=== Generating (published_papers) === === Generating (teaching_experience) === === Generating (misc) === === Generating (research_projects) === === Generating (presentations) === ==== begin registerFile(/WWW/pub2/data/ERD/person/376686/researchmap/published_papers.jsonl) ==== line:1, {"insert":{"user_id":"6000007612","type":"published_papers"},"similar_merge":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119529","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38928090","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410124","label":"url"}],"paper_title":{"en":"Deficiency Causes Mitoribosome Excess in Diabetic Nephropathy Mediated by Transcriptional Repressor HIC1.","ja":"Deficiency Causes Mitoribosome Excess in Diabetic Nephropathy Mediated by Transcriptional Repressor HIC1."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Sakamaki Yusuke"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"Sakamaki Yusuke"},{"name":"𦚰野 修"}]},"description":{"en":"overexpression preserved mitoribosomal function, suggesting its protective role in DN.","ja":"overexpression preserved mitoribosomal function, suggesting its protective role in DN."},"publication_date":"2024-06-09","publication_name":{"en":"International Journal of Molecular Sciences","ja":"International Journal of Molecular Sciences"},"volume":"Vol.25","number":"No.12","languages":["eng"],"referee":true,"identifiers":{"doi":["10.3390/ijms25126384"],"issn":["1422-0067"]},"published_paper_type":"scientific_journal"},"priority":"input_data"} line:2, {"insert":{"user_id":"6000007612","type":"published_papers"},"similar_merge":{"see_also":[{"@id":"https://repo.lib.tokushima-u.ac.jp/ja/119528","label":"url"},{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/38587753","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410122","label":"url"}],"paper_title":{"en":"Ability of NAD and Sirt1 to epigenetically suppress albuminuria.","ja":"Ability of NAD and Sirt1 to epigenetically suppress albuminuria."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"},{"name":"Inagaki Taizo"},{"name":"Minato Masanori"},{"name":"Yamaguchi Sumiyo"},{"name":"Shimizu Ikuko"},{"name":"Miyakami Shinji"},{"name":"Tada Miho"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"},{"name":"稲垣 太造"},{"name":"湊 将典"},{"name":"Yamaguchi Sumiyo"},{"name":"清水 郁子"},{"name":"Miyakami Shinji"},{"name":"多田 美穂"},{"name":"𦚰野 修"}]},"description":{"en":"The time for diabetic nephropathy (DN) to progress from mild to severe is long. Thus, methods to continuously repress DN are required to exert long-lasting effects mediated through epigenetic regulation. In this study, we demonstrated the ability of nicotinamide adenine dinucleotide (NAD) and its metabolites to reduce albuminuria through Sirt1- or Nampt-dependent epigenetic regulation. We previously reported that proximal tubular Sirt1 was lowered before glomerular Sirt1. Repressed glomerular Sirt1 was found to epigenetically elevate Claudin-1. In addition, we reported that proximal tubular Nampt deficiency epigenetically augmented TIMP-1 levels in Sirt6-mediated pathways, leading to type-IV collagen deposition and diabetic fibrosis. Altogether, we propose that the Sirt1/Claudin-1 axis may be crucial in the onset of albuminuria at the early stages of DN and that the Nampt/Sirt6/TIMP-1 axis promotes diabetic fibrosis in the middle to late stages of DN. Finally, administration of NMN, an NAD precursor, epigenetically potentiates the regression of the onset of DN to maintain Sirt1 and repress Claudin-1 in podocytes, suggesting the potential use of NAD metabolites as epigenetic medications for DN.","ja":"The time for diabetic nephropathy (DN) to progress from mild to severe is long. Thus, methods to continuously repress DN are required to exert long-lasting effects mediated through epigenetic regulation. In this study, we demonstrated the ability of nicotinamide adenine dinucleotide (NAD) and its metabolites to reduce albuminuria through Sirt1- or Nampt-dependent epigenetic regulation. We previously reported that proximal tubular Sirt1 was lowered before glomerular Sirt1. Repressed glomerular Sirt1 was found to epigenetically elevate Claudin-1. In addition, we reported that proximal tubular Nampt deficiency epigenetically augmented TIMP-1 levels in Sirt6-mediated pathways, leading to type-IV collagen deposition and diabetic fibrosis. Altogether, we propose that the Sirt1/Claudin-1 axis may be crucial in the onset of albuminuria at the early stages of DN and that the Nampt/Sirt6/TIMP-1 axis promotes diabetic fibrosis in the middle to late stages of DN. Finally, administration of NMN, an NAD precursor, epigenetically potentiates the regression of the onset of DN to maintain Sirt1 and repress Claudin-1 in podocytes, suggesting the potential use of NAD metabolites as epigenetic medications for DN."},"publication_date":"2024-04-08","publication_name":{"en":"Clinical and Experimental Nephrology","ja":"Clinical and Experimental Nephrology"},"volume":"Vol.28","number":"No.7","starting_page":"599","ending_page":"607","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1007/s10157-024-02502-w"],"issn":["1437-7799"]},"published_paper_type":"scientific_journal"},"priority":"input_data"} line:3, {"insert":{"user_id":"6000007612","type":"published_papers"},"similar_merge":{"see_also":[{"@id":"https://www.ncbi.nlm.nih.gov/pubmed/37199399","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400706","label":"url"}],"paper_title":{"en":"PCK1 Protects against Mitoribosomal Defects in Diabetic Nephropathy in Mouse Models.","ja":"PCK1 Protects against Mitoribosomal Defects in Diabetic Nephropathy in Mouse Models."},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Sakamaki Yusuke"},{"name":"Tamaki Masanori"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"Sakamaki Yusuke"},{"name":"田蒔 昌憲"},{"name":"𦚰野 修"}]},"description":{"en":"PCK1 preserves mitoribosomal function and may play a novel protective role in DN.","ja":"PCK1 preserves mitoribosomal function and may play a novel protective role in DN."},"publication_date":"2023-05-18","publication_name":{"en":"Journal of the American Society of Nephrology","ja":"Journal of the American Society of Nephrology"},"volume":"Vol.34","number":"No.8","starting_page":"1343","ending_page":"1365","languages":["eng"],"referee":true,"identifiers":{"doi":["10.1681/ASN.0000000000000156"],"issn":["1533-3450"]},"published_paper_type":"scientific_journal"},"priority":"input_data"} ==== end registerFile(/WWW/pub2/data/ERD/person/376686/researchmap/published_papers.jsonl, kdLNnZMBwbWENEENzrSs) ==== ==== begin registerFile(/WWW/pub2/data/ERD/person/376686/researchmap/misc.jsonl) ==== line:1, {"insert":{"user_id":"6000007612","type":"misc"},"similar_merge":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390299528381041664/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410125","label":"url"}],"paper_title":{"en":"特集 糖尿病性腎症研究の最前線 糖尿病性腎症の発症機序 糖尿病性腎症におけるミトコンドリアの役割","ja":"特集 糖尿病性腎症研究の最前線 糖尿病性腎症の発症機序 糖尿病性腎症におけるミトコンドリアの役割"},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"},{"name":"Minato Masanori"},{"name":"Inagaki Taizo"},{"name":"清水 郁子"},{"name":"山口 純代"},{"name":"宮上 慎司"},{"name":"多田 美穂"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"},{"name":"湊 将典"},{"name":"稲垣 太造"},{"name":"清水 郁子"},{"name":"山口 純代"},{"name":"宮上 慎司"},{"name":"多田 美穂"},{"name":"𦚰野 修"}]},"publication_date":"2024-02-25","publication_name":{"en":"Kidney and Dialysis","ja":"腎と透析"},"volume":"Vol.96","number":"No.2","starting_page":"159","ending_page":"164","languages":["jpn"],"invited":true,"identifiers":{"doi":["10.24479/kd.0000001187"],"issn":["0385-2156"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"} line:2, {"insert":{"user_id":"6000007612","type":"misc"},"similar_merge":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1390862168254226944/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=410126","label":"url"}],"paper_title":{"en":"特集 血液透析患者の血圧を再考する 5.透析患者の至適血圧-腹膜透析も含む","ja":"特集 血液透析患者の血圧を再考する 5.透析患者の至適血圧-腹膜透析も含む"},"authors":{"en":[{"name":"Wakino Shu"},{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"Shibata Eriko"}],"ja":[{"name":"𦚰野 修"},{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"柴田 恵理子"}]},"publication_date":"2024-02-10","publication_name":{"en":"The Japanese Journal of Clinical Dialysis","ja":"臨床透析"},"volume":"Vol.40","number":"No.2","starting_page":"155","ending_page":"163","languages":["jpn"],"identifiers":{"doi":["10.19020/cd.0000002887"],"issn":["0910-5808"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"} line:3, {"insert":{"user_id":"6000007612","type":"misc"},"similar_merge":{"see_also":[{"@id":"https://search.jamas.or.jp/link/ui/2023126589","label":"url"},{"@id":"https://cir.nii.ac.jp/crid/1520013962704009344/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=401012","label":"url"}],"paper_title":{"en":"近位尿細管から見た糖尿病性腎臓病と肥満関連腎症","ja":"近位尿細管から見た糖尿病性腎臓病と肥満関連腎症"},"authors":{"en":[{"name":"Wakino Shu"},{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"}],"ja":[{"name":"𦚰野 修"},{"name":"長谷川 一宏"},{"name":"田蒔 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修"}]},"publication_date":"2022-06","publication_name":{"en":"Nephrology","ja":"腎臓内科"},"volume":"Vol.15","number":"No.6","starting_page":"659","ending_page":"664","languages":["jpn"],"identifiers":{"issn":["2435-1903"]},"misc_type":"introduction_scientific_journal"},"priority":"input_data"} line:6, {"insert":{"user_id":"6000007612","type":"misc"},"similar_merge":{"see_also":[{"@id":"https://cir.nii.ac.jp/crid/1520292395539278336/","label":"url"},{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=391084","label":"url"}],"paper_title":{"en":"ミトコンドリアとSIRT1/NMNと腎臓病","ja":"ミトコンドリアとSIRT1/NMNと腎臓病"},"authors":{"en":[{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"},{"name":"坂巻 裕介"},{"name":"Wakino Shu"}],"ja":[{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"},{"name":"坂巻 裕介"},{"name":"𦚰野 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日本内科学会"},"is_international_presentation":false},"priority":"input_data"} line:3, {"insert":{"user_id":"6000007612","type":"presentations","id":"47342512"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411405","label":"url"}],"presentation_title":{"en":"皮膚潰瘍病変患者における血中亜鉛濃度の意義","ja":"皮膚潰瘍病変患者における血中亜鉛濃度の意義"},"presenters":{"en":[{"name":"Wakino Shu"},{"name":"Shibata Eriko"},{"name":"Minato Masanori"},{"name":"Shimizu Ikuko"},{"name":"Hasegawa Kazuhiro"}],"ja":[{"name":"𦚰野 修"},{"name":"柴田 恵理子"},{"name":"湊 将典"},{"name":"清水 郁子"},{"name":"長谷川 一宏"}]},"event":{"en":"Journal of Japanese Society for Dialysis Therapy","ja":"日本透析医学会雑誌"},"publication_date":"2023-05","languages":["jpn"],"is_international_presentation":false},"priority":"input_data"} line:4, {"insert":{"user_id":"6000007612","type":"presentations","id":"47342513"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=411401","label":"url"}],"presentation_title":{"en":"AKD(acute kidney disease and disorders)を考える 透析導入阻止のためのAKD治療戦略","ja":"AKD(acute kidney disease and disorders)を考える 透析導入阻止のためのAKD治療戦略"},"presenters":{"en":[{"name":"Wakino Shu"},{"name":"Hasegawa Kazuhiro"},{"name":"Tamaki Masanori"}],"ja":[{"name":"𦚰野 修"},{"name":"長谷川 一宏"},{"name":"田蒔 昌憲"}]},"event":{"en":"Japanese Journal of Nephrology","ja":"日本腎臓学会誌"},"publication_date":"2023-05","languages":["jpn"],"is_international_presentation":false},"priority":"input_data"} line:5, {"insert":{"user_id":"6000007612","type":"presentations","id":"47243896"},"force":{"see_also":[{"@id":"https://web.db.tokushima-u.ac.jp/cgi-bin/edb_browse?EID=400928","label":"url"}],"presentation_title":{"en":"心血管内分泌の新知見 尿細管糸球体連関とDKD","ja":"心血管内分泌の新知見 尿細管糸球体連関とDKD"},"presenters":{"en":[{"name":"Hasegawa 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