徳島大学 / 教育研究者総覧 --- 田代征記

専門分野

研究テーマ

著書・論文

著書:

1.	六車直樹, 伊東進, 三宅秀則, 松村敏信, 田代征記 : 胆嚢癌におけるムチン発現と患者予後に関する検討, 財団法人日本消化器病学会, 東京, 2001年1月.
2.	北川哲也, 北市隆, 堀隆樹, 吉栖正典, 田埜和利, 加藤逸夫, 余喜多史郎, 田代征記, 門田康正, 香川征 : 静脈およびリンパ管疾患と外科, --- 悪性腫瘍による下大静脈症候群の検査，診断，治療方針，治療，予後 ---, 日本アクセル・シュプリンガー出版, 1997年2月.

学術論文(審査論文):

1.	濵田吉則, 嵩原裕夫, 安藤久美, 糸井隆夫, 神澤輝実, 越永従道, 須山正文, 信川文誠, 藤井秀樹, 渡辺康宏, 田代征記 : 小児胆管径の基準値からみた胆管拡張の定義の問題点, 胆と膵, Vol.31, No.11, 1269-1272, 2010年. (キーワード) 和文論文 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1573105975973089152 (CiNii: 1573105975973089152)
2.	森根裕二, 島田光生, 久山寿子, 高松英夫, 田代征記 : 全国集計からみた先天性胆道拡張症, 膵・胆管合流異常の胆道癌発生率とその特徴, 胆と膵, Vol.31, No.11, 1293-1299, 2010年. (キーワード) 和文論文 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1572824500996378752 (CiNii: 1572824500996378752)
3.	Hirofumi Kanemura, Kenji Kusumoto, Hidenori Miyake, Seiki Tashiro, Kazuhito Rokutan and Mitsuo Shimada : Geranylgeranylacetone prevents acute liver damage after massive hepatectomy in rats through suppression of a CXC chemokine GRO1 and induction of heat shock proteins., Journal of Gastrointestinal Surgery, Vol.13, No.1, 66-73, 2008. (要約) BACKGROUND AND METHODS: Acute liver failure after massive hepatectomy remains a challenging problem. In this study, using a microarray designed to monitor the side effects of drugs, we examined changes in gene expression in the remnant liver during the 24 h after hepatectomy and the effects of a nontoxic heat shock protein (HSP) 70 inducer, geranylgeranylacetone (GGA), after 90% hepatectomy in rats. RESULTS: A single oral administration of 100 mg/kg GGA significantly suppressed the release of aminotransferases and improved survival compared with vehicle administration. The hepatectomy upregulated 74 genes and downregulated 95. Interestingly, ten cytokine genes were upregulated, while no cytokine-related gene was downregulated. Among the ten cytokine genes, a potent chemoattractant for neutrophils, GRO1, was most rapidly and markedly upregulated after 90% hepatectomy. GGA effectively suppressed the up-regulation of GRO1 messenger ribonucleic acid, and this was validated by Northern hybridization. Microarray and immunoblot analyses showed that, in addition to HSP70 and HSP27, GGA preferentially induced an endoplasmic reticulum chaperone, BIP. CONCLUSION: Considering hemodynamic and metabolic overloading as a primary cause of acute lever failure, the ER stress response enhanced by GGA may also play an important role in the prevention of overload-induced liver damage. (キーワード) Animals / Anti-Ulcer Agents / Autoradiography / Blotting, Western / Chemokine CXCL1 / Disease Models, Animal / Diterpenes / Dose-Response Relationship, Drug / 遺伝子発現 (gene expression) / HSP70 Heat-Shock Proteins / Hepatectomy / Liver Failure, Acute / 男性 (male) / Microarray Analysis / RNA (RNA) / Rats / Rats, Wistar / Reverse Transcriptase Polymerase Chain Reaction (出版サイトへのリンク) ● Publication site (DOI): 10.1007/s11605-008-0604-x (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 18683011 ● Summary page in Scopus @ Elsevier: 2-s2.0-57849122454 (DOI: 10.1007/s11605-008-0604-x, PubMed: 18683011, Elsevier: Scopus)
4.	Otsuka Toshihiro, Keisuke Izumi, Itsuo Tokunaga, Takako Gotohda, Ipposhi Kaneshige, Yoshiharu Takiguchi, Kaneda Shinya, Nobuo Satake, Takamasa Ohnishi, Seiki Tashiro and Mitsuo Shimada : Prevention of lethal hepatic injury in Long-Evans Cinnamon(LEC) rats by D-galactosamine hydrochloride, The Journal of Medical Investigation : JMI, Vol.53, No.1-2, 81-86, 2006. (要約) Repeated injections of D-galactosamine hydrochloride (GalN) increase the survival rate of Long-Evans Cinnamon (LEC) rats, an animal model of Wilson's disease. The aim of the present study was to investigate the mechanism of GalN for prevention of spontaneous lethal hepatic injury in LEC rats. Male LEC rats were given a single subcutaneous injection of 300 mg/kg of GalN or vehicle (0.9% NaCl) at 14 weeks, and killed at 28 weeks of age. Next, 6-week-old male LEC rats were given weekly subcutaneous injections of 300 mg/kg of GalN or vehicle for 3 or 12 weeks, and their hepatic 8-hydroxydeoxy-2'-guanosine (8-OHdG), glutathione peroxidase (GPX), and catalase activities were measured. None of GalN-treated rats died of hepatic injury (0/12), whereas the mortality rate of control rats given 0.9% NaCl was 17% (2/12). GalN administration for 12 weeks decreased the hepatic 8-OHdG, and GalN administration for either 3 or 12 weeks increased the glutathione peroxidase activity. GalN administration increased the serum level of alanine aminotransferase, and accelerated megalocytic degeneration of the hepatocytes. GalN treatment is effective in preventing lethal hepatitis in LEC rats and decrease of oxidative DNA damage by GalN plays an important role in increase of the survival rate. (キーワード) Animals / Disease Models, Animal / Free Radicals / Galactosamine / Hepatolenticular Degeneration / Humans / Liver / Male / Rats / Rats, Inbred LEC (徳島大学機関リポジトリ) ● Metadata: 110791 (出版サイトへのリンク) ● Publication site (DOI): 10.2152/jmi.53.81 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 16537999 ● Summary page in Scopus @ Elsevier: 2-s2.0-33645835974 (徳島大学機関リポジトリ: 110791, DOI: 10.2152/jmi.53.81, PubMed: 16537999, Elsevier: Scopus)
5.	Masahiko Fujii, M Ishikawa, M Iuchi and Seiki Tashiro : Effect of Follistatin on Rat Liver Regeneration and Tumor Growth after Portal Occlusion, Hepato-Gastroenterology, Vol.52, No.63, 833-838, 2005. (要約) Although follistatin is known to be a positive regulator in liver regeneration after hepatectomy, the effects of follistatin on regeneration following portal occlusion and on tumor growth are not known. In experiment 1, rats were injected with follistatin immediately following 70% hepatic resection (HR), ligation of the left portal vein (PVL), or ligation of the left hepatic artery (HAL). Hepatic regenerative activity then was determined. In experiment 2, rats were injected intraportally with AH130 tumor cells. At 7 days after injection, the same procedures were carried out as in experiment 1. In a third experiment, in vitro, AH130 cells were cultured with various concentrations of activin A or follistatin, and cell proliferation was assessed. In experiment 1, follistatin did not accelerate liver regeneration after PVL. In experiment 2, follistatin significantly increased liver weight after HR, but did not accelerate tumor growth in any group. In vitro, neither follistatin nor activin affected proliferation of tumor cells. The mechanism of liver regeneration after PVL appears to differ from that after HR. Follistatin acts as a growth promoter for hepatocytes, but not for cancer cells. (キーワード) Activins / Animals / Cell Division / Follistatin / Inhibin-beta Subunits / Injections, Intravenous / Ligation / Liver Neoplasms, Experimental / Liver Regeneration / Male / Portal Vein / Rats / Tumor Cells, Cultured (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 15966215 ● Search Scopus @ Elsevier (PMID): 15966215 (PubMed: 15966215)
6.	Kazuhito Takamura, Kunihiro Tsuchida, Hidenori Miyake, Seiki Tashiro and Hiromu Sugino : Possible endocrine control by follistatin 315 during liver regeneration based on changes of activin receptor after partial hepatectomy in rat, Hepato-Gastroenterology, Vol.52, No.61, 60-66, 2005. (要約) Activin A is an autocrine inhibitor of cell growth in the liver. The biological activity of activin A is mediated by a heteromeric receptor complex. Follistatin (FS) binds to activin and inhibits its biological effects, and acts as a negative regulator of muscle cells. The role of activin receptors during liver regeneration following a hepatectomy has not been fully assessed. This study investigates the mechanism underlying how activin receptors regulate hepatocyte growth, and the effects of intravenous administration of FS during liver regeneration. The expression of both activins and activin receptors in the liver after a 70% partial hepatectomy (HT) was assessed by RT-PCR and immunohistochemistry. FS 315 or 288 was infused for different periods of time based on changes in hepatocyte activin receptor expression after HT. Activin receptor expression peaked between 48 and 72 hours after HT. 72 hours after HT, an injection of FS 315 resulted in a more potent stimulation of DNA synthesis and produced a greater increase in body weight compared with the control rats. The expression of activin receptors after peak DNA synthesis might be a key component in the downregulation of DNA synthesis. Intravenous administration of FS 315 might promote liver regeneration and have anabolic actions. (キーワード) Activin Receptors, Type II / Animals / Follistatin / Hepatectomy / Hepatocytes / Inhibin-beta Subunits / Injections, Intravenous / Liver Regeneration / Male / Proteins / Rats (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 15782995 ● Summary page in Scopus @ Elsevier: 2-s2.0-14144256099 (PubMed: 15782995, Elsevier: Scopus)
7.	H Miyake, Seiki Tashiro, Masahiko Fujii, K Sasaki and T Takagi : Efficacy of only left side approach in a case of unsuccessful reconstruction of the right hepatic artery after left hepatic lobectomy with caudal lobectomy, Hepato-Gastroenterology, Vol.51, No.56, 372-374, 2004. (キーワード) hilar cholangiocarcinoma / extended hepatic resection / vascular resection / hepatic artery / BILE-DUCT / RESECTION / CARCINOMA (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 15086162 ● Summary page in Scopus @ Elsevier: 2-s2.0-1842587992 (PubMed: 15086162, Elsevier: Scopus)
8.	Hidenori Miyamoto, Tatsuya Murakami, Kunihiro Tsuchida, Hiromu Sugino, Hidenori Miyake and Seiki Tashiro : Tumor-stroma interaction of human pancreatic cancer: acquired resistance to anticancer drugs and proliferation regulation is dependent on extracellular matrix proteins, Pancreas, Vol.28, No.1, 38-44, 2004. (要約) Pancreatic cancer is one of the major causes of cancer-related deaths in industrialized countries. It is known that pancreatic cancer is resistant to chemotherapy and that cancer cells are surrounded by extracellular matrix (ECM) proteins including collagen I, collagen IV, fibronectin, and laminin. To examine the role of ECM proteins in acquired resistance to anticancer drugs and proliferation regulation in pancreatic cancers. We used an in vitro model of ECM-induced chemoresistance and cell proliferation of cancer cell lines (MIA PaCa-2, PANC-1, and Capan-1) with 3 different malignancy grades and found that resistance to cytotoxic drugs and proliferation regulation was dependent on ECM proteins. Pancreatic cancer cell lines, especially MIA PaCa-2 cells, adhering to any of the ECM proteins showed decreased cytotoxicity of anticancer drugs, except for gemcitabine. PANC-1 and Capan-1 cells adhering to fibronectin, collagen I, and collagen IV proliferated more than the controls. ECM proteins have important roles in acquired resistance to anticancer drugs and cell proliferation regulation of pancreatic cancer cells. Therefore, the expression of ECM proteins in pancreatic cancer specimens could provide valuable information to aid anticancer drug cytotoxicity, and gemcitabine would be useful for treatment of patients with pancreatic cancer. (キーワード) pancreatic cancer / 細胞外マトリックス (extracellular matrix) / resistance / proliferation / anticancer drug / gemcitabine (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 14707728 ● CiNii @ 国立情報学研究所 (CRID): 1573950400004636032 ● Search Scopus @ Elsevier (PMID): 14707728 (PubMed: 14707728, CiNii: 1573950400004636032)
9.	藤井正彦, 三宅秀則, 鷹村和人, 田代征記 : 特集 Oncogenic emergencyとその対応:肝癌, 日本外科学会誌, Vol.105, No.4, 292-295, 2004年.
10.	藤井正彦, 田代征記, 三宅秀則, 栗田信浩, 鷹村和人 : 特集手術器械の使い方:膵臓の剥離と切離法, 手術, Vol.58, No.5, 657-662, 2004年.
11.	三宅秀則, 和田大助, 藤井正彦, 森根裕二, 居村暁, 田代征記 : 特集肝胆膵領域におけるエビデンスは?:胆嚢癌に対する肝SIVa SV切除の意義, 胆と膵, Vol.25, No.4, 207-212, 2004年.
12.	Masahiko Fujii, Hidenori Miyake, Katsuya Sasaki, Toshihide Takagi, Kazuhito Takamura and Seiki Tashiro : Arterial infusion chemotherapy for the patient of unresectable pancreatic carcinoma with liver metastasis: a case report, The Journal of Medical Investigation : JMI, Vol.50, No.3,4, 199-202, 2003. (要約) Despite various treatment trials for unresectable pancreatic carcinoma with liver metastases, the outcome has not been satisfactory. This paper reports a case of pancreatic carcinoma with multiple liver metastases that responded well to arterial infusion chemotherapy. A 65-year-old male was diagnosed with multiple liver tumors, and needle biopsy revealed adenocarcinoma. With endoscopic ultrasonography, a tumor in the pancreatic head was detected, and pancreatic carcinoma with multiple liver metastases was diagnosed. He received arterial infusion chemotherapy: cisplatin at a dose of 10 mg/body/day and 500 mg/body/day of 5-fluorouracil After 14 days administration, liver metastases had decreased in number and size, but thereafter, because of hepatic arterial occlusion, the same dose of drugs was administered intravenously. The patient was discharged from the hospital and was given chemotherapy 3 days a week on an outpatient basis. Although the chemotherapy was effective, it was stopped because of severe general fatigue 5 months after discharge. His general status continued to gradually worsen, and he died 12 months after diagnosis. Prognosis of pancreatic carcinoma with liver metastases is poor; however, transarterial infusion chemotherapy may be effective to improve the prognosis and quality of life of the patients. (キーワード) pancreatic carcinoma / arterial infusion chemotherapy / liver matastases (徳島大学機関リポジトリ) ● Metadata: 110704 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 13678391 ● Search Scopus @ Elsevier (PMID): 13678391 (徳島大学機関リポジトリ: 110704, PubMed: 13678391)
13.	三宅秀則, 藤井正彦, 佐々木克哉, 鷹村和人, 高木敏秀, 田代征記 : 胆嚢癌NOW:ss胆嚢癌の治療をどうする, --- 主として胆嚢床切除の立場から ---, 臨床外科, Vol.58, No.2, 169-172, 2003年. (キーワード) ss胆囊癌 / 漿膜下浸潤 / 胆囊床切除 / 肝床切除 / 肝区域切除 (出版サイトへのリンク) ● Publication site (DOI): 10.11477/mf.1407101327 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1390002209883447168 ● Search Scopus @ Elsevier (DOI): 10.11477/mf.1407101327 (DOI: 10.11477/mf.1407101327, CiNii: 1390002209883447168)
14.	新谷保実, 藤中雄一, 高橋幸志, 伊藤祐司, 加藤修司, 井上大輔, 松本俊夫, 栗田信浩, 田代征記, 坂東良美, 佐野壽昭, 宮恵子 : 空腹時の血漿インスリン濃度は常に低値で，グルカゴンにより著明なインスリン分泌反応が誘導されたインスリノーマの1例, 糖尿病, Vol.46, No.8, 679-684, 2003年.
15.	藤井正彦, 三宅秀則, 鷹村和人, 田代征記 : 特集:術前・術後のサーベイランスプログラム膵胆管合流異常, 外科, Vol.65, No.12, 1509-1514, 2003年.
16.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 鷹村和人, 田代征記 : 多臓器浸潤をきたした胃内分泌細胞癌の1例, 臨床外科, Vol.58, No.13, 1693-1696, 2003年.
17.	宮本英典, 栗田信浩, 三宅秀則, 藤井正彦, 佐々木克哉, 高木敏秀, 鷹村和人, 森根裕二, 居村暁, 田代征記 : 当科におけるStageIVb膵癌の治療成績, 四国医学雑誌, Vol.59, No.1-2, 30-34, 2003年. (徳島大学機関リポジトリ) ● Metadata: 110581 (徳島大学機関リポジトリ: 110581)
18.	中村多希, 奥村仙示, 津田紀子, 水野昭, 三宅秀則, 田代征記, 鶴尾美穂, 武田英二 : 肝硬変患者に対する就寝前夜食(LES)療法の長期継続効果:エネルギー代謝および耐糖能異常の改善がみられた1症例, 栄養-評価と治療, Vol.19, No.4, 461-465, 2002年.
19.	三宅秀則, 安藤勤, 藤井正彦, 佐々木克哉, 田代征記 : 肝門部胆管癌に対する肝中央2区域+尾状葉切除, 外科治療, Vol.86, No.3, 321-327, 2002年. (キーワード) 肝門部胆管癌 / 肝中央2区域切除 / 尾状葉切除 / 残肝機能 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1523669555646200576 (CiNii: 1523669555646200576)
20.	三宅秀則, 藤井正彦, 佐々木克哉, 高木敏秀, 田代征記 : 癌診療に役立つ最新データ:胆嚢癌の診断に関する最新のデータ, 臨床外科, Vol.57, No.11, 230-234, 2002年.
21.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 田代征記 : 高度肥満による皮下埋め込み式CVポートの変位が原因となりカテーテル逸脱をきたした1例, 四国医学雑誌, Vol.58, No.3, 174-177, 2002年. (徳島大学機関リポジトリ) ● Metadata: 110546 (徳島大学機関リポジトリ: 110546)
22.	佐々木克哉, 三宅秀則, 栗田信浩, 藤井正彦, 田代征記 : 膵管非癒合に合併した膵管内乳頭腫瘍の1切除例, 四国医学雑誌, Vol.58, No.3, 168-173, 2002年. (徳島大学機関リポジトリ) ● Metadata: 110545 (徳島大学機関リポジトリ: 110545)
23.	佐々木克哉, 三宅秀則, 藤井正彦, 小笠原卓, 田代征記 : 膵体部非浸潤性膵管内乳頭癌に対し脾温存膵体尾部切除術を施行した1例, 四国医学雑誌, Vol.58, No.3, 178-182, 2002年.
24.	佐々木克哉, 三宅秀則, 藤井正彦, 小笠原卓, 安藤勤, 田代征記 : 膵頭部膵管内乳頭腫瘍に対し膵頭十二指腸第Ⅱ部切除術を行った1例, 四国医学雑誌, Vol.58, No.1-2, 61-65, 2002年. (徳島大学機関リポジトリ) ● Metadata: 110528 (徳島大学機関リポジトリ: 110528)
25.	佐々木克哉, 三宅秀則, 細川亜裕, 藤井正彦, 安藤勤, 田代征記 : 左側胆嚢症に対し腹腔鏡下胆嚢摘出術を施行した3例の検討, 四国医学雑誌, Vol.58, No.1-2, 51-56, 2002年. (徳島大学機関リポジトリ) ● Metadata: 110526 (徳島大学機関リポジトリ: 110526)
26.	佐々木克哉, 三宅秀則, 篠原永光, 藤井正彦, 安藤勤, 田代征記 : 急性胆嚢炎で発症し術前にDICとなった胆嚢腺扁平上皮癌の1例, 四国医学雑誌, Vol.58, No.1-2, 41-45, 2002年. (徳島大学機関リポジトリ) ● Metadata: 110524 (徳島大学機関リポジトリ: 110524)
27.	三宅秀則, 藤井正彦, 佐々木克哉, 高木敏秀, 田代征記 : 肝切除のコツ肝切離にsray suture は有効か, 臨床外科, Vol.57, No.5, 647-649, 2002年.
28.	M. Ishikawa, Masahiko Fujii, M. Iuchi, T. Miyauchi and Seiki Tashiro : Effect of intrahepatic omental implantation on angiogensis in rat liver with artery ligation, Clinical and Experimental Medicine, Vol.1, No.1, 27-33, 2001. (要約) Despite early surgical intervention, the results of treating hepatic artery thrombosis remain poor. The use of omental flaps is well documented for its angiogenic potential in promoting neovascularization in ischemic tissues. This experimental study evaluated the formation of new blood-vessels after omental implantation (OI) in rats after ligation of the hepatic artery. Wistar rats were used and divided into the following groups: I OI with HAL, II OI without HAL, III hepatic artery ligation (HAL). For angiography, measurements were made of liver tissue blood flow by the laser Doppler method and of hepatic artery flow by the colored microsphere method (CMS), and immunohistochemical study was done for vascular endothelial growth factor (VEGF). The rats were killed at 1, 3, 7 or 30 days after laparotomy. Relative arterial hepatic blood flow in the implanted lobe of group I, as determined by CMS, reached 50% of control values 7 days after surgery. Angiography and microscopic studies of the excised liver revealed distinct angiogenesis surrounding the omental implant in the liver for 7 days postoperatively. The formation of new blood-vessels after OI was not observed in livers without HAL. Omental implantation appears to be useful in preventing organ anoxia after hepatic artery thrombosis. (キーワード) Omental implantation / Hepatic artery ligation (出版サイトへのリンク) ● Publication site (DOI): 10.1007/s10238-001-8006-3 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 11467399 ● Search Scopus @ Elsevier (PMID): 11467399 ● Search Scopus @ Elsevier (DOI): 10.1007/s10238-001-8006-3 (DOI: 10.1007/s10238-001-8006-3, PubMed: 11467399)
29.	三宅秀則, 安藤勤, 藤井正彦, 田代征記 : 最新の標準治療胆嚢癌ー進行癌, 外科, Vol.63, No.12, 1600-1606, 2001年.
30.	藤井正彦, 三宅秀則, 鳥羽昭三, 佐々木克哉, 安藤勤, 田代征記 : 巨大胆嚢粘液癌の1例, 日臨外医会誌, Vol.62, No.6, 1502-1508, 2001年.
31.	藤井正彦, 三宅秀則, 佐々木克哉, 安藤勤, 松村敏信, 田代征記, 八木恵子 : 結節浸潤型胆嚢癌との鑑別が困難であった黄色肉芽腫性胆嚢炎の1例, 日臨外医会誌, Vol.62, No.5, 1274-1279, 2001年.
32.	三宅秀則, 田上誉史, 安藤勤, 松山和男, 藤井正彦, 佐々木克哉, 松村敏信, 原田雅光, 安部正博, 松本俊夫, 田代征記 : 膵悪性リンパ腫の1例, 消化器画像, Vol.13, No.2, 151-153, 2001年.
33.	Takamasa Ohnishi, Eiji Takeda, Shiro Yogita, Hidenori Miyake, Takafumi Kinoshita, Yoshiyasu Terashima, Toshio Matsumoto and Seiki Tashiro : Effects of alendronate on bone metastases and hypercarcemia after surgery for hepatocellular carcinoma, Japanese Journal of Clinical Oncology, Vol.30, No.9, 410-413, 2000. (要約) Alendronate, a bisphosphonate compound, lowers serum calcium in patients with cancer-associated hypercalcemia through its inhibitory effect on bone resorption and as a result symptoms associated with hypercalcemia improve. This study was carried out to investigate the effects of alendronate in patients with hypercalcemia due to bone metastasis of hepatocellular carcinoma (HCC). Two patients were evaluated. Their corrected serum calcium and alpha-fetoprotein (AFP) levels and their computed tomography (CT), bone scintigraphy and magnetic resonance imaging (MRI) findings were evaluated before and during alendronate treatment. After treatment, not only the corrected serum calcium levels but also AFP levels and bone pain decreased; in addition, the regression of the metastatic focus was noted in the MRI analysis. These tumor inhibitory effects of alendronate have not been reported in HCC before; and alendronate might serve to prevent bone metastases in patients with HCC. In conclusion, two patients who developed hypercalcemia associated with bone metastasis after surgery for HCC were treated with alendronate and they experienced alleviation of the pain due to bone metastasis, improvement of their quality of life and a marked decrease in AFP levels with tumor regression. (キーワード) Aged / Alendronate / Bone Neoplasms / Carcinoma, Hepatocellular / Female / Humans / Hypercalcemia / Liver Neoplasms / Male (出版サイトへのリンク) ● Publication site (DOI): 10.1093/jjco/hyd105 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 11095140 ● Summary page in Scopus @ Elsevier: 2-s2.0-0033661348 (DOI: 10.1093/jjco/hyd105, PubMed: 11095140, Elsevier: Scopus)
34.	Shiro Yogita, Seiki Tashiro, Masamitsu Harada, Tetsuya Kitagawa and Itsuo Katoh : Hepatocelluar carcinoma with extension into the right atrium: report of a successful liver resection by hepatic vascular exclusion using cardiopulmonary bypass,, The Journal of Medical Investigation : JMI, Vol.47, No.3-4, 155-160, 2000. (要約) We report a successful liver resection using cardiopulmonary bypass with, total hepatic vascular exclusion (THVE) for hepatocellular carcinoma (HCC), with extension into the right atrium. A 61-year-old man with a cirrhotic liver was referred to our department with HCC in the medial segment of the left lobe of the liver, and tumor thrombus extending into the right atrium. During surgery, a left lobe and caudate lobe of the liver were transected leaving the left lobe of the liver connected to the inferior vena cava (IVC) by only the left and middle hepatic trunks, and then the intracaval tumor thrombus and the left lobe of the liver were removed en bloc using cardiopulmonary bypass with total hepatic vascular exclusion (THVE). Cardiac arrest was not performed during THVE, and the patient had an uneventful postoperative course and was discharged from the hospital 2 months following surgery. He died of multiple pulmonary metastases 4 years and 8 months after surgery; however, imaging showed no evidence of recurrence in the remnant liver during that period. In conclusion, by performing dissection of the hepatic parenchyma to the hepatic vein prior to removal of the tumor thrombus, the period of extracorporeal circulation, duration of warm ischemic time to the liver, and intraoperative blood loss were all reduced and a radical operation could be performed safely without scattering tumor cells during extirpation of the tumor. (キーワード) Carcinoma, Hepatocellular / Cardiopulmonary Bypass / Heart Atria / Humans / Liver / Liver Neoplasms / Male / Middle Aged / Thrombosis (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 11019497 ● Search Scopus @ Elsevier (PMID): 11019497 (PubMed: 11019497)
35.	Toru Kikutsuji, Masamitsu Harada, Seiki Tashiro, Setsuko Ii, Maki Moritani, Takashi Yamaoka and Mitsuo Itakura : Expression of somatostatin receptor subtypes and growth inhibition in human exocrine pancreatic cancers., Journal of Hepato-Biliary-Pancreatic Surgery, Vol.7, No.5, 496-503, 2000. (要約) The antiproliferative effects of somatostatin and its analogs on human pancreatic cancers were studied: (1) by evaluating the gene expression of somatostatin receptor (sstr) subtypes in human pancreatic cancer cell lines and cancer tissue specimens, (2) by evaluating the antiproliferative effects of somatostatin analogs, and (3) by evaluating the effect of sstr-2 cDNA transduction. Using a reverse transcriptase polymerase chain reaction (RT-PCR), the gene expression of five sstr subtypes (sstr-1 to -5) was examined in eight cell lines, and in ten pancreatic cancer tissues and in the normal surrounding pancreatic tissues. The antiproliferative effects of somatostatin (SS) -14 and its two analogs (SMS 201-995, RC-160) were examined by means of an MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (thiazolyl blue)) assay on three cell lines and Panc-1 transfectants with human sstr (hsstr)-2A cDNA. Sstr-2 was expressed in all samples tested. All examined cell lines simultaneously expressed sstr-2 and -5, while most of the examined pancreatic cancer tissues did not express both of these subtypes simultaneously. Somatostatin analogs inhibited epidermal growth factor (EGF)-stimulated pancreatic cancer cell proliferation. The cell proliferation was further and significantly inhibited by 14% in stable transfectants of Panc-1 cells with hsstr-2A. Based on these findings, it is concluded that somatostatin analogs with their antiproliferative effects mediated by sstr-2 could be potentially useful in the treatment of pancreatic cancers. (キーワード) Cell Division / Humans / Octreotide / Pancreatic Neoplasms / Receptors, Somatostatin / Reverse Transcriptase Polymerase Chain Reaction / Somatostatin / Tumor Cells, Cultured (出版サイトへのリンク) ● Publication site (DOI): 10.1007/s005340070021 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 11180877 ● Summary page in Scopus @ Elsevier: 2-s2.0-0034569570 (DOI: 10.1007/s005340070021, PubMed: 11180877, Elsevier: Scopus)
36.	Hisami Okumura, Kaori Genjida, Kimi Yokota, Yutaka Taketani, Kyoko Morita, Ken-ichi Miyamoto, Hidenori Miyake, Seiki Tashiro and Eiji Takeda : Daily pattern of energy metabolism in cirrhosis., Nutrition, Vol.15, No.10, 749-754, 1999. (要約) The daily pattern of energy expenditure and the oxidation rates of carbohydrates, fats, and protein were evaluated by indirect calorimetry in 18 control subjects (Group 1) and 34 cirrhotic patients who were divided into Groups 2a and 2b showing indocyanin green retention rates at 15 min of <30% and 30% or more, respectively. The ratio of resting energy expenditure to basal energy expenditure (%REE) was higher in the cirrhotic patients than in the controls at 8:30 AM and 2:30 PM. The oxidation rates of carbohydrates and fats under fasting conditions in Group 2b patients were respectively lower, and higher than in Group 1 and 2a patients. After the subjects ate, glucose became the substrate preferentially metabolized, and the proportion of fat metabolized was reduced from 82.9+/-5.1% to 43.9+/-21.9% and from 70.7+/-14.1% to 46.8+/-13.9% in the patients with advanced and less advanced cirrhosis, respectively, and from 59.4+/-27.2% to 48.4+/-18.5% in the controls. The fasting concentrations of non-esterified fatty acids in Group 2b were also significantly higher than those in the Group 1 and Group 2a patients. After eating, these concentrations fell and reached similar levels in the patients and controls. These data indicated that the patients with cirrhosis developed the catabolic state of starvation in the morning because of a lack of glycogen stores. Therefore, frequent meal supplementation to prevent early-onset starvation and energy deficiency may be advisable in such patients to maintain a well-nourished condition. (キーワード) Aged / Basal Metabolism / Calorimetry, Indirect / Dietary Carbohydrates / Dietary Fats / Dietary Proteins / エネルギー代謝 (energy metabolism) / Fasting / Fatty Acids, Nonesterified / Food / Glycogen / Humans / Liver Cirrhosis / Middle Aged / Oxidation-Reduction (出版サイトへのリンク) ● Publication site (DOI): 10.1016/S0899-9007(99)00149-5 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 10501287 ● Search Scopus @ Elsevier (PMID): 10501287 ● Search Scopus @ Elsevier (DOI): 10.1016/S0899-9007(99)00149-5 (DOI: 10.1016/S0899-9007(99)00149-5, PubMed: 10501287)
37.	Nobuhiro Kurita, D Wada, M Harada, H Kawasaki and Seiki Tashiro : Gastric function after pylorus-preserving duodenectomy in dogs, Hepato-Gastroenterology, Vol.45, No.19, 260-267, 1998. (要約) The purpose of this study was to delineate the alterations in gastric physiology after pylorus-preserving duodenectomy (PPD). The duodenum was transected 2 cm distal to the pyloric ring in five dogs. A patch graft around the papilla of Vater was preserved. Reconstruction was completed using an end-to-end duodenojejunostomy, anastomosis of the patch graft to the jejunum, and cholecystojejunostomy, in that order. Gastric acid analysis, serum gastrin and secretin concentrations, and gastric emptying were investigated before and one and three months after surgery. The postoperative mean basal and maximal acid outputs were not significantly increased. Fasting serum gastrin concentrations remained unchanged. The fasting serum secretin concentrations one month after surgery increased significantly as compared to the controls. There were no significant differences in meal-stimulated gastrin or secretin responses or gastric emptying after surgery. The acid output and gut hormone release after PPD were maintained at preoperative levels. Preservation of the duodenal bulb is postulated to be responsible for the maintenance of near-normal gastric physiology. (キーワード) Animals / Cell Count / Dogs / Duodenum / Gastric Acid / Gastric Emptying / Gastrin-Secreting Cells / Gastrins / Pancreaticoduodenectomy / Parietal Cells, Gastric / Pylorus / Secretin / Stomach (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 9496524 ● Summary page in Scopus @ Elsevier: 2-s2.0-0031938397 (PubMed: 9496524, Elsevier: Scopus)
38.	Takamasa Ohnishi, Yogita Shiro, Seiki Tashiro, Chone Yoshifumi, Kitaura Keisuke, Hino Akiko and Keisuke Izumi : Effects of tacrolimus (FK506) on induction of glutathione S-transferase P-positive foci and liver cell regeneration after partial hepatectomy in F344 rats, Hepatology Research, Vol.10, No.1, 58-65, 1998. (出版サイトへのリンク) ● Publication site (DOI): 10.1016/S1386-6346(97)00101-0 (文献検索サイトへのリンク) ● Search Scopus @ Elsevier (DOI): 10.1016/S1386-6346(97)00101-0 (DOI: 10.1016/S1386-6346(97)00101-0)
39.	北市隆, 北川哲也, 堀隆樹, 増田裕, 小川佳宏, 堀家一哉, 渡辺美恵, 市川洋一, 加藤逸夫, 香川征, 田代征記, 門田康正, 黒上和義 : 下大静脈内腫瘍栓を伴う悪性腫瘍の外科治療, 静脈学, Vol.9, 101-111, 1998年.
40.	北市隆, 北川哲也, 堀隆樹, 増田裕, 小川佳宏, 堀家一哉, 渡辺美恵, 市川洋一, 加藤逸夫, 香川征, 田代征記, 門田康正, 黒上和義 : 下大動脈内腫瘍栓を伴う悪性腫瘍の外科治療, 静脈学, Vol.9, No.2, 101-111, 1998年. (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1570291226242229760 (CiNii: 1570291226242229760)
41.	北川哲也, 北市隆, 堀隆樹, 吉栖正典, 田埜和利, 加藤逸夫, 余喜多史郎, 田代征記, 門田康正, 香川征 : 悪性腫瘍による下大静脈症候群の検査，診断，治療方針，治療，予後, 静脈およびリンパ管疾患と外科, 115-118, 1997年.
42.	Chiyo Horie, Hiroyuki Iwahana, Takahiro Horie, Ichiro Shimizu, Katsuhiko Yoshimoto, Shiro Yogita, Seiki Tashiro, Susumu Ito and Mitsuo Itakura : Detection of Different Quasispecies of Hepatitis C Virus Core Region in Cancerous and Non-cancerous Regions., Biochemical and Biophysical Research Communications, Vol.218, No.3, 674-681, 1996.
43.	田代征記, 余喜多史郎, 原田雅光, 福田洋, 和田大助, 北川哲也, 加藤逸夫 : 下大静脈・右心房への腫瘍栓を伴う肝細胞癌の手術, 消化器外科, Vol.19, 133-142, 1996年.

学術論文(紀要・その他):

1.	栗田信浩, 田代征記 : 尾側膵切除における断端処理のピットフォール, 外科, Vol.64, No.6, 643-646, 2002年. (出版サイトへのリンク) ● Publication site (DOI): 10.15106/j00393.2002273244 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1390564238029016832 ● Search Scopus @ Elsevier (DOI): 10.15106/j00393.2002273244 (DOI: 10.15106/j00393.2002273244, CiNii: 1390564238029016832)
2.	栗田信浩, 田代征記, 西井博 : 小児に対する手術, 消化性潰瘍臨床と基礎, Vol.19, 110-114, 2001年.

学術レター:

1.	庄野文章, 西岡安彦, 清水一郎, 田代征記, 濱佳子, 伊東進, 水口和生 : 徳島大学医学部附属病院における抗菌薬適正使用の取り組みと使用状況, 医療薬学, Vol.29, No.5, 611-615, 2003年. (要約) The working committee for the proper use of antibiotics was organized at Tokushima University Hospital in March 2002, and the institutional guidelines for the selection and use of antibiotics were established. The infection control team (ICT) also started monitoring the use of particular antibiotics, including third- and fourthgeneration cephalosporins, calbapenems and anti-MRSA antibiotics, and reported on their use in August 2002. To assess these activities, we examined the status of antibiotic use for injections and compared our findings with the previous data. The results were as follows : 1) the reports required were presented in 47% of the total antibiotics used ; 2) after starting the surveillance, the use of the third- and fourth-generation cephalosporins and calbapenems shifted to the first- and second-generation drugs, while the use of anti-MRSA drugs clearly decreased ; 3) these changes resulted in a savings of more than two million yen in medical expenditures in a month. These results suggest that closely monitoring the use of antibiotics may thus be beneficial not only for promoting the proper use of antibiotics but also for reducing overall medical costs. (キーワード) 抗生物質 (antibiotics) / anti-MRSA antibiotics / proper use / ICT (出版サイトへのリンク) ● Publication site (DOI): 10.5649/jjphcs.29.611 (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1390282679752865920 ● Search Scopus @ Elsevier (DOI): 10.5649/jjphcs.29.611 (DOI: 10.5649/jjphcs.29.611, CiNii: 1390282679752865920)

総説・解説:

1.	Yusuke Arakawa, Kazuo Yoshioka, H Kamo, K Kawano, T Yamaguchi, Y Sumise, N Okitsu, S Ikeyama, K Morimoto, Y Nakai and Seiki Tashiro : Huge retroperitoneal dedifferentiated liposarcoma presented as acute pancreatitis: report of a case., The Journal of Medical Investigation : JMI, Vol.60, No.1-2, 164-168, 2013. (要約) A 74-year-old male with abdominal pain was admitted to the emergency room in our hospital. The high value of serum amylase was shown in his blood test. The postcontrast computed tomography (CT) showed the huge retroperitoneal tumor with a thin-walled mass occupying most of the part of the right retroperitoneal space. The tumor spread into the soft tissues around the pancreas; as a result, the duodenum was compressed and the pancreas was displaced to the right side. The irregular pancreatic outline, obliterated peripancreatic fatty tissue and fluid in the left anterior pararenal space were revealed, so acute pancreatitis was diagnosed. The diagnostic biopsy of retroperitoneal tumor was done, and the pathological findings of retroperitoneal mass revealed dedifferentiated liposarcoma. The medical treatment against acute pancreatitis was performed firstly. After the patient recovered from that, the surgical resection of the tumor with the right kidney and right adrenal gland was completed successfully. The patient remained well, without any evidence of recurrence three months after surgery. However, the histology showed dedifferentiated liposarcoma; therefore, postoperative regular examination is necessary. J. Med. Invest. 60: 164-168, February, 2013 (キーワード) 英文論文 (徳島大学機関リポジトリ) ● Metadata: 106059 (出版サイトへのリンク) ● Publication site (DOI): 10.2152/jmi.60.164 (文献検索サイトへのリンク) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 23614927 ● CiNii @ 国立情報学研究所 (CRID): 1390001204245091456 ● Search Scopus @ Elsevier (PMID): 23614927 ● Search Scopus @ Elsevier (DOI): 10.2152/jmi.60.164 (徳島大学機関リポジトリ: 106059, DOI: 10.2152/jmi.60.164, PubMed: 23614927, CiNii: 1390001204245091456)

国際会議:

1.	Masahiko Fujii, H Miyake, K Sasaki, T Takagi and Seiki Tashiro : Surgical resection for hepatocellular carcinoma with a diameter of 10cm or larger, 18th World Congress of Digestive Surgery, Hong Kong, Dec. 2002.
2.	Kazuhito Takamura, Seiki Tashiro, Kunihiro Tsuchida and Hiromu Sugino : After partial hepatectomy liver regeneration is associated with expression of activin receptors in rat, Digestive Disease Week, San Francisco, May 2002.
3.	Nobuhiro Kurita, H Miyake, K Sasaki, M Fujii, T Andou, T Matsumura and Seiki Tashiro : Long-term results of chemoradiation for advanced pancreatic cancer, 5th World Congress of Hepato-pancreato-Biliary Association, Tokyo, Apr. 2002.
4.	Masahiko Fujii, H Miyake, K Sasaki, T Ando and Seiki Tashiro : Arterial infusion chemotherapy for the patient of unresectable pancreatic carcinoma with liver metastasis, 5th World Congress of Hepato-pancreato-Biliary Association, Tokyo, Apr. 2002.
5.	Nobuhiro Kurita, Hidenori Miyake, T Yamaguchi, K Sasaki, M Fuzii, H Oda, K Matsuyama, T Andoh, M Harada, S Yogita and Seiki Tashiro : Long-term results after pylorus-preserving pancreatoduodenectomy (PpPD), 9th United European Gastroenterology Week, Amsterdam, Oct. 2001.
6.	Masahiko Fujii, H Miyake, K Sasaki and Seiki Tashiro : Effect of Preoperative Portal Embolization on Changes of Liver Volume and Induction of Heat Shock Protein 70, 9th United European Gastroenterology Week, Amsterdam, Oct. 2001.
7.	Nobuhiro Kurita, M Nishi and Seiki Tashiro : The effect of combination of 5-fluorouracyl and cisplatin in the treatment of patients with carcinomatous peritonitis associated with gastric cancer, 4th International Gastric Cancer Congress, New York, Apr. 2001.
8.	Toshiko Tada, Yoshiyasu Terashima, Fumiko Hashimoto, Yasuko Matsushita and Seiki Tashiro : Analysis of the QOL of Post-operative Patients with Colon Cancer Using the EORTC, Pan-Pacific Conference of ISQOL, Tokyo, Apr. 2001.
9.	Masahiko Fujii, M Ishikawa, M Iuchi and Seiki Tashiro : Effect of follistatin on liver regeneration and proliferation of experimental liver cancer, International Association for the Study of the Liver, Fukuoka, Jun. 2000.
10.	Masahiko Fujii, M Ishikawa, M Iuchi and Seiki Tashiro : Effect of follistatin for liver regeneration and proliferation of experimental liver cancer, 11th Asian Pacific Congress for Digestive Disease, Hong Kong, Mar. 2000.

国内講演発表:

1.	後藤正和, 三浦連人, 木下貴史, 松山和男, 森本忠興, 西岡将規, 島田光生, 田代征記 : 下部直腸癌に対する術前化学放射線療法の効果 (第66回日本消化器外科学会総会), 日本消化器外科学会総会, 841, 2011年7月. (キーワード) 直腸腫瘍 / 治療成績 / ネオアジュバント療法 / 放射線化学療法 / ヒト (Homo sapiens)
2.	森根裕二, 島田光生, 高松英夫, 田代征記 : PD-8-1 全国集計からみた非拡張型膵・胆管合流異常の特徴と胆道癌発生頻度(PD8 パネルディスカッション(8)胆道拡張のない膵・胆管合流異常症,膵管形成異常症の治療:成人領域と小児領域,第111回日本外科学会定期学術集会), 日本外科学会雑誌, Vol.112, No.1, 277, 2011年5月. (文献検索サイトへのリンク) ● CiNii @ 国立情報学研究所 (CRID): 1571135652014252288 (CiNii: 1571135652014252288)
3.	濵田吉則, 嵩原裕夫, 安藤久美, 糸井隆夫, 神澤輝実, 越永従道, 信川正文, 藤井秀樹, 田代征記 : 小児胆管径の基準値に関する検討-日本膵・胆管合流異常研究会診断基準検討委員会報告第2報-, 第110回日本外科学会定期学術集会, 2010年4月.
4.	藤井正彦, 森根裕二, 居村暁, 池本哲也, 田代征記, 寺澤敏秀, 三宅秀則, 島田光生 : ss胆嚢癌に対する至適術式としてのS4a+S5区域切除術, 第105回日本外科学会総会, 2005年5月.
5.	藤井正彦, 三宅秀則, 鷹村和人, 倉橋三穂, 田代征記 : 高齢者肝細胞癌に対する手術適応と治療成績, 第90回日本消化器病学会総会, 2004年4月.
6.	森本慎也, 西村範行, 佐々木卓也, 田代征記 : Rabファミリー低分子量G蛋白質Rab13によるタイトジャンクションの接着分子Occludinの小胞輸送制御機構, 第104回日本外科学会学術集会, 2004年4月.
7.	藤井正彦, 三宅秀則, 鷹村和人, 倉橋三穂, 池山鎮夫, 西岡将規, 田代征記 : 再発胆嚢癌に対するS4上部温存拡大肝右葉+尾状葉切除および肝外胆管切除術, 第65回日本臨床外科学会総会, 2003年11月.
8.	森本慎也, 西村範行, 北村明子, 品原和加子, 山本恭代, 寺井智也, 坂根亜由子, 真鍋進治, 田代征記, 佐々木卓也 : Rabファミリー低分子量G蛋白質によるタイトジャンクション膜蛋白質の小胞輸送の制御機構, 第62回日本癌学会総会, 2003年9月.
9.	藤井正彦, 佐々木克哉, 高木敏秀, 鷹村和人, 三宅秀則, 田代征記 : 高齢の大腸癌非合併，家族性大腸線種症患者に発生した十二指腸乳頭部癌の1例, 第58回日本消化外科学会総会, 2003年7月.
10.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 鷹村和人, 田代征記 : Hinf2 進行胆嚢癌に対する肝中央2区域切除+幽門輪温存膵頭十二指腸切除術, 第15回日本肝胆膵外科学会, 2003年5月.
11.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 田代征記 : 多臓器浸潤を伴う壁外発育型胃神経内分泌癌の1例, --- 本邦報告例の検討を含めて ---, 第89回日本消化器病学会総会, 2003年4月.
12.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 栗田信浩, 田代征記 : 胃原発巨大神経内分泌に対する結腸部分切除合併，膵頭十二指腸切除術, 第64回日本臨床外科学会総会, 2002年11月.
13.	栗田信浩, 安藤勤, 寺嶋吉保, 田代征記, 松村敏信 : 腹腔鏡補助下に切除した成人腸重複症の1例, 第15回日本内視鏡外科学会総会, 2002年9月.
14.	藤井正彦, 三宅秀則, 佐々木克哉, 高木敏秀, 田代征記 : 巨大肝細胞癌に対する外科的治療の意義, 第57回日本消化器病学会総会, 2002年7月.
15.	栗田信浩, 西正晴, 岩田貴, 松村敏信, 三宅秀則, 安藤勤, 吉川幸造, 田代征記 : Laparoscopy assisted distal gastrectomyの2例, 第16回四国内視鏡外科研究会, 2002年2月.
16.	藤井正彦, 鳥羽昭三, 佐々木克哉, 安藤勤, 三宅秀則, 松村敏信, 田代征記 : Hinf3 巨大胆嚢粘液癌に対するS4 上部温存拡大肝右葉切除，胆管切除術, 第63回日本臨床外科学会総会, 2001年10月.
17.	山崎誠司, 松村敏信, 田代征記, 吉本勝彦, 森谷眞紀, 板倉光夫 : 膵α細胞にTGF-β1を発現させた膵島移植モデルにおける免疫寛容の誘導, 第56日本消化器外科学会, 2001年7月.
18.	藤井正彦, 三宅秀則, 佐々木克哉, 松山和男, 栗田信浩, 安藤勤, 原田雅光, 余喜多史郎, 田代征記 : 術前門脈枝塞栓術後のHSPによる肝予備能向上の可能性について, 第56回日本消化器病学会総会, 2001年7月.
19.	栗田信浩, 三宅秀則, 山口剛史, 細川亜裕, 佐々木克哉, 藤井正彦, 小田浩睦, 松山和男, 安藤勤, 原田雅光, 松村敏信, 余喜多史郎, 田代征記 : 当科におけるインスリノーマ症例の検討, 第32回膵臓学会, 2001年7月.
20.	栗田信浩, 原田雅光, 山口剛史, 佐々木克哉, 藤井正彦, 小田浩睦, 松山和男, 安藤勤, 三宅秀則, 松村敏信, 余喜多史郎, 田代征記 : 当科におけるPpPD術後の遠隔成績, 日本肝胆膵外科関連会議, 2001年6月.
21.	藤井正彦, 三宅秀則, 佐々木克哉, 小笠原卓, 鳥羽昭三, 松山和男, 松村敏信, 安藤勤, 原田雅光, 余喜多史郎, 田代征記 : HCCに対する右下肝臓脈温存，右前区域+右後上亜区域切除術, 第13回日本肝胆膵外科学会, 2001年6月.
22.	黒葛原健太朗, 石田弓, 上岡千世, 寺嶋吉保, 田代征記 : 当院緩和ケア室における心理臨床の現状と課題, 日本緩和医療学会第6回総会, 2001年6月. (キーワード) 徳島大学医学部附属病院緩和ケア室 / 臨床心理士 / オンコロジー·カウンセリング
23.	藤井正彦, 三宅秀則, 鳥羽昭三, 佐々木克哉, 松山和男, 安藤勤, 原田雅光, 松村敏信, 余喜多史郎, 田代征記 : 結節浸潤胆嚢癌との鑑別が困難であった黄色肉芽腫性胆嚢炎の1例, 第87回日本消化器病学会総会, 2001年4月.
24.	栗田信浩, 西正晴, 尾形頼彦, 岩田貴, 小田浩睦, 佐々木賢二, 松山和男, 寺嶋吉保, 田代征記 : 胃癌による癌性腹膜炎症例に対する低用量CDDP/5FU療法の臨床成績., 第73回日本胃癌学会総会, 2001年3月.
25.	藤井正彦, 三宅秀則, 鳥羽昭三, 佐々木克哉, 松山和男, 安藤勤, 原田雅光, 松村敏信, 余喜多史郎, 田代征記 : 胆嚢粘液癌の1例, 第34回日本消化器画像診断研究会, 2001年2月.
26.	藤井正彦, 三宅秀則, 佐々木克哉, 佐々木憲二, 安藤勤, 寺嶋吉保, 余喜多史郎, 田代征記 : リバーザー動注化学療法により著明な縮小効果を認めた大腸癌肝移転の2症例, 第22回リザーバー研究会, 2001年2月.
27.	藤井正彦, 三宅秀則, 佐々木克哉, 尾方信也, 松山和男, 安藤勤, 原田雅光, 余喜多史郎, 田代征記 : 当科における術前門脈枝塞栓術症例の検討, 第62回日本臨床外科学会, 2000年11月.
28.	藤井正彦, 西正晴, 篠原永光, 鳥羽昭三, 田代征記 : 光線力学的治療を行った早期胃癌の1例, 第74回日本消化器病学会四国支部例会, 2000年11月.
29.	多田敏子, 桑村由美, 南川貴子, 橋本文子, 寺嶋吉保, 田代征記 : 大腸がん患者のQOL評価の試み, 第221回徳島医学会学術集会, 2000年8月.
30.	藤井正彦, 大塚敏弘, 三宅秀則, 開野友佳理, 尾方信也, 松山和男, 安藤勤, 原田雅光, 余喜多史郎, 田代征記 : 術前門脈枝塞栓術の肝再生と肝機能に及ぼす影響, 第12回日本胆膵外科学会, 2000年5月.
31.	藤井正彦, 嵩原裕夫, 田代征記 : 徳島県主幹病院のアンケート調査結果から見たHPNの現状とその認識, 第4回四国臨床栄養研究会, 2000年1月.
32.	北川哲也, 北市隆, 堀隆樹, 増田裕, 小川佳宏, 掘家一哉, 渡辺美恵, 加藤逸夫, 香川征, 田代征記, 門田康正, 黒上和義 : 大静脈内腫瘍栓または浸潤を有する悪性腫瘍に対する外科治療の検討, 第18回日本静脈学会総会, 1998年5月.
33.	栗田信浩, 和田大助, 原田雅光, 河崎秀樹, 田代征記 : 犬における十二指腸球部温存十二指腸切除術後の残胃機能, --- 特に胃酸分泌について ---, 第15回四国消化性潰瘍研究会, 1998年5月.
34.	栗田信浩, 和田大助, 原田雅光, 三宅秀則, 西正晴, 田代征記 : 教室における胃悪性リンパ腫の検討, 第24回胃外科研究会, 1995年1月.

名誉教授 : 田代征記

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名誉教授 : 田代 征記

研究活動

専門分野

研究テーマ

著書・論文

著書:

学術論文(審査論文):

学術論文(紀要・その他):

学術レター:

総説・解説:

国際会議:

国内講演発表:

名誉教授 : 田代征記