Tokushima University / Educator and Researcher Directory --- Suzue, Masashi

Field of Study

Subject of Study

Book / Paper

Academic Paper (Judged Full Paper):

1.	Ken-ichi Suga, Yoshifumi Wakata, Manami Suzuki, Shunsuke Takeuchi, Masashi Suzue, Ryuji Nakagawa and Maki Urushihara : Neonatal hyperkalemia associated with maternal ritodrine: A retrospective study, Pediatrics International, Vol.65, No.1, e15604, 2023. (Summary) We recently reported on a late preterm infant born at 36 weeks' gestation with serious arrhythmia due to hyperkalemia associated with long-term maternal ritodrine administration. It is unknown whether ritodrine alone increases the risk of neonatal hyperkalemia in infants born at 34-36 weeks' gestation. This single-center, retrospective, cohort study enrolled late preterm infants (34-36 gestational weeks) born between 2004 and 2018. Cases with maternal magnesium sulfate use were not sufficient for statistical analysis and so were excluded from the study. Risk factors for the occurrence of hyperkalemia were determined based on clinical relevance and previous reports. In all, 212 late preterm infants with maternal ritodrine use and 400 infants without tocolysis were included in the study. Neonatal hyperkalemia occurred in 5.7% (12/212) in the ritodrine group and 1.8% (7/400) in the control group. The risk of neonatal hyperkalemia was significantly increased by maternal ritodrine administration with a crude odds ratio (OR) of 3.37 (95% confidence interval [CI]: 1.30-8.69; p < 0.01) and an adjusted OR of 3.71 (95% CI: 1.41-9.74; p < 0.01) on multivariable analysis. Long-term tocolysis (≥28 days) with ritodrine increased the risk of neonatal hyperkalemia with 9.3% (11/118) of infants developing hyperkalemia (adjusted OR 4.86; 95% CI: 1.59-14.83; p < 0.01). Neonatal hyperkalemia was not found within 2 weeks of ritodrine administration. This research suggests that late preterm infants born after long-term ritodrine administration are at risk of neonatal hyperkalemia and require special attention. (Keyword) Pregnancy / infant / female / Infant, Newborn / Humans / Ritodrine / Obstetric Labor, Premature / Retrospective Studies / Cohort Studies / Hyperkalemia / Infant, Premature (Link to Publication Site) ● Publication site (DOI): 10.1111/ped.15604 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 37551666 ● Summary page in Scopus @ Elsevier: 2-s2.0-85166784295 (DOI: 10.1111/ped.15604, PubMed: 37551666, Elsevier: Scopus)
2.	Hiroki Sato, Ken-ichi Suga, Masashi Suzue, Yukako Honma, Yasunobu Hayabuchi, Shunsuke Miyai, Hiroki Kurahashi and Ryuji Nakagawa : Novel large deletion involving EVC and EVC2 in Ellis-van Creveld syndrome, Human Genome Variation, Vol.9, No.1, 15, 2022. (Summary) Ellis-van Creveld syndrome is an autosomal recessive skeletal dysplasia that is characterized by thoracic hypoplasia, polydactyly, oral abnormalities, and congenital heart disease. It is caused by pathogenic variants in the EVC or EVC2 genes. We report a case of a newborn with a compound heterozygous variant comprising NM_147127.5: c.1991dup:[p.Lys665Glufs*10] in the EVC2 gene and a novel large deletion involving exon 1 in EVC and exons 1-7 in EVC2. (Tokushima University Institutional Repository) ● Metadata: 118010 (Link to Publication Site) ● Publication site (DOI): 10.1038/s41439-022-00190-0 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 35581188 ● Summary page in Scopus @ Elsevier: 2-s2.0-85130247944 (Tokushima University Institutional Repository: 118010, DOI: 10.1038/s41439-022-00190-0, PubMed: 35581188, Elsevier: Scopus)
3.	Ken-ichi Suga, Masashi Suzue, Ryuji Nakagawa and Hiroki Ishibashi : Neonatal Fever with Hepatomegaly and a Rapidly Involuting Congenital Hemangioma, The Journal of Pediatrics, Vol.237, 315-316, 2021. (Keyword) congenital (Tokushima University Institutional Repository) ● Metadata: 117564 (Link to Publication Site) ● Publication site (DOI): 10.1016/j.jpeds.2021.06.003 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 34118240 ● Search Scopus @ Elsevier (PMID): 34118240 ● Search Scopus @ Elsevier (DOI): 10.1016/j.jpeds.2021.06.003 (Tokushima University Institutional Repository: 117564, DOI: 10.1016/j.jpeds.2021.06.003, PubMed: 34118240)
4.	Keita Osumi, Ken-ichi Suga, Masashi Suzue, Ryuji Nakagawa and Shoji Kagami : Neonatal rebound hyperkalemia associated with ritodrine alone: a case report, BMC Pediatrics, Vol.21, No.1, 370, 2021. (Summary) Betamimetics have been used for tocolysis extensively in the past, and one of them, ritodrine is widely used in Japan. Various adverse events have been reported for this agent, including newborn hypoglycemia and hypokalemia, as well as maternal hypokalemia and rebound hyperkalemia; however, cases of neonatal rebound hyperkalemia are not described in the literature. A male infant born at 36 weeks of gestation by cesarean section at a local maternity clinic suddenly entered cardiopulmonary arrest with ventricular tachycardia and fibrillation due to hyperkalemia (K, 8.7 mmol/L). No monitoring, examination of blood electrolyte levels, or infusions had been performed prior to this event. Maternal infusion of ritodrine (maximum dose, 170 μg/min) had been performed for 7 weeks prior to cesarean section. After resuscitation combined with calcium gluconate, the infant died at 4 months old due to serious respiratory failure accompanied by acute lung injury following shock. No cause of hyperkalemia other than rebound hyperkalemia associated with ritodrine was identified. This case report serves as a warning regarding the potential risk of neonatal rebound hyperkalemia in association with maternal long-term ritodrine administration. (Keyword) Cesarean Section / Female / Humans / Hyperkalemia / Infant / Infant, Newborn / Male / Obstetric Labor, Premature / Pregnancy / Ritodrine / Tocolytic Agents (Tokushima University Institutional Repository) ● Metadata: 117418 (Link to Publication Site) ● Publication site (DOI): 10.1186/s12887-021-02840-8 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 34465290 ● Search Scopus @ Elsevier (PMID): 34465290 ● Search Scopus @ Elsevier (DOI): 10.1186/s12887-021-02840-8 (Tokushima University Institutional Repository: 117418, DOI: 10.1186/s12887-021-02840-8, PubMed: 34465290)
5.	Masashi Suzue, Maki Urushihara, Ryuji Nakagawa, T Saijo and Shoji Kagami : Urinary angiotensinogen level is increased in preterm neonates., Clinical and Experimental Nephrology, Vol.19, No.2, 293-297, 2015. (Summary) All components of the renin-angiotensin system (RAS) are abundantly synthesized in the developing kidney, suggesting that the RAS plays an important role in renal development. To examine this system in human neonates, we measured urinary angiotensinogen levels in preterm and full-term neonates and examined the relationship between urinary angiotensinogen levels and gestational age. Urine and plasma samples were collected from 20 preterm and 18 full-term neonates at birth. Angiotensinogen levels were measured using enzyme-linked immunosorbent assay. Plasma angiotensinogen concentrations were not increased in preterm neonates compared with that in full-term neonates (P = 0.7288). However, the urinary angiotensinogen-to-creatinine ratio was significantly higher in preterm neonates compared with that in full-term neonates (P = 0.0011). Importantly, the urinary angiotensinogen-to-creatinine ratio dropped significantly with increasing gestational age (P = 0.0010), whereas the plasma angiotensinogen concentration was not correlated with gestational age (P = 0.7814). These results suggest that urinary angiotensinogen levels may indicate the involvement of intrarenal RAS activation in prenatal renal development. (Keyword) Angiotensinogen / Biomarkers / Case-Control Studies / Creatinine / Gestational Age / Humans / Infant, Newborn / Premature Birth / Renin-Angiotensin System / Term Birth (Tokushima University Institutional Repository) ● Metadata: 109717 (Link to Publication Site) ● Publication site (DOI): 10.1007/s10157-014-0977-y (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 24792728 ● Summary page in Scopus @ Elsevier: 2-s2.0-84939894853 (Tokushima University Institutional Repository: 109717, DOI: 10.1007/s10157-014-0977-y, PubMed: 24792728, Elsevier: Scopus)
6.	Masashi Suzue, Kazuhiro Mori, MIki Inoue, Yasunobu Hayabuchi, Ryuji Nakagawa and Shoji Kagami : Developmental changes in the left ventricular diastolic wall strain on M-mode echocardiography, Journal of Echocardiography, Vol.12, No.3, 98-105, 2014. (Summary) The diastolic wall strain (DWS) of the left ventricle has been proposed as an indicator of left ventricular (LV) wall stiffness. The DWS is calculated as follows using M-mode echocardiography:[Formula: see text]Although this index is simple and clinically useful, normal values for children, including neonates, have not been reported. The DWS was measured in 235 healthy people, ranging from neonates to adults. They were classified into 8 subgroups according to their age. The DWS was compared with conventional echocardiographic parameters for left ventricle function, including shortening fraction of the left ventricle, the Tei index, E/A of mitral flow, mitral annular tissue Doppler velocity during systole (s') and during early diastole (e'), and the E/e' ratio. The DWS in the just after birth group was 0.28 ± 0.11, which was significantly lower than that of the remaining groups (p < 0.05), except for the neonate group at 5-10 days after birth. The DWS was highest in the 1-9 years of age group, and then gradually decreased with age. Stepwise regression of various echocardiographic parameters showed that e' was the most relevant parameter for the DWS (β = 0.64). Normal values for the DWS of the left ventricle change with age. The data reported in this study can be used as normal values for the DWS of the left ventricle determined by M-mode echocardiography. (Keyword) Adolescent / Adult / Child / Child, Preschool / Diastole / Echocardiography / Echocardiography, Doppler / female / Heart Ventricles / Humans / infant / Infant, Newborn / Systole / Ventricular Dysfunction, Left / Ventricular Function, Left / Young Adult (Link to Publication Site) ● Publication site (DOI): 10.1007/s12574-014-0222-4 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 27276893 ● Summary page in Scopus @ Elsevier: 2-s2.0-84907781830 (DOI: 10.1007/s12574-014-0222-4, PubMed: 27276893, Elsevier: Scopus)
7.	Tatsuo Mori, Kenji Mori, Masashi Suzue, Hiromichi Ito and Shoji Kagami : Effective treatment of a 13-year-old boy with steroid-dependent ocular myasthenia gravis using tacrolimus, Brain & Development, Vol.35, No.5, 445-448, 2013. (Summary) Over the past several years, tacrolimus has attracted attention as a new therapeutic drug for myasthenia gravis (MG), but few reports have considered its use for MG in pediatric patients, and most of these have focused on severe systemic MG. In this case report, we used tacrolimus to successfully treat a 13-year-old boy with ocular MG who had suffered from severe steroid complications, including a failure of thrive and osteoporosis. He first showed symptoms of ocular MG at age 2 years 3 months. At age 13 years, he was receiving PSL (3.75 mg/day), but the symptoms of ocular MG recurred. We increased the dosage of oral PSL up to 30 mg/day, and three courses of mPSL pulse therapy were applied, but these therapies had only limited effect, and his symptoms worsened. Tacrolimus was started at 0.4 mg/day (0.011 mg/kg/day), and every 2 weeks the dose was gradually increased by 0.2 mg/day. His symptoms of MG began to improve 3 weeks after the initial administration of tacrolimus. Approximately 3 months after the start of tacrolimus administration, PSL was discontinued. Currently, at 1 year and 4 months after the start of tacrolimus administration, while slight ptosis is observed in the evening, it does not influence his daily life, and his condition remains comparable to that when he stopped taking PSL. No adverse effects of tacrolimus have been recognized. In pediatric patients with steroid-dependent ocular MG without thymectomy, tacrolimus may be a safe and effective alternative to steroid and thymectomy. (Keyword) Adolescent / Body Weight / Developmental Disabilities / Follow-Up Studies / Humans / Immunosuppressive Agents / Male / Myasthenia Gravis / Tacrolimus (Tokushima University Institutional Repository) ● Metadata: 114873 (Link to Publication Site) ● Publication site (DOI): 10.1016/j.braindev.2012.06.012 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 22840813 ● Summary page in Scopus @ Elsevier: 2-s2.0-84875894491 (Tokushima University Institutional Repository: 114873, DOI: 10.1016/j.braindev.2012.06.012, PubMed: 22840813, Elsevier: Scopus)

Academic Letter:

1.	Masashi Suzue, Ken-ichi Suga, Yutaka Taketani, Ryuji Nakagawa and Maki Urushihara : Chylous ascites complicating perinatal severe hypophosphatasia in an infant on high-setting ventilation and enzyme replacement therapy., Pediatrics International, Vol.65, No.1, 2023. (Keyword) Infant / Humans / Hypophosphatasia / Enzyme Replacement Therapy / Chylous Ascites / Respiration / Immunoglobulin G / Alkaline Phosphatase (Link to Publication Site) ● Publication site (DOI): 10.1111/ped.15585 (Link to Search Site for Scientific Articles) ● PubMed @ National Institutes of Health, US National Library of Medicine (PMID): 37475513 ● Search Scopus @ Elsevier (PMID): 37475513 ● Search Scopus @ Elsevier (DOI): 10.1111/ped.15585 (DOI: 10.1111/ped.15585, PubMed: 37475513)

Proceeding of Domestic Conference:

1.	Masashi Suzue, Ryuji Nakagawa, Ken-ichi Suga, Takashi Kaji, Hiroki Ishibashi and Maki Urushihara : 出生後の気道確保難渋が予想され，娩出時臍帯非切断下帝王切開(EXIT)での娩出を行った頸部リンパ管腫の一例, 第160回日本小児科学会徳島地方会, Dec. 2023.
2.	Masashi Suzue, Ryuji Nakagawa and Ken-ichi Suga : 多発性嚢胞性病変を伴う間質性肺気腫に対して肺実質持続吸引を施行した超低出生体重児の1例, 第67回日本成育医学会学術集会, Vol.--, No.--, --, Nov. 2023.
3.	Masashi Suzue, Ryuji Nakagawa, Ken-ichi Suga, Eiji Kondou and Maki Urushihara : 新生児聴覚スクリーニングで発見に至った難聴のみの先天性CMV感染症の一例, 第75回中国四国小児科学会学術集会, Oct. 2023.
4.	奥村友里香, Ken-ichi Suga, Masashi Suzue and Ryuji Nakagawa : Noonan syndrome like disorder with loose anagen hair 2の1例, 第59回日本周産期・新生児医学会総会・学術集会, Jul. 2023.
5.	鈴木真波, Ken-ichi Suga, Masashi Suzue and Ryuji Nakagawa : コステロ症候群の2例, 第59回日本周産期・新生児医学会総会・学術集会, Jul. 2023.
6.	Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa, Eiji Kondou and Maki Urushihara : 鼻腔開通術後のステント留置で良好な経過が得られた先天性後鼻腔閉鎖症の1例, 第159回日本小児科学会徳島地方会, Jun. 2023.
7.	Masashi Suzue, Ryuji Nakagawa and Ken-ichi Suga : 多発性嚢胞性病変を伴う間質性肺気腫に対して肺実質持続吸引を施行した超低出生体重児の1例, 第119回徳島周産期研究会, May 2023.
8.	Masashi Suzue, Ken-ichi Suga and Ryuji Nakagawa : 腸回転異常を伴わない小腸捻転を来たしたSGAの1例, 第66回日本新生児成育医学会, Dec. 2022.
9.	Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa and Maki Urushihara : 母体重症妊娠高血圧症候群に伴う新生児急性呼吸障害の臨床的検討, 第158回日本小児科学会徳島地方会, Dec. 2022.
10.	Ken-ichi Suga, Masashi Suzue and Ryuji Nakagawa : 早産児の非乏尿性高K血症はEarly aggressive nutritionによって減少している, 第66回日本新生児成育医学会, Nov. 2022.
11.	Atsuko Yoshida, Junki Imaizumi, Aya Shirakawa, Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa, Takeshi Iwasa and Takashi Kaji : 当院におけるExtended-spectrum β-lactamase(ESBL)産生菌保菌妊婦の現状, 第58回日本周産期・新生児医学会学術集会, Jul. 2022.
12.	Masashi Suzue, Ryuji Nakagawa and Ken-ichi Suga : 当院で実施したPICC関連静脈炎抑制の試み, 第58回日本周産期・新生児医学会学術集会, Jul. 2022.
13.	Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa and Maki Urushihara : 当センターで経験したCHARGE症候群の3例, 第157回日本小児科学会徳島地方会, Jun. 2022.
14.	Yuri Hayashi, Yasunobu Hayabuchi, Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa, Mikio Sugano, Takashi Kitaichi and Hiroki Hata : 周術期に心室頻拍(VT)・異所性節五部頻拍(JET)・異所性心房頻拍(EAT)が認められ，管理に難渋した完全大血管転換症の1例, 第117回日本小児科学会徳島地方会, Jun. 2022.
15.	Masashi Suzue, Ken-ichi Suga, Ryuji Nakagawa, Hiroki Ishibashi, Noriko Yokota and Hiroki Mori : 腸回転異常を伴わない中腸軸捻転を来したSGA児の一例, 第118回徳島周産期研究会, May 2022.
16.	Shoko Fukura, Masashi Suzue, Ken-ichi Suga, Yukako Homma, Yasunobu Hayabuchi, Hiroyoshi Watanabe and Ryuji Nakagawa : 重症一過性骨髄異常増殖症に対し，少量シタラビン療法を行った21トリソミーの1例, 第135回日本小児科学会徳島地方会, Jun. 2021.
17.	藤中ちひろ, Masashi Suzue and Yasunobu Hayabuchi : 総肺静脈還流異常Ibの一新生児例:市中病院の超音波検査士の立場から, 第5回日本小児超音波研究会, Nov. 2019.
18.	Maki Urushihara, Masashi Suzue, Ryuji Nakagawa, Takahiko Saijyou and Shoji Kagami : 腎発生における尿中アンジオテンシノーゲンの臨床的意義の検討, 第45回日本腎臓学会西部学術大会, Oct. 2015.
19.	Masashi Suzue, Maki Urushihara, Ryuji Nakagawa and Shoji Kagami : 早産児における尿中アンジオテンシノーゲンの臨床的意義の検討, 第9回日本DOHaD研究会学術集会, Aug. 2015.
20.	Masashi Suzue, Maki Urushihara, Ryuji Nakagawa, Takahiko Saijyou and Shoji Kagami : 早産児における尿中アンジオテンシノーゲン, 第49回日本小児腎臓病学会学術集会, Jun. 2014.
21.	Masashi Suzue, Miho Sakata, Yasunobu Hayabuchi, Shoji Kagami, Ryuji Nakagawa, 渡辺典子, Takahiko Saijyou, Mikio Sugano and 北山哲也 : 9番染色体部分トリソミー・21番染色体部分モノソミーの1例, 第64回日本小児科学会中国四国地方会, Dec. 2012.
22.	Masashi Suzue, Shoji Kagami, Ryuji Nakagawa, 渡辺典子, Takahiko Saijyou and Hiroki Ishibashi : 腹膜透析管理を行っている両側性多嚢胞性異形成腎の1例, 第139回日本小児科学会徳島地方会, Dec. 2012.
23.	森達夫, Kenji Mori, Masashi Suzue, Hiromichi Ito and Shoji Kagami : Tacrolimusが有効でステロイドを中止し得た眼筋型重症筋無力症の1小児例, 第115回日本小児科学会学術集会, Apr. 2012.
24.	Kazumi Okamura, Hiroyoshi Watanabe, 永井隆, Masashi Suzue, Nami Inoue, Kazunori Sekine, Koichi Tamura and Shoji Kagami : 化学療法が奏功したMPNSTの一例, 第53回日本小児血液・がん学会, Nov. 2011.
25.	森達夫, Masashi Suzue, Hiromichi Ito, Kenji Mori and Shoji Kagami : Tacrolimusが有効でステロイドを中止し得た眼筋型重症筋無力症の1小児例, 第26回徳島県小児科合同症例検討会, Sep. 2011.
26.	Kazumi Okamura, Hiroyoshi Watanabe, Masashi Suzue, Nami Inoue, Miho Sakata, Yukiko Kinoshita, Shuji Kondo, Yasunobu Hayabuchi, 清水真樹, Katsuyoshi Tamaki and Shoji Kagami : Fontan術後の肝過形成の2例, 第52回日本小児血液学会・第26回日本小児がん学会, Dec. 2010.
27.	Masashi Suzue, Ryuji Nakagawa, Takahiko Saijyou, Ken-ichi Suga, 渡辺典子, Miho Sakata, Yasunobu Hayabuchi and Shoji Kagami : 右大動脈弓に声帯麻痺を合併した1例, 第135回日本小児科学会徳島地方会, Dec. 2010.
28.	岡村和美, Hiroyoshi Watanabe, Masashi Suzue, Nami Inoue, Shoji Kagami, 清水真樹 and Katsuyoshi Tamaki : Fontan 術後の肝過形成の1例, 第135回日本小児科学会徳島地方会, Dec. 2010.

Et cetera, Workshop:

1.	Shoko Fukura, Masashi Suzue, Ken-ichi Suga and Ryuji Nakagawa : 重症一過性骨髄異常増殖症に対し少量シタラビン療法を行った21トリソミーの1例, 第39回四国新生児医療研究会, May 2022.
2.	Masashi Suzue, Ken-ichi Suga and Ryuji Nakagawa : 徳島県におけるシナジス投与期間の決定について, 第39回四国新生児医療研究会, May 2022.
3.	Ken-ichi Suga, Masashi Suzue and Ryuji Nakagawa : オール四国で取り組む多施設共同研究マルチサイトカインプロファイルによる新生児呼吸障害の臨床研究プロトコール, 第39回四国新生児医療研究会, May 2022.
4.	Masashi Suzue, Ken-ichi Suga and Ryuji Nakagawa : 新生児中枢性尿崩症に対してデスモプレシン口腔内崩壊錠で治療したDe Morsier症候群の1例, 第73回中国四国小児科学会, Nov. 2021.
5.	Masashi Suzue, Ken-ichi Suga and Ryuji Nakagawa : デスモプレシン口腔内崩壊錠で治療した中枢性尿崩症の超早産児例, 第117階徳島周産期研究会, Sep. 2021. (Keyword) congenital
6.	Hiroki Sato, Ken-ichi Suga, Masashi Suzue, Ryuji Nakagawa, Aya Gohji, Yukako Honma, Shunsuke Takeuchi, Takahiro Tayama, Mikiko Takei, Keisuke Fujioka, Akemi Ono, Miki Shono, Koichi Sichijo, Shuji Kondo and Yasunobu Hayabuchi : Ellis-van Creveld 症候群の1例, 第38階四国新生児医療研究会, Jun. 2021. (Keyword) congenital
7.	Ken-ichi Suga, Keita Osumi, Masashi Suzue and Ryuji Nakagawa : A case of hypophosphatasia complicated by chylous ascites, 第65回日本新生児成育医学会, May 2021. (Keyword) congenital
8.	Masashi Suzue, Ken-ichi Suga, Takashi Kaji and Ryuji Nakagawa : 周産期重症型低ホスファターゼ症の1例, 第116回徳島周産期研究会, Dec. 2020.
9.	Maki Urushihara, 庄野実希, Masashi Suzue and Shoji Kagami : 新生児におけるアンジオテンシノーゲンの臨床的意義の検討, 第24回日本小児高血圧研究会, Aug. 2017.
10.	Yumiko Kotani, 岡田朝美, 近藤梨恵子, Sato Matsura, Masashi Suzue, 岸揚子, 山上貴司, 長谷川高誠 and Shoji Kagami : 多彩な骨折歴をもつCOL1A2 遺伝子異常症の1 例, 第148回日本小児科学会徳島地方会, Jun. 2017.
11.	Masashi Suzue, 阿部孝典, Ryuji Nakagawa, 渡辺典子, Takahiko Saijyou and Shoji Kagami : 低出生体重児の難治性低血糖に対するジアゾキシド投与, 第140回日本小児科学会徳島地方会, Jun. 2013.

Grants-in-Aid for Scientific Research (KAKEN Grants Database @ NII.ac.jp)

3歳児検尿における慢性腎臓病と先天性腎尿路奇形の新規バイオマーカーの開発 (Project/Area Number: 23K07269 )
血管内皮細胞障害に着目した新生児急性呼吸窮迫症候群の病態解明と治療法の開発 (Project/Area Number: 21K15867 )
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Assistant Professor : Suzue, Masashi

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Grants-in-Aid for Scientific Research (KAKEN Grants Database @ NII.ac.jp)