Tokushima University / Educator and Researcher Directory --- Inagaki, Mai

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https://orcid.org/0000-0001-8664-6424 (ORCID)

Field of Study

Subject of Study

Book / Paper

Academic Paper (Judged Full Paper):

1.	Makoto Amifuji, Mai Inagaki, Masahito Yoshida, Takayuki Doi and Masanori Tachikawa : Characteristics of membrane transport, metabolism, and target protein binding of cyclic depsipeptide destruxin E in HeLa cells, Drug Metabolism and Pharmacokinetics, Vol.58, 101028, 2024. (Summary) Cyclic peptides have attracted attention as new modalities for drug development owing to their unique pharmacokinetic and pharmacodynamic properties. Destruxin E, a 19-membered cyclodepsipeptide, is a promising candidate drug for cancer therapy. The purpose of the present study was to clarify the molecular mechanisms underlying membrane transport, metabolism, and the binding for target molecules of destruxin E in human cervical carcinoma HeLa cells used as a model of cancer cells. The influx transport and the intracellular metabolism of destruxin E were non-saturable and saturable, respectively, at up to 10 μM. The intracellular amounts of destruxin E and destruxin E-diol after incubation of destruxin E with the cells significantly decreased at 4 °C compared to those at 37 °C. Destruxin E-diol, but not destruxin E, undergoes efflux transport out of cells via P-gp/MDR1/ABCB1 and BCRP/ABCG2. The epoxide hydrolase EPHX2 functions as a potent metabolizing enzyme that can convert the epoxide of destruxin E to the destruxin E-diol. Treatment with an EPHX2 inhibitor increased the intracellular destruxin E levels and enhanced the inhibitory activity of vacuolar type-H ATPase. These results suggest that epoxide hydrolase could be a regulatory factor for intracellular destruxin E levels and its pharmacological activity. (Link to Publication Site) ● Publication site (DOI): 10.1016/j.dmpk.2024.101028 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 39265438 ● Summary page in Scopus @ Elsevier: 2-s2.0-85203442068 (DOI: 10.1016/j.dmpk.2024.101028, PubMed: 39265438, Elsevier: Scopus)
2.	Kazuki Sone, Yuka Sakamaki, Satomi Hirose, Mai Inagaki, Masanori Tachikawa, Daisuke Yoshino and Kenichi Funamoto : Hypoxia suppresses glucose-induced increases in collective cell migration in vascular endothelial cell monolayers, Scientific Reports, Vol.14, No.1, 5164, 2024. (Summary) Blood glucose levels fluctuate during daily life, and the oxygen concentration is low compared to the atmosphere. Vascular endothelial cells (ECs) maintain vascular homeostasis by sensing changes in glucose and oxygen concentrations, resulting in collective migration. However, the behaviors of ECs in response to high-glucose and hypoxic environments and the underlying mechanisms remain unclear. In this study, we investigated the collective migration of ECs simultaneously stimulated by changes in glucose and oxygen concentrations. Cell migration in EC monolayer formed inside the media channels of microfluidic devices was observed while varying the glucose and oxygen concentrations. The cell migration increased with increasing glucose concentration under normoxic condition but decreased under hypoxic condition, even in the presence of high glucose levels. In addition, inhibition of mitochondrial function reduced the cell migration regardless of glucose and oxygen concentrations. Thus, oxygen had a greater impact on cell migration than glucose, and aerobic energy production in mitochondria plays an important mechanistic role. These results provide new insights regarding vascular homeostasis relative to glucose and oxygen concentration changes. (Keyword) Humans / Endothelial Cells / Glucose / Hypoxia / Oxygen / Cell Movement / Cell Hypoxia / Cells, Cultured (Link to Publication Site) ● Publication site (DOI): 10.1038/s41598-024-55706-1 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 38431674 ● Summary page in Scopus @ Elsevier: 2-s2.0-85186566337 (DOI: 10.1038/s41598-024-55706-1, PubMed: 38431674, Elsevier: Scopus)
3.	Mai Inagaki and Masanori Tachikawa : Transport characteristics of placenta-derived extracellular vesicles and its relevance to placenta-to-maternal tissues communication., Chemical & Pharmaceutical Bulletin, Vol.70, No.5, 324-329, 2022. (Summary) The placenta, a unique organ that helps maintain a healthy pregnancy, plays a pivotal role in maternal adaptation to pregnancy and releases extracellular vesicles (EVs), autacoids, and hormones. EVs are membranous vesicles released by all types of cells, including placental trophoblasts, which are involved in intracellular communication by delivering their cargo, such as proteins, nucleic acids, and lipids, to the targeted cells in a neighboring or distant location. Recently, an increasing number of publications have reported that EVs secreted from the placenta into maternal circulation deliver their cargo to maternal organs and mediate placenta-to-maternal communication during pregnancy. This review provides an overview of the transport mechanism of placenta-derived EVs to maternal organs. (Keyword) Extracellular Vesicles / Female / Humans / Nucleic Acids / Placenta / Pregnancy / Proteins (Link to Publication Site) ● Publication site (DOI): 10.1248/cpb.c22-00072 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 35491187 ● Search Scopus @ Elsevier (PMID): 35491187 ● Search Scopus @ Elsevier (DOI): 10.1248/cpb.c22-00072 (DOI: 10.1248/cpb.c22-00072, PubMed: 35491187)
4.	Keisuke Kitakaze, Miho Oyadomari, Jun Zhang, Yoshimasa Hamada, Yasuhiro Takenouchi, Kazuhito Tsuboi, Mai Inagaki, Masanori Tachikawa, Yoshio Fujitani, Yasuo Okamoto and Seiichi Oyadomari : ATF4-mediated transcriptional regulation protects against β-cell loss during endoplasmic reticulum stress in a mouse model., Molecular Metabolism, Vol.54, 2021. (Summary) We conclude that transcriptional regulation by ATF4 maintains β-cell identity via ISR modulation. This mechanism provides a promising target for the treatment of diabetes. (Link to Publication Site) ● Publication site (DOI): 10.1016/j.molmet.2021.101338 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 34547510 ● Summary page in Scopus @ Elsevier: 2-s2.0-85115920278 (DOI: 10.1016/j.molmet.2021.101338, PubMed: 34547510, Elsevier: Scopus)

Review, Commentary:

1.	Mai Inagaki and Masanori Tachikawa : 胎盤 - 母体臓器連関を担う細胞外小胞の輸送機構, 月刊『細胞』, Vol.56, No.3, 13-16, Mar. 2024.
2.	Masanori Tachikawa and Mai Inagaki : 脳の発達を支える血液脳関門物流システムの可塑的変化, Clinical Neuroscience, Vol.40, No.12, 1540-1543, Dec. 2022.

Proceeding of International Conference:

1.	Mai Inagaki : As a researcher, teacher, leader, manager, communicator, decision maker, caregiver, life-long learner, and as a person, Symposium to Promote Gender Balance and Diversity in Science, Diverse Perspectives of Woman in Science To Support the Next Generation, Feb. 2024.
2.	Masanori Tachikawa, Moemi Hidaka, Yuka Sakamaki, Kenichi Funamoto and Mai Inagaki : Usefulness of the human blood-brain barrier on a chip for brain-targeting drug development, Twentieth International Conference on Flow Dynamics, Nov. 2023.
3.	Mai Inagaki : Role of the blood-brain barrier transport systems for extracellular vesicles in placenta-to-brain communication, IRCMS Symposium, Rise of Diversity in Science, Sep. 2023.
4.	Eisuke Nakano, Mai Inagaki and Masanori Tachikawa : Reuptake mechanisms of human placental trophoblast cells-derived extracellular vesicles in the placental trophoblast cells, 2023 International Joint Meeting of the 23rd International Conference on Cytochrome P450 and the 38th Annual Meeting of Japanese Society for the Study of Xenobiotics, Sep. 2023.
5.	Toshiki Shimizu, Mai Inagaki, Makoto Amifuji, Shota Sasaki, Toshiro Kaneko and Masanori Tachikawa : Differential enhancement effects of non-equilibrium atmospheric pressure plasma irradiation on membrane transport in human-derived cells, 2023 International Joint Meeting of the 23rd International Conference on Cytochrome P450 and the 38th Annual Meeting of Japanese Society for the Study of Xenobiotics, Sep. 2023.
6.	Sakura Mama, Mai Inagaki, Harunori Yoshikawa, Ken Imai and Masanori Tachikawa : Determination of transporter mRNA levels in the isolated polysomes of human blood-brain barrier endothelial cells, 2023 International Joint Meeting of the 23rd International Conference on Cytochrome P450 and the 38th Annual Meeting of Japanese Society for the Study of Xenobiotics, Sep. 2023.
7.	Hiroaki Yata, Tsubasa Inokuma, Mai Inagaki, Makoto Amifuji and Masanori Tachikawa : Glucose transporter-mediated transport of newly synthesized creatine analog in human blood brain barrier endothelial cells, 2023 International Joint Meeting of the 23rd International Conference on Cytochrome P450 and the 38th Annual Meeting of Japanese Society for the Study of Xenobiotics, Sep. 2023.
8.	Kai Hirasawa, Mai Inagaki, Miku Inai, Makoto Amifuji, Hidetaka Kosako and Masanori Tachikawa : Proteomics-based comparison of transporter expression profiles in BeWo and JEG-3 cells, International Federation of Placenta Association 2023, Sep. 2023.
9.	Mai Inagaki, Eisuke Nakano and Masanori Tachikawa : Reuptake system of extracellular vesicles in human trophoblast cell line, International Federation of Placenta Association 2023, Sep. 2023.
10.	Mai Inagaki, Masahito Yoshida, Tsukasa Sugiyama, Ayaka Taii, Yu Tsuyusaki, Tomohide Goto, Takahito Wada, Takayuki Doi, Tetsuya Terasaki and Masanori Tachikawa : y+L-type amino acid transporter y+LAT2 functions as a transport system for creatine prodrug in human brain microvessel endothelial cells, The Cerebral Vascular Biology, CVB 2023 meeting, Jun. 2023.
11.	Masanori Tachikawa, Mai Inagaki, Kenichi Funamoto, Hinori Sano, Eisuke Nakano, Miku Inai, Momoko Sato and Yuka Sakamaki : Characteristics of placenta-derived extracellular vesicles (pEVs) at the human blood-brain barrier, The Cerebral Vascular Biology, CVB 2023 meeting, Jun. 2023.
12.	Masanori Tachikawa, Mai Inagaki, Hinori Sano, Momoko Sato, Hidetaka Kosako and Kenichi Funamoto : Placenta-derived Extracellular Vesicles: their unique characteristics of the Blood-Brain Barrier (BBB) transport, 25th Symposium Signal Transduction at the Blood-Brain Barriers, Jun. 2023.
13.	Masanori Tachikawa and Mai Inagaki : Placenta-derived Extracellular Vesicles: their uniqueness and characteristics of the human Blood-Brain Barrier transport., 15th International Symposium on Nanomedicine (ISMN2022), Dec. 2022.
14.	Yuka Sakamaki, Mai Inagaki, Momoko Sato, Kenichi Funamoto and Masanori Tachikawa : Visualization of extracellular vesicles transport across brain microvasculature in a human 3D blood-brain barrier chip, Nineteenth International Conference on Flow Dynamics, Nov. 2022.
15.	Mai Inagaki : Decoding the placenta-to-maternal organs communication, Special conference at IRCM, Oct. 2022.
16.	Momoko Sato, Mai Inagaki, Yuka Sakamaki, Kenichi Funamoto and Masanori Tachikawa : Reconstruction of 3D human brain microvasculature on a chip using brain endothelial cells, astrocytes and pericytes, Eighteenth International Conference on Flow Dynamics, Oct. 2021.
17.	Yuka Sakamaki, Mai Inagaki, Momoko Sato, Kenichi Funamoto and Masanori Tachikawa : Reconstruction of perfusable human 3D microvasculature on a chip as an evaluation model of cancer cell extravasation and drug transport, Eighteenth International Conference on Flow Dynamics, Oct. 2021.
18.	Momoko Sato, Yuka Sakamaki, Mai Inagaki, Kenichi Funamoto and Masanori Tachikawa : 3D Human Blood-Brain Barrier Chip for Central Nervous System Drug Development, Seventeenth International Conference on Flow Dynamics, Oct. 2020.

Proceeding of Domestic Conference:

1.	矢田浩晃, 猪熊翼, Mai Inagaki, 網藤惇 and Masanori Tachikawa : 血液脳関門グルコーストランスポーターを介して輸送されるクレアチンプロドラッグの開発, 日本薬剤学会第39年会, May 2024.
2.	平沢介, 茂谷康, Mai Inagaki, 田良島典子, 南川典昭, 小迫英尊 and Masanori Tachikawa : ヒト脳微小血管内皮細胞におけるSTING経路活性化に伴う細胞膜タンパク質変動の解明, 日本薬剤学会第39年会, May 2024.
3.	万々桜, Mai Inagaki, 吉川治孝 and Masanori Tachikawa : ヒト脳微小血管内皮細胞ポリソーム画分における輸送体mRNAの発現量解析, 日本薬剤学会第39年会, May 2024.
4.	橋本彩伽, Mai Inagaki, 田良島典子, 南川典昭 and Masanori Tachikawa : ヒト血液脳関門・くも膜関門におけるcGAS-STING経路が関わる自然免疫応答機構の役割, 日本薬剤学会第39年会, May 2024.
5.	日高萌実, 佐々木一成, Mai Inagaki and Masanori Tachikawa : グローバルプロテオミクスによる妊娠マウスの脳微小血管において発現変動するタンパク質の同定, 日本薬学会第144年会, Mar. 2024.
6.	佐野陽乃里, 山本圭, Mai Inagaki, 三木寿美, 髙塚雅貴 and Masanori Tachikawa : ヒト胎盤絨毛細胞から分泌される細胞外小胞の特性とヒト脳微小血管内皮細胞への取り込み機構, 日本薬学会第144年会, Mar. 2024.
7.	稲井美紅, Mai Inagaki, 田丸浩 and Masanori Tachikawa : スイホウガンを用いた抗ヒト脳微小血管内皮細胞抗体の作製, 日本薬学会第144年会, Mar. 2024.
8.	橋本彩伽, Mai Inagaki, 酒巻祐花, 万々桜, 梶弘和 and Masanori Tachikawa : ヒト脳関門を模倣するスフェロイド・マイクロ流体モデル, 2023年度生体医歯工学共同研究拠点成果報告会, Mar. 2024.
9.	Masanori Tachikawa and Mai Inagaki : プロテオミクス×マイクロ流体デバイスで挑むヒト血液脳関門を突破する抗体開発, 第45回日本神経組織培養研究会, Dec. 2023.
10.	髙塚雅貴, Mai Inagaki and Masanori Tachikawa : フローサイトメトリーと超解像度顕微鏡を用いたヒト脳血管内皮細胞における胎盤由来細胞外小胞の取込み特性と細胞内局在性解析, 第62回日本薬学会・日本薬剤師会・日本病院薬剤師会中国四国支部学術大会, Oct. 2023.
11.	Masanori Tachikawa and Mai Inagaki : 胎盤-脳連関機構に立脚したヒト血液脳関門・細胞外小胞輸送システムの解明, 第44回生体膜と薬物の相互作用シンポジウム, Oct. 2023.
12.	網藤惇, 今野源, 吉田将人, 土井隆行, Mai Inagaki, 寺崎哲也 and Masanori Tachikawa : 中分子環状デプシペプチドDestruxin Eの細胞内動態を規定する取り込み，排出及び代謝機構の解明, 第44回生体膜と薬物の相互作用シンポジウム, Oct. 2023.
13.	平沢介, 茂谷康, Mai Inagaki, 小迫英尊 and Masanori Tachikawa : Biotinylation by Antibody Recognition法を用いたヒト脳血管内皮細胞におけるCD147-膜輸送体複合体の分子実体解明, 日本薬剤学会第38年会, May 2023.
14.	Makoto Amifuji, Mai Inagaki, 吉田将人, 土井隆行 and Masanori Tachikawa : ヒト胎盤栄養膜細胞(BeWo細胞)におけるクレアチンプロドラッグ輸送機構の解明, 日本薬学会第143年会, Mar. 2023.
15.	繁昌志帆, 手賀悠真, 赤沼伸乙, 久保義行, Mai Inagaki, Masanori Tachikawa and 細谷健一 : ヒト脳毛細血管内皮細胞株hCMEC/D3細胞におけるcreatine輸送の特徴, 日本薬学会第143年会, Mar. 2023.
16.	Mai Inagaki, 中野瑛介 and Masanori Tachikawa : 胎盤分泌細胞外小胞のヒト胎盤栄養膜細胞への内在化機構, 日本薬学会第143年会, Mar. 2023.
17.	Terutsugu Tanizawa, Mai Inagaki, Hidetaka Kosako, Hidenori ANDO, Tatsuhiro Ishida and Masanori Tachikawa : 抗ヒト脳微小血管内皮細胞抗体の標的受容体の探索, 日本薬学会第143年会, Mar. 2023.
18.	HASHIMOTO Ayaka, Mai Inagaki, Noriko Saito-Tarashima, Shunya Yamauchi, Noriaki Minakawa and Masanori Tachikawa : 環状ジヌクレオチドによるヒト脳微小血管内皮細胞STING経路の活性化, 日本薬学会第143年会, Mar. 2023.
19.	Mai Inagaki, 佐藤桃子, 船本健一 and Masanori Tachikawa : マイクロ流体デバイス上に構築した3次元ヒト脳血管網の特性解析, 第37回日本薬物動態学会, Nov. 2022.
20.	平沢介, Mai Inagaki, 稲井美紅, 小迫英尊 and 立川正憲 : ヒト胎盤関門モデル細胞(BeWo細胞・JEG-3細胞)におけるトランスポータータンパク質発現プロファイルの比較解析, 第61回日本薬学会・日本薬剤師会・日本病院薬剤師会中国四国支部学術大会, Nov. 2022.
21.	今井健, Mai Inagaki, 佐藤桃子, 船本健一 and Masanori Tachikawa : マイクロ流体デバイスを用いた3次元ヒト脳血管網の再構築, 第61回日本薬学会・日本薬剤師会・日本病院薬剤師会中国四国支部学術大会, Nov. 2022.
22.	中野瑛介, Mai Inagaki and Masanori Tachikawa : ヒト胎盤栄養膜細胞(BeWo細胞)が分泌する細胞外小胞の分泌元細胞への再取り込み機構, 第16回次世代を担う若手のための医療薬科学シンポジウム, Oct. 2022.
23.	Masanori Tachikawa and Mai Inagaki : マイクロ流体デバイスを用いた三次元ヒト血液脳関門の再構築と特性解析, 第44回神経組織培養研究会, Sep. 2022.
24.	酒巻祐花, Mai Inagaki, 佐藤桃子, 中野瑛介, 船本健一 and Masanori Tachikawa : マイクロ流体デバイスを用いた三次元血管網モデルの構築と胎盤由来細胞外小胞の動態可視化, 日本薬剤学会第37年会, May 2022.
25.	杉下友香, Mai Inagaki, 馬渡一諭, 小迫英尊, 三宅雅人, 親泊政一 and Masanori Tachikawa : ヒト脳血管内皮細胞(hCMEC/D3細胞)におけるCD147-トランスポーター複合体の役割, 日本薬剤学会第37年会, May 2022.
26.	Masanori Tachikawa and Mai Inagaki : ヒト血液脳関門における細胞外小胞輸送システムの多様性と特異性, 日本薬学会第142年会, Mar. 2022.
27.	Momoko Sato, Mai Inagaki, YUKA Sakamaki, 船本健一 and Masanori Tachikawa : マイクロ流体デバイスを用いた三次元ヒト脳微小血管網の構築, 日本薬学会第142年会, Mar. 2022.
28.	Mai Inagaki, Hinori Sano, Miku Inai, 赤沼伸乙, 細谷健一 and Masanori Tachikawa : ヒト脳血管内皮細胞における胎盤栄養膜細胞から分泌される細胞外小胞の輸送特性, 第36回日本薬物動態学会, Nov. 2021.
29.	高橋駿太, Mai Inagaki, 野口幸希, 西村友宏 and 登美斉俊 : マウス胎盤におけるPGE2受容体の発現解析, 第29回日本胎盤学会学術集会, Nov. 2021.
30.	Mai Inagaki, Tsukasa Sugiyama, Momoko Sato, 吉田将人, 土井隆行, 和田敬仁, 新保裕子, 露崎悠, 後藤知英, 寺崎哲也 and Masanori Tachikawa : ヒト血液脳関門モデルhCMEC/D3細胞におけるクレアチンプロドラッグの輸送特性, 第42回生体膜と薬物の相互作用シンポジウム, Oct. 2021.
31.	北風圭介, 親泊美帆, 張君, 濱田良真, 竹之内康広, 坪井一人, Mai Inagaki, Masanori Tachikawa, 藤谷与士夫, 岡本安雄 and 親泊政一 : ATF4を介した転写制御は小胞体ストレスによる膵β細胞の喪失を防ぐ, 第62回日本生化学会中国・四国支部例会, Sep. 2021.
32.	Mai Inagaki and Masanori Tachikawa : 胎盤治療の基盤としての胎盤関門・細胞外小胞輸送システム, 日本薬剤学会第36年会, May 2021.
33.	Masanori Tachikawa and Mai Inagaki : 末梢から中枢への情報伝達制御装置としての血液脳関門物流システムの役割と脳への薬物送達, 日本薬剤学会第36年会, May 2021.
34.	MIKU Inai, Mai Inagaki, 赤沼伸乙, 細谷健一 and Masanori Tachikawa : マイクロRNAの妊娠マウス脳への分布とヒト脳血管内皮細胞における胎盤由来細胞外小胞を介した輸送, 日本薬剤学会第36年会, May 2021.
35.	EISUKE Nakano, MIKU Inai, Mai Inagaki and Masanori Tachikawa : ヒト胎盤栄養膜細胞(BeWo細胞)由来細胞外小胞の胎盤への再取り込み輸送機構の解明, 日本薬剤学会第36年会, May 2021.
36.	HINORI Sano, MIKU Inai, Mai Inagaki and Masanori Tachikawa : ヒト脳血管内皮細胞(hCMEC/D3細胞)におけるヒト胎盤絨毛細胞株BeWo細胞から分泌される細胞外小胞の輸送特性, 日本薬剤学会第36年会, May 2021.
37.	YUKA Sakamaki, Mai Inagaki, Momoko Sato, 船本健一 and Masanori Tachikawa : マイクロ流体デバイスを用いた灌流性を有する三次元ヒト微小血管網の再構築, 日本薬剤学会第36年会, May 2021.
38.	Tohru Kinoshita, Daiki Ohno, 小迫英尊, Mai Inagaki and Masanori Tachikawa : 網羅的プロテオミクスを用いたヒト脳毛細血管内皮細胞への内在化活性を示す脳転移性メラノーマSK-Mel-28由来細胞外小胞の特性解析, 日本薬剤学会第36年会, May 2021.
39.	Makoto Amifuji, 今野源, 吉田将人, 土井隆行, Mai Inagaki, 寺崎哲也 and Masanori Tachikawa : 中分子環状デプシペプチドDestruxin Eの細胞膜輸送・細胞内代謝・分子標的V-ATPase阻害における立体特異性の解明, 日本薬剤学会第36年会, May 2021.
40.	Tsukasa Sugiyama, Mai Inagaki, Momoko Sato, 吉田将人, 土井隆行, 和田敬仁, 新保裕子, 露崎悠, 後藤知英, 寺崎哲也 and Masanori Tachikawa : ヒト脳血管内皮細胞におけるクレアチンプロドラッグのクレアチントランスポーター非依存的輸送の実証, 日本薬剤学会第36年会, May 2021.
41.	Mai Inagaki, 佐野陽乃里, 中野瑛介, 登美斉俊 and Masanori Tachikawa : ヒト胎盤絨毛細胞株BeWo細胞由来エクソソームのヒト脳血管内皮細胞 (hCMEC/D3)への内在化, 日本薬学会第141年会, Mar. 2020.

Grants-in-Aid for Scientific Research (KAKEN Grants Database @ NII.ac.jp)

血液循環-胎盤分泌膜小胞を主軸としたヒト胎盤関門輸送機能の個別化予測法の基盤構築 (Project/Area Number: 23K14385 )
Placenta-to-brain communication-related transport characteristics of placental trophoblast-derived exosomes in the blood-brain barrier endothelial and parenchymal cells (Project/Area Number: 21K15314 )
Human endogenous retrovirus envelope protein-mediated transport system of placenta-derived exosomes in human placental trophoblasts (Project/Area Number: 20K22708 )
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Assistant Professor : Inagaki, Mai

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