347","label":"url"}],"paper_title":{"en":"血液透析濾過器の性能評価と使い分け","ja":"血液透析濾過器の性能評価と使い分け"},"authors":{"en":[{"name":"友 雅司"},{"name":"Mineshima Michio"},{"name":"Wakino Shu"},{"name":"武本 佳昭"}],"ja":[{"name":"友 雅司"},{"name":"峰島 三千男"},{"name":"𦚰野 修"},{"name":"武本 佳昭"}]},"publication_date":"2023","publication_name":{"en":"J. Jpn. Soc. Dial. Ther.","ja":"透析会誌"},"volume":"Vol.56","number":"No.3","starting_page":"83","ending_page":"84","languages":["jpn"],"identifiers":{"doi":["10.4009/jsdt.56.83"]}},"priority":"input_data"}
==== end registerFile(/WWW/pub2/data/ERD/person/375084/researchmap/published_papers.jsonl, VksZQZMBwbWENEEN9LkP) ====
==== begin registerFile(/WWW/pub2/data/ERD/person/375084/researchmap/misc.jsonl) ====
line:1, {"insert":{"user_id":"5000065922","type":"misc","id":"47539028"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390581003357831808/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410387","label":"url"}],"paper_title":{"en":"増大号 AKI·CKDの診断·治療に臨床検査を活かせ 4章 腎疾患を知る-臨床検査ができること 臨床検査で迫る腎疾患 糖尿病性腎症·腎臓病と腎硬化症","ja":"増大号 AKI·CKDの診断·治療に臨床検査を活かせ 4章 腎疾患を知る-臨床検査ができること 臨床検査で迫る腎疾患 糖尿病性腎症·腎臓病と腎硬化症"},"authors":{"en":[{"name":"Tamaki Masanori"},{"name":"Wakino Shu"}],"ja":[{"name":"田蒔 昌憲"},{"name":"𦚰野 修"}]},"publication_date":"2024-04-15","publication_name":{"en":"Journal of Medical Technology","ja":"臨床検査"},"volume":"Vol.68","number":"No.4","starting_page":"524","ending_page":"527","languages":["jpn"],"identifiers":{"doi":["10.11477/mf.1542203599"],"issn":["0485-1420"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
line:2, {"insert":{"user_id":"5000065922","type":"misc","id":"47539029"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1520299596038519296/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410386","label":"url"}],"paper_title":{"en":"特集 微量元素 みいつけた : 生理作用・疾患・くすりと食品にクローズアップ! ; 場面別! 知っておきたい欠乏症とその予防・治療の方法","ja":"特集 微量元素 みいつけた : 生理作用・疾患・くすりと食品にクローズアップ! ; 場面別! 知っておきたい欠乏症とその予防・治療の方法"},"authors":{"en":[{"name":"Wakino Shu"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"}],"ja":[{"name":"𦚰野 修"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"}]},"publication_date":"2024-03","publication_name":{"en":"The Journal of Practical Pharmacy","ja":"薬局"},"volume":"Vol.75","number":"No.3","starting_page":"346","ending_page":"350","languages":["jpn"],"identifiers":{"issn":["0044-0035"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
line:3, {"insert":{"user_id":"5000065922","type":"misc","id":"47539030"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390299528381041664/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410125","label":"url"}],"paper_title":{"en":"特集 糖尿病性腎症研究の最前線 糖尿病性腎症の発症機序 糖尿病性腎症におけるミトコンドリアの役割","ja":"特集 糖尿病性腎症研究の最前線 糖尿病性腎症の発症機序 糖尿病性腎症におけるミトコンドリアの役割"},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"},{"name":"Minato Masanori"},{"name":"Inagaki Taizo"},{"name":"清水 郁子"},{"name":"山口 純代"},{"name":"宮上 慎司"},{"name":"多田 美穂"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"},{"name":"湊 将典"},{"name":"稲垣 太造"},{"name":"清水 郁子"},{"name":"山口 純代"},{"name":"宮上 慎司"},{"name":"多田 美穂"},{"name":"𦚰野 修"}]},"publication_date":"2024-02-25","publication_name":{"en":"Kidney and Dialysis","ja":"腎と透析"},"volume":"Vol.96","number":"No.2","starting_page":"159","ending_page":"164","languages":["jpn"],"invited":true,"identifiers":{"doi":["10.24479/kd.0000001187"],"issn":["0385-2156"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
line:4, {"insert":{"user_id":"5000065922","type":"misc","id":"47539031"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390862168254226944/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410126","label":"url"}],"paper_title":{"en":"特集 血液透析患者の血圧を再考する 5.透析患者の至適血圧-腹膜透析も含む","ja":"特集 血液透析患者の血圧を再考する 5.透析患者の至適血圧-腹膜透析も含む"},"authors":{"en":[{"name":"Wakino Shu"},{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"}],"ja":[{"name":"𦚰野 修"},{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"}]},"publication_date":"2024-02-10","publication_name":{"en":"The Japanese Journal of Clinical Dialysis","ja":"臨床透析"},"volume":"Vol.40","number":"No.2","starting_page":"155","ending_page":"163","languages":["jpn"],"identifiers":{"doi":["10.19020/cd.0000002887"],"issn":["0910-5808"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
line:5, {"insert":{"user_id":"5000065922","type":"misc","id":"47539032"},"force":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390861859460431104/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411341","label":"url"}],"paper_title":{"en":"特集 病因·病態生理から読み解く腎·泌尿器疾患のすべて Ⅴ.AKI 5.心臓手術におけるAKI","ja":"特集 病因·病態生理から読み解く腎·泌尿器疾患のすべて Ⅴ.AKI 5.心臓手術におけるAKI"},"authors":{"en":[{"name":"Wakino Shu"}],"ja":[{"name":"𦚰野 修"}]},"publication_date":"2023-12-15","publication_name":{"en":"腎と透析","ja":"腎と透析"},"volume":"Vol.95","number":"No.7","starting_page":"361","ending_page":"366","languages":["jpn"],"identifiers":{"doi":["10.24479/kd.0000001057"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"}
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