Tokushima University / Educator and Researcher Directory --- Matsuda, Taku

Field of Study

Subject of Study

Book / Paper

Academic Paper (Judged Full Paper):

1.	Hiroshi Koyama, Kohhei Nakajima, Izumi Yamaguchi, Noriya Enomoto, Taku Matsuda, Hiroshi Kagusa, T. Keiko Kitazato and Yasushi Takagi : Cystatin C regulates cell division in primary central nervous system lymphoma, Scientific Reports, 16, 1, 2026. (Summary) Primary central nervous system lymphoma (PCNSL) is associated with cerebral inflammation characterized by elevated neutrophils, monocytes, platelets, and lymphocytes. However, the comprehensive profile of inflammation-related molecules in PCNSL remains unclear. In this study, we conducted proteomic analysis of PCNSL tissue compared with normal brain to identify factors influencing neuroinflammation and potential therapeutic targets. In contrast to the general elevation of inflammatory molecules, cystatin C (Cyst C) showed markedly reduced expression. This result was confirmed by western blotting and mRNA analysis. Immunohistochemistry revealed widespread Cyst C positivity in glial cells of the normal brain, whereas PCNSL tissue was dominated by Cyst C-negative B cells. To further investigate its role, cultured PCNSL cells were treated with recombinant human Cyst C. Treatment reduced viable cell counts in a dose- and time-dependent manner over 4 days without inducing cell death, indicating inhibition of cell division. Immunohistochemistry demonstrated that Cyst C increased p21 expression and decreased cyclin-dependent kinase 1 and Cyclin B. These findings identify Cyst C as a regulator of PCNSL cell division, highlighting it as a potential therapeutic target. (Keyword) Cell division / Cyclin-dependent kinases / Cystatin C / Neuroinflammation / p21 / Primary central nervous system lymphoma (Link to Publication Site) ● Publication site (DOI): 10.1038/s41598-026-44039-w (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 41826426 ● Summary page in Scopus @ Elsevier: 2-s2.0-105040825194 (DOI: 10.1038/s41598-026-44039-w, PubMed: 41826426, Elsevier: Scopus)
2.	Kaito Suzuki, Taku Matsuda, Keijiro Hara, Kazumi Nakamura, Megumi Kuno, Kazuhisa Miyake, Masaaki Korai, Nori Sato, Tetsuya Matsuura and Yasushi Takagi : Posttraumatic cerebrospinal fluid leak with cognitive dysfunction, especially visual memory disturbance: illustrative case, Journal of Neurosurgery. Case Lessons, 11, 10, 2026. (Summary) Cerebrospinal fluid (CSF) leaks are associated with various clinical and neurological manifestations. Although cognitive dysfunction has been reported in patients with a CSF leak, its detailed characteristics and cognitive profile remain unclear. A 17-year-old male patient developed progressive cognitive decline after minor head trauma. The initial neuroimaging findings were unremarkable; however, over several months, he exhibited memory impairment, academic decline, and personality changes. Neuropsychological testing has demonstrated significant deficits in visual memory, reduced working memory, and reduced processing speed. A detailed interview revealed a subtle orthostatic headache that had been present since the head injury, prompting further evaluation. Spinal MRI and CT myelography confirmed a CSF leak in the cervical spine. The patient underwent placement of a blind lumbar epidural blood patch, which resulted in immediate headache relief and gradual cognitive improvement, particularly in visual memory. CSF leaks can manifest primarily as cognitive dysfunction, even without typical headache or imaging findings, leading to diagnostic delays and potential misdiagnosis of psychiatric illnesses. Visual memory impairment might represent a characteristic and underrecognized feature of a CSF leak. Comprehensive neuropsychological assessment with domain-specific testing is essential for accurate diagnosis of atypical presentations. https://thejns.org/doi/10.3171/CASE25939. (Link to Publication Site) ● Publication site (DOI): 10.3171/CASE25939 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 41802294 ● Search Scopus @ Elsevier (PMID): 41802294 ● Search Scopus @ Elsevier (DOI): 10.3171/CASE25939 (DOI: 10.3171/CASE25939, PubMed: 41802294)
3.	Hiroshi Koyama, Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Joji Fujikawa, Keisuke Shinohara, Yoshihiko Nishida and Yasushi Takagi : Rare abdominal manifestations in a patient with Parkinson's disease after deep-brain stimulation of the subthalamic nucleus: A case report, Case Reports in Neurology, 18, 1, 128-133, 2026. (Summary) Abdominal pain is an occasionally observed non-motor symptom during "off" periods in Parkinson's disease (PD), often associated with end-of-dose wearing-off (EODWO). However, severe abdominal manifestations such as painful abdominal contraction (PAC) are rare, and their pathogenesis remains unclear. We report a unique case in which both PAC and belly dancer's dyskinesia (BDD) emerged following subthalamic nucleus deep brain stimulation (STN-DBS). A 74-year-old woman with PD underwent bilateral STN-DBS. Following a postoperative reduction in her levodopa dosage, she developed continuous abdominal pain with a downward-pulling sensation, which was diagnosed as PAC. Symptoms worsened in upright positions and improved supine. With an increase in STN-DBS stimulation intensity, she subsequently developed wave-like involuntary contractions of the abdominal wall muscles, consistent with BDD, while her PAC remained unchanged. STN-DBS adjustments and dopaminergic agents other than levodopa provided only partial relief, with PAC showing minimal improvement. Restoration of the preoperative levodopa dosage led to marked alleviation of PAC. Additional STN stimulation effectively managed recurrent EODWO-related pain. Four months postoperatively, motor symptoms improved significantly, with only mild peak-dose BDD remaining. This case illustrates that PAC may result from reduced baseline dopamine levels and may not respond to STN-DBS alone. The co-occurrence of PAC and BDD suggests a complex interplay between motor and non-motor circuits, possibly involving spinal plasticity and central pattern generators. Clinicians should consider that some cases of severe abdominal pain in PD may represent distinct non-motor phenomena that appear to respond preferentially to levodopa administration. (Link to Publication Site) ● Publication site (DOI): 10.1159/000550603 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 41777619 ● Search Scopus @ Elsevier (PMID): 41777619 ● Search Scopus @ Elsevier (DOI): 10.1159/000550603 (DOI: 10.1159/000550603, PubMed: 41777619)
4.	Ryoma Morigaki, Taku Matsuda, Kazuhisa Miyake and Yasushi Takagi : 若手に知ってほしい不随意運動症基礎研究の魅力, Functional Neurosurgery, 63, 26-31, 2025. (Tokushima University Institutional Repository) ● Metadata: 2012835 (Tokushima University Institutional Repository: 2012835)
5.	Hiroshi Oomae, Taku Matsuda, Maki Fujimoto, Yuka Nakanowatari, Megumi Yamada, Nori Sato, Kimiko Umemura, Ryoma Morigaki, Tetsuya Matsuura and Yasushi Takagi : Safety and Selection Criteria of Single-joint Hybrid Assistive Limb for Upper Limb Paralysis After Stroke: A Phase I Trial, Neurologia Medico-Chirurgica, 65, 1, 15-21, 2024. (Summary) Robotic rehabilitation is a high-intensity intervention for upper limb paralysis after a stroke. This study explored the safety and feasibility of using a single-joint hybrid assistive limb (HAL-SJ) exoskeletal device for upper limb paralysis in patients with acute stroke. In total, 11 patients with stroke (6 with moderate paralysis and 5 with severe paralysis) were enrolled between October 2021 and October 2023 in a stroke care unit. The patients underwent HAL training 3 times a week for 6 sessions. No serious adverse events related to HAL-SJ occurred, and participants demonstrated significant improvements in the Fugl-Meyer Assessment Upper Extremity (FMA-UE) and Action Research Arm Test (ARAT) (pre- vs. post-intervention; p < 0.05). The minimal clinically important difference (MCID) for FMA-UE was surpassed in 5 patients (83.3%) with moderate paralysis and 2 (40.0%) with severe paralysis. Regarding ARAT, 4 patients (66.7%) with moderate paralysis exceeded the MCID, whereas none (0.0%) with severe paralysis did. These findings suggest that HAL-SJ is both safe and feasible, particularly for individuals with moderate paralysis. (Keyword) Humans / Male / Stroke Rehabilitation / Female / Aged / Middle Aged / Paralysis / Upper Extremity / Exoskeleton Device / Stroke / Feasibility Studies (Link to Publication Site) ● Publication site (DOI): 10.2176/jns-nmc.2024-0137 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 39581619 ● Search Scopus @ Elsevier (PMID): 39581619 ● Search Scopus @ Elsevier (DOI): 10.2176/jns-nmc.2024-0137 (DOI: 10.2176/jns-nmc.2024-0137, PubMed: 39581619)
6.	Hiroshi Ohmae, Taku Matsuda, Maki Fujimoto, Yuka Nakanowatari, Megumi Yamada, Nori Sato, Kimiko Umemura, Ryoma Morigaki, Tetsuya Matsuura and Yasushi Takagi : Safety and Selection Criteria of Single-joint Hybrid Assistive Limb for Upper Limb Paralysis After Stroke: A Phase I Trial., Neurologia Medico-Chirurgica, 65, 1, 15-21, 2024. (Summary) Robotic rehabilitation is a high-intensity intervention for upper limb paralysis after a stroke. This study explored the safety and feasibility of using a single-joint hybrid assistive limb (HAL-SJ) exoskeletal device for upper limb paralysis in patients with acute stroke. In total, 11 patients with stroke (6 with moderate paralysis and 5 with severe paralysis) were enrolled between October 2021 and October 2023 in a stroke care unit. The patients underwent HAL training 3 times a week for 6 sessions. No serious adverse events related to HAL-SJ occurred, and participants demonstrated significant improvements in the Fugl-Meyer Assessment Upper Extremity (FMA-UE) and Action Research Arm Test (ARAT) (pre- vs. post-intervention; p < 0.05). The minimal clinically important difference (MCID) for FMA-UE was surpassed in 5 patients (83.3%) with moderate paralysis and 2 (40.0%) with severe paralysis. Regarding ARAT, 4 patients (66.7%) with moderate paralysis exceeded the MCID, whereas none (0.0%) with severe paralysis did. These findings suggest that HAL-SJ is both safe and feasible, particularly for individuals with moderate paralysis. (Keyword) Humans / Male / Female / Middle Aged / Stroke Rehabilitation / Aged / Paralysis / Upper Extremity / Stroke / Exoskeleton Device / Adult / Patient Selection / Feasibility Studies (Tokushima University Institutional Repository) ● Metadata: 2013066 (Link to Publication Site) ● Publication site (DOI): 10.2176/jns-nmc.2024-0137 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 39581619 ● Search Scopus @ Elsevier (PMID): 39581619 ● Search Scopus @ Elsevier (DOI): 10.2176/jns-nmc.2024-0137 (Tokushima University Institutional Repository: 2013066, DOI: 10.2176/jns-nmc.2024-0137, PubMed: 39581619)
7.	Ryoma Morigaki, Ryosuke Miyamoto, Kazuhisa Miyake, Hiroshi Omae, Kaito Suzuki, Taku Matsuda, Hiroshi Koyama, Emi Ishitani, Yuishin Izumi and Yasushi Takagi : Striking Efficacy of Pallidal Deep Brain Stimulation in a Patient with Predominant Abductor Laryngeal Dystonia: A Case Report., Movement Disorders Clinical Practice, 11, 11, 1462-1464, 2024. (Link to Publication Site) ● Publication site (DOI): 10.1002/mdc3.14189 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 39132907 ● Search Scopus @ Elsevier (PMID): 39132907 ● Search Scopus @ Elsevier (DOI): 10.1002/mdc3.14189 (DOI: 10.1002/mdc3.14189, PubMed: 39132907)
8.	Taku Matsuda, Ryoma Morigaki, Hiroaki Hayasawa, Hiroshi Koyama, Teruo Oda, Kazuhisa Miyake and Yasushi Takagi : Striatal parvalbumin interneurons are activated in a mouse model of cerebellar dystonia, Disease Models & Mechanisms, 17, 5, 2024. (Summary) Dystonia is thought to arise from abnormalities in the motor loop of the basal ganglia; however, there is an ongoing debate regarding cerebellar involvement. We adopted an established cerebellar dystonia mouse model by injecting ouabain to examine the contribution of the cerebellum. Initially, we examined whether the entopeduncular nucleus (EPN), substantia nigra pars reticulata (SNr), globus pallidus externus (GPe) and striatal neurons were activated in the model. Next, we examined whether administration of a dopamine D1 receptor agonist and dopamine D2 receptor antagonist or selective ablation of striatal parvalbumin (PV, encoded by Pvalb)-expressing interneurons could modulate the involuntary movements of the mice. The cerebellar dystonia mice had a higher number of cells positive for c-fos (encoded by Fos) in the EPN, SNr and GPe, as well as a higher positive ratio of c-fos in striatal PV interneurons, than those in control mice. Furthermore, systemic administration of combined D1 receptor agonist and D2 receptor antagonist and selective ablation of striatal PV interneurons relieved the involuntary movements of the mice. Abnormalities in the motor loop of the basal ganglia could be crucially involved in cerebellar dystonia, and modulating PV interneurons might provide a novel treatment strategy. (Keyword) Cerebellar dystonia / Cholinergic interneuron / Dopamine D1 receptor agonist / Dopamine D2 receptor antagonist / Parvalbumin interneuron (Link to Publication Site) ● Publication site (DOI): 10.1242/dmm.050338 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 38616770 ● Summary page in Scopus @ Elsevier: 2-s2.0-85193103310 (DOI: 10.1242/dmm.050338, PubMed: 38616770, Elsevier: Scopus)
9.	Hiroshi Kagusa, Yamaguchi Izumi, Shono Kenji, Mizobuchi Yoshifumi, Shikata Eiji, Taku Matsuda, Takeshi Miyamoto, Keijiro Hara, Kitazato T Keiko, Yoshihiro Uto, Kanematsu Yasuhisa and Yasushi Takagi : Differences in amyloid-β and tau/p-tau deposition in blood-injected mouse brains using micro-syringe to mimic traumatic brain microhemorrhages, Journal of Chemical Neuroanatomy, 130, 102258, 2023. (Summary) Cerebral microbleeds (CMBs) due to traumatic brain injuries (TBI) have been shown to lead to cognitive decline and impairment. CMBs caused by TBI may be associated with pathophysiological mechanisms involving inflammation and the accumulation of amyloid-β (Aβ), tau, and phosphorylated tau (p-tau), contributing to cognitive abnormalities. However, their relationships remain unclear. To test our hypothesis that Aβ, tau, and p-tau are accumulated and regulated separately in mice with injuries imitating CMBs from TBI, we studied. Seven-week-old C57BL/6 male mice were injected with 15 μL of heparinized autologous blood or saline by micro-syringe into the front lobe. Expression profiles and regulation of Aβ, tau, and p-tau were assessed immunohistochemically over time. On day 7 after blood injection, Iba-1 and S100B cells in damaged cortex adjacent to the injection site were higher than saline injection group and non-injected sham. On days 3-14, Aβ deposition were gradually increased but normalized by day 28. In contrast, tau/p-tau deposition gradually increased during days 14-28 and dispersed along the corticomedullary junction adjacent to hem deposits, indicating different expression profiles from Aβ. Deposits of Aβ, but not tau/p-tau, were phagocytosed by CD163 macrophages increased by Gc-protein macrophage-activating factor during days 7-28, suggesting different mechanisms of deposition and regulation between Aβ and tau/p-tau. Deposition and regulation differ between Aβ and tau/p-tau in mice with injuries mimicking CMBs from TBI. Further clarification of relationships between the pathologies of cognitive impairment and their neurodegenerative consequences is needed. (Keyword) Amyloid-β / Cognitive impairment / Tauopathy / Traumatic cerebral microbleed (Tokushima University Institutional Repository) ● Metadata: 2012257 (Link to Publication Site) ● Publication site (DOI): 10.1016/j.jchemneu.2023.102258 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 36925083 ● Search Scopus @ Elsevier (PMID): 36925083 ● Search Scopus @ Elsevier (DOI): 10.1016/j.jchemneu.2023.102258 (Tokushima University Institutional Repository: 2012257, DOI: 10.1016/j.jchemneu.2023.102258, PubMed: 36925083)
10.	Hiroshi OMAE, Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda and Yasushi Takagi : 高齢パーキンソン病患者に対する視床下核脳深部刺激療法による変化の検討, Geriatric Neurosurgery, 36, 43-48, 2023.

Proceeding of International Conference:

1.	Keijiro Hara, Taku Matsuda, Kazuhisa Miyake and Yasushi Takagi : Two Cases of CSF Leakage in Young Adults with Higher Brain Dysfunction as the Chief Complaint, The 11th Joint Neurosurgical Convention, Feb. 2026.
2.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Joji Fujikawa, Keisuke Shinohara and Yasushi Takagi : Basal ganglia-cerebellar interplay in dystonia: Implication for stereotactic neurosurgical treatment., First Asian Deep Brain Stimulation Forum (ADBS2025), Sep. 2025.
3.	Taku Matsuda, Ryoma Morigaki, Hiroaki Hayasawa, Hiroshi Koyama, Teruo Oda, Kazuhisa Miyake and Yasushi Takagi : Striatal parvalbumin interneurons, not cholinergic interneurons, are activated in a mouse model of cerebellar dystonia, NEURO2024, Fukuoka, Jul. 2024.

Proceeding of Domestic Conference:

1.	Taku Matsuda and Yasushi Takagi : チームで挑む急性期病院における脳卒中患者へのHybrid Assistive Limbの運用, STROKE2026, Mar. 2026.
2.	Kazuhisa Miyake, 多田里穂, Taku Matsuda, Ryoma Morigaki and Yasushi Takagi : Cathode刺激にAnode刺激を追加することで改善が認められたジストニアの2症例, 第65回日本定位・機能神経外科学会, Feb. 2026.
3.	Taku Matsuda, Ryoma Morigaki, Kazuhisa Miyake, 岡久哲也, 大前博司, Tetsuya Matsuura and Yasushi Takagi : ジストニア周術期におけるリハビリテーション部との多職種連携, 第65回日本定位・機能神経外科学会, Feb. 2026.
4.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Joji Fujikawa, Keisuke Shinohara, Yasushi Takagi, Graybiel M. Ann and Satoshi Goto : 古典的方法を改良したストリオソームの研究ーハンチントン病研究を例としてー, 第65回日本定位・機能神経外科学会, Feb. 2026.
5.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Joji Fujikawa, Keisuke Shinohara and Yasushi Takagi : 病態回路に応じた定位治療の最適化:ジストニアに対する新たな視点, 第65回日本定位・機能神経外科学会, Feb. 2026.
6.	多田里穂, Taku Matsuda, Ryoma Morigaki, Kazuhisa Miyake and Yasushi Takagi : 淡Covie-19罹患後のジストニアに対して淡蒼球視床路凝固術・淡蒼球凝固術を行った全身性ジストニアの1例, 第65回日本定位・機能神経外科学会, Feb. 2026.
7.	多田里穂, Taku Matsuda, Ryoma Morigaki, Kazuhisa Miyake and Yasushi Takagi : 淡蒼球視床路凝固術・淡蒼球凝固術を行ったジストニアの3症例, 第100回日本脳神経外科学会中国四国支部学術集会, Dec. 2025.
8.	OMAE Hiroshi, Taku Matsuda, 藤本真希, 桃谷寛太, 松浦哲太 and Yasushi Takagi : HAL医療用単関節タイプによる急性期脳卒中患者への介入:上肢機能と半側空間無視への効果―4症例の検討―, 第1回日本サイバニクス医療研究会, Nov. 2025.
9.	Masaaki Korai, Taku Matsuda, Keijiro Hara, 鈴木海斗, Kazuhisa Miyake and Yasushi Takagi : 外傷後の高次脳機能障害の精査で脳脊髄液漏出症と診断された一例, 第53回日本頭痛学会総会, Nov. 2025.
10.	Taku Matsuda, Hiroshi Kagusa, Izumi Yamaguchi, Takeshi Miyamoto, Masaaki Korai, Kenji Shimada and Yasushi Takagi : 脳卒中急性期患者における両下肢タイプHybrid Assistive Limbを利用した歩行訓練の即時効果の検討, 日本脳神経外科学会第84回学術総会, Oct. 2025.
11.	Kazuhisa Miyake, Taku Matsuda, Ryoma Morigaki and Yasushi Takagi : パーキンソン病に対し脳深部刺激療法を導入し，その後，ホスレボドパ・ホスカルビドパ水和物持続皮下注射療法を導入した2症例の経験, 第84回日本脳神経外科総会, Oct. 2025.
12.	Taku Matsuda and Yasushi Takagi : 高次脳機能障害の精査で明らかとなった脳脊髄液漏出症の一例, 第62回日本リハビリテーション医学会学術集会, Jun. 2025.
13.	Taku Matsuda, Kimiko Umemura, Nori Sato, 森脇好乃美 and Tetsuya Matsuura : 高次脳機能障害の精査で明かとなった脳脊髄液漏出症の一例, 第62回日本リハビリテーション医学会学術集会, Jun. 2025.
14.	Ryoma Morigaki, Ryosuke Miyamoto, Kazuhisa Miyake, Taku Matsuda, Yasushi Takagi and Yuishin Izumi : 基底核と小脳，変わりゆくジストニアの外科治療, 第66回日本神経学会学術大会, May 2025.
15.	Taku Matsuda, Hiroshi Kagusa, Takeshi Miyamoto, Masaaki Korai, Kenji Shimada, Nori Sato, Kimiko Umemura, Tetsuya Matsuura and Yasushi Takagi : 急性期脳卒中患者における両下肢タイプHybrid Assistive Limbの即時効果の検討, STROKE2025, Mar. 2025.
16.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Joji Fujikawa, Keisuke Shinohara and Yasushi Takagi : 首下がり全身性ジストニアの一例, 第7回中国四国機能外科懇話会, Mar. 2025.
17.	Keijiro Hara, Taku Matsuda, Kaito Suzuki, Kazuhisa Miyake and Yasushi Takagi : 外傷後の高次脳機能障害の精査で脳脊髄液漏出症と診断された一例, 第48回日本脳神経外傷学会, Feb. 2025.
18.	Kazuhisa Miyake, Taku Matsuda, Ryoma Morigaki and Yasushi Takagi : 手術中の既存電極の変化に関して，2症例の検討, 第64回日本定位・機能神経外科学会, Feb. 2025.
19.	Taku Matsuda, Ryoma Morigaki, Kazuhisa Miyake and Yasushi Takagi : 小脳性ジストニアモデルマウスの線条体ではパルブアルブミン陽性介在ニューロンの活性化がみられる, 第64回日本定位・機能神経外科学会, Feb. 2025.
20.	Ryoma Morigaki, Taku Matsuda, Keisuke Shinohara, Joji Fujikawa, Kazuhisa Miyake and Yasushi Takagi : ジストニアにおける線条体ストリオソーム構造と小脳の異常, 第64回日本定位・機能神経外科学会, Feb. 2025.
21.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Ryosuke Miyamoto, OMAE Hiroshi, Yuishin Izumi and Yasushi Takagi : ジストニアに対する両側淡蒼球刺激術における最近の刺激方法, 第64回日本定位・機能神経外科学会, Feb. 2025.
22.	Hiroshi OMAE, 藤本真希, Taku Matsuda, Ryoma Morigaki and Yasushi Takagi : 急性期脳卒中患者に対するSinglejoint type of Hybrid Assistive Limbの効果 ―ケースシリーズ研究―, 第58回日本作業療法学会, Nov. 2024.
23.	Taku Matsuda, Keijiro Hara, 鈴木海斗, Kazuhisa Miyake and Yasushi Takagi : A case of cerebrospinal fluid leaks diagnosed by examination of posttraumatic higher brain dysfunction, 日本脳神経外科学会第83回学術総会, Oct. 2024.
24.	Taku Matsuda, Keijiro Hara, Kaito Suzuki, Kazuhisa Miyake and Yasushi Takagi : 外傷後の高次脳機能障害の精査で脳脊髄液漏出症と診断された一例, 23回舞連カンファレンス, Aug. 2024.
25.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda and Yasushi Takagi : 赤核振戦に伴う痛みに対して著効した脳深部刺激療法の一例, 第6回中国四国機能神経外科懇話会, Mar. 2024.
26.	Ryoma Morigaki, Kazuhisa Miyake, Ryosuke Miyamoto, 大前博司, Taku Matsuda, Hiroshi Koyama, Yuishin Izumi and Yasushi Takagi : GPi-DBSが著効した外転型痙攣性発声障害の一例, 第63回日本定位機能外科学会, Feb. 2024.
27.	Taku Matsuda, Hiroshi Kagusa, Nori Sato, 梅山公子, Tetsuya Matsuura, Yasuhisa Kanematsu and Yasushi Takagi : HALによる歩行訓練で歩容の改善を得られた深部感覚障害による歩行障害の一例, 第12回日本脳神経HAL研究会, Dec. 2023.

Et cetera, Workshop:

1.	Ryoma Morigaki, Kazuhisa Miyake, Taku Matsuda, Hiroshi Koyama and Yasushi Takagi : 最近のジストニア手術症例の検討, 第50回徳島脳神経外科談話会, Dec. 2024.

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Designated Assistant Professor : Matsuda, Taku

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